Use your antibodies-online credentials, if available.
Il n’y a pas de produits dans votre liste de comparaison.
Votre panier est vide.
Afficher toutes les espèces
Afficher tous les synonymes
Sélectionnez vos espèces et l'application
anti-Human PTH Anticorps:
anti-Mouse (Murine) PTH Anticorps:
anti-Rat (Rattus) PTH Anticorps:
Vous arrivez à notre recherche pré-filtrée.
Human Monoclonal PTH Primary Antibody pour IHC (fro), IHC (p) - ABIN2476062
Uzych: Hawaii and the AIDS epidemic. dans Hawaii medical journal 1990
Show all 4 Pubmed References
Human Monoclonal PTH Primary Antibody pour ELISA, WB - ABIN1724741
Koshizuka, Ogata, Shiraki, Hosoi, Seichi, Takeshita, Nakamura, Kawaguchi: Distinct association of gene polymorphisms of estrogen receptor and vitamin D receptor with lumbar spondylosis in post-menopausal women. dans European spine journal : official publication of the European Spine Society, the European Spinal Deformity Society, and the European Section of the Cervical Spine Research Society 2006
Show all 2 Pubmed References
Human Monoclonal PTH Primary Antibody pour ELISA, WB - ABIN966922
French, Hamilton, Mattano, Sather, Devidas, Nachman, Relling: A PAI-1 (SERPINE1) polymorphism predicts osteonecrosis in children with acute lymphoblastic leukemia: a report from the Children's Oncology Group. dans Blood 2008
Show all 3 Pubmed References
Human Monoclonal PTH Primary Antibody pour ELISA, ICC - ABIN260228
Koh, Hogue, Sosa: A Novel Ex Vivo Method for Visualizing Live-Cell Calcium Response Behavior in Intact Human Tumors. dans PLoS ONE 2016
Dkk-1 (Montrer DKK1 Anticorps) and PTH levels in psoriatic arthritis are lower than healthy female controls, in contrast with rheumatoid arthritis, in which they are increased
Scientific evidence of genetic association of serum PTH level among individuals with different pathologic conditions remains deficient and published results provide weak evidence (review and meta-analysis).
Clinical data on secondary hyperparathyroidism, mainly derived from patients with chronic kidney disease, indicate a potential inverse association between leptin (LEP (Montrer LEP Anticorps)) and parathyroid hormone (PTH) in some, but not all studies. [REVIEW]
Parathyroid hormone controls bone and kidney homeostasis via GNAS (Montrer GNAS Anticorps) and Gq-G11 (Montrer STK19 Anticorps) heterotrimeric G proteins. (Review)
Patients with chronic kidney disease undergoing parathyroidectomy for primary hyperparathyroidism have similar intraoperative parathyroid hormone degradation kinetics to those with normal kidney function.
PTH levels are significantly higher in untreated sustained hypertension patients than white-coat hypertension patients and normotensive subjects.
results revealed that PTH treatment on HCs (Montrer HLCS Anticorps), either continuous or pulsatile, does not exhibit any positive effect, and indicates that exogenous PTH administration after fracture has no effect on HCs (Montrer HLCS Anticorps). PTH may not have a positive effect at the fracture site during the early stage of fracture healing in which haematoma formation occurs.
Pre-incubation of muscle fibers or myotubes with physiological concentrations of PTH, concentration-dependently reduced uptake of labelled 25-hydroxyvitamin D3.
Baseline PTH level was not associated with changes in frailty status in men.
Elevated PTH induces the transition of endothelial cells to chondrogenic cells via endothelial-mesenchymal transition, possibly mediated by the nuclear translocation of beta-catenin (Montrer CTNNB1 Anticorps).
suppression of miR (Montrer MLXIP Anticorps)-185 targeting PTH could promote osteoblast growth and proliferation during fracture healing through activating Wnt (Montrer WNT2 Anticorps)/beta -catenin (Montrer CTNNB1 Anticorps) axis
lack of endogenous PTH may reduce VEGF (Montrer VEGFA Anticorps) expression in bone marrow mesenchymal stem cellsderived osteoblasts.
findings identify p38alpha (Montrer MAPK14 Anticorps) MAPK (Montrer MAPK1 Anticorps) as a key component of PTH signaling in osteoblast lineage cells and highlight its requirement in iPTH osteoanabolic activity
FGF23 (Montrer FGF23 Anticorps) may be an important modulator of PTH signaling in bone and kidney.
These findings suggest that trabecular bone formation can occur independently of the CaSR (Montrer CASR Anticorps), and that the CaSR (Montrer CASR Anticorps) plays a collaborative role in the PTH anabolic effects on bone.
Mmp13 (Montrer MMP13 Anticorps) is selectively regulated of by 1,25-Dihydroxyvitamin D3, PTH, and Osterix (Montrer SP7 Anticorps) through distal enhancers.
PTHrP and PTH mediate wasting through a common mechanism involving PTHR (Montrer PTH1R Anticorps).
Usp2 (Montrer USP2 Anticorps) is required for the PTH1-34-induced proliferation of osteoblasts
These results highlight the role of distal enhancers in the regulation of RANKL (Montrer TNFSF11 Anticorps) expression by PTH and perhaps 1,25(OH)2D3 and suggest that the RL-D2 and RL-D5 enhancers contribute in either an additive or synergistic manner to regulate bone remodeling.
These results indicate that PTH-mediated inhibition of renal phosphate transport involves phosphorylation of S77 of the NHERF-1 PDZ I domain and the dissociation of NHERF-1/Npt2a complexes.
These data highlight the ability of PTH to phosphorylate beta-catenin (Montrer CTNNB1 Anticorps) directly via PKA.
These data suggest that prostaglandin E2 acting via EP4R (Montrer PTGER4 Anticorps) on bone marrow macrophages committed to the osteoblast cell lineage, stimulated secretion of a factor or factors that acted to suppress PTH-stimulated osteoblast differentiation.
These results suggest that in vivo PTH treatment increased in vitro osteoclastogenesis and resorption without altering the number of osteoclast precursors.
Osteoblast 2-deoxyglucose uptake and glycogen (Montrer GYS2 Anticorps) synthesis were increased after exposure to low concentrations (0.1 nmol/l and above) of PTH.
Parathyroid hormone stimulated growth and decreased Col-X deposition via phosphotidylinositol-3,4,5 triphosphate kinase and mitogen activating protein kinase pathways in avian sterna.
likely involvement of the Sp family in regulating PTH gene expression through interactions with an Sp1 (Montrer SP1 Anticorps) DNA element in the hormone's promoter.
alternative cis (Montrer CISH Anticorps)-acting protein-binding elements may determine the regulation of PTH mRNA stability in response to changes in serum calcium and phosphate
a possible role for the Wnt (Montrer WNT2 Anticorps) signaling pathway in PTH actions in bone
Data suggest that calcium-mediated destabilization of parathyroid hormone mRNA requires gene transcription and involves increases in cytosolic Ca.
The protein encoded by this gene is a hormone secreted by parathyroid cells. This hormone elevates blood Ca2+ level by dissolving the salts in bone and preventing their renal excretion. Defects in this gene are a cause of familial isolated hypoparathyroidism (FIH).
, parathyroid hormone 1
, preproparathyroid hormone
, hypothalamic parathyroid hormone
, thyroid hormone
, parathyroid hormone