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anti-Human GPR3 Anticorps:
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Human Polyclonal GPR3 Primary Antibody pour ELISA, WB - ABIN314298
Tanaka, Ishii, Kasai, Yoon, Saeki: Neural expression of G protein-coupled receptors GPR3, GPR6, and GPR12 up-regulates cyclic AMP levels and promotes neurite outgrowth. dans The Journal of biological chemistry 2007
Hamster Polyclonal GPR3 Primary Antibody pour ICC, IF - ABIN1048790
Ledent, Demeestere, Blum, Petermans, Hämäläinen, Smits, Vassart: Premature ovarian aging in mice deficient for Gpr3. dans Proceedings of the National Academy of Sciences of the United States of America 2005
these results suggest an important role of the allosteric sodium binding site for GPR3 activity and open a possible avenue for the modulation of Abeta production in the Alzheimer's Disease.
Results demonstrate that GPR3 signals at the plasma membrane and can be silenced by GRK2/beta-arrestin overexpression. These results also strongly implicate the serine and/or threonine residues in the third intracellular loop in the regulation of GPR3 activity.
Gpr3 stimulates Abeta production via interactions with APP and beta-arrestin2.
GPR3 is a key factor in the regulation of the nervous system and follicle development.[review]
GPR3 is expressed in the ovary and testes, and is necessary for the regulation of meiosis in mouse.
Mutations in GPR3 are not a common cause of premature ovarian failure in Chinese women.
control of gene expression in vascular endothelial cells in the presence of fluid shear stress and classifies it as a sphingosine 1-phosphate receptor.
We conclude that perturbations in GPR3 are not a common explanation for POF in this population.
These results demonstrate that GPR3 potentiates meiotic competence, most likely by raising cAMP.
GPR3 appears to have a key role in the thermogenic response of interscapular brown adipose tissue and may represent a new therapeutic target in age-related obesity.
This study found that the expression of GPR3 in CGNs exerted prosurvival effects against apoptosis-inducing stimuli, such as potassium deprivation, hypoxia, and ROS.
Altered signalling pathways of GPR3 could contribute to the neurobiological substrate involved in developing addiction to cocaine
Involvement of the orphan GPR3 receptor is demonstrated for the first time in expression and development of neuropathic pain and in analgesia induced by morphine.
the GPR3 receptor is a link in communication between the somatic cells and oocyte of the ovarian follicle and is crucial for the regulation of meiosis
Gpr3-defective mice may constitute a relevant model of premature ovarian failure due to early oocyte aging.
the cAMP levels required for maintaining meiotic arrest in mouse and rat oocytes are dependent on the expression of Gpr3 and/or Gpr12
GPR3 specifically in the oocyte, rather than in the follicle cells, is responsible for maintenance of meiotic arrest in mouse oocytes.
GPR3-G(s) activation in response to stimulation of an exogenously expressed beta(2)-adrenergic receptor causes Galpha(s) to move from the oocyte plasma membrane into the cytoplasm.
Gpr3 is epistatic to Pde3a and fertility as well as meiotic arrest in the PDE3A-deficient oocyte is dependent on the activity of GPR3.
GPR3 qualifies as a new player in the modulation of behavioral responses to stress and constitutes a novel promising pharmacological target for treatment of emotional disorders
GPR3 is a novel antiproliferative mediator of CGPs in the postnatal development of murine cerebellu
Orphan receptor with constitutive G(s) signaling activity that activate cyclic AMP. Has a potential role in modulating a number of brain functions, including behavioral responses to stress (By similarity), amyloid-beta peptide generation in neurons and neurite outgrowth (By similarity). Maintains also meiotic arrest in oocytes (By similarity).
G protein-coupled receptor 3
, ACCA orphan receptor
, G-protein coupled receptor 3
, adenylate cyclase constitutive activator
, G protein-coupled receptor R4
, G-protein-coupled receptor R4