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anti-Mouse (Murine) ICOS Anticorps:
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Mouse (Murine) Monoclonal ICOS Primary Antibody pour BR, FACS - ABIN1176983
Buonfiglio, Bragardo, Redoglia, Vaschetto, Bottarel, Bonissoni, Bensi, Mezzatesta, Janeway Jr, Dianzani: The T cell activation molecule H4 and the CD28-like molecule ICOS are identical. dans European journal of immunology 2001
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Mouse (Murine) Monoclonal ICOS Primary Antibody pour FACS - ABIN2689139
Chambers: The expanding world of co-stimulation: the two-signal model revisited. dans Trends in immunology 2001
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Human Monoclonal ICOS Primary Antibody pour FACS - ABIN2689138
Dong, Nurieva: Regulation of immune and autoimmune responses by ICOS. dans Journal of autoimmunity 2003
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Human Polyclonal ICOS Primary Antibody pour IHC (p), ELISA - ABIN545650
Riley, Mao, Kobayashi, Biery, Burchard, Cavet, Gregson, June, Linsley: Modulation of TCR-induced transcriptional profiles by ligation of CD28, ICOS, and CTLA-4 receptors. dans Proceedings of the National Academy of Sciences of the United States of America 2002
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Human Monoclonal ICOS Primary Antibody pour Func, FACS - ABIN610261
Fusaro, Wang, Chellappan: Differential regulation of Rb family proteins and prohibitin during camptothecin-induced apoptosis. dans Oncogene 2002
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Human Monoclonal ICOS Primary Antibody pour Func, FACS - ABIN610262
Saitoh, Pizzi, Wang: Perturbation of SUMOlation enzyme Ubc9 by distinct domain within nucleoporin RanBP2/Nup358. dans The Journal of biological chemistry 2002
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Human Polyclonal ICOS Primary Antibody pour ELISA, IHC - ABIN4222313
Strauss, Bergmann, Szczepanski, Lang, Kirkwood, Whiteside: Expression of ICOS on human melanoma-infiltrating CD4+CD25highFoxp3+ T regulatory cells: implications and impact on tumor-mediated immune suppression. dans Journal of immunology (Baltimore, Md. : 1950) 2008
study has revealed a new intracellular mechanism in which Akt2 regulates ICOS expression via FoxO-1 and this signaling axis regulates the differentiation and function of NKT17 cells. This study provides a new linker between cell metabolism and function of iNKT cells.
These results indicate that ICOS plays a significant role in orchestrating the innate immune response to Staphylococcus aureus and other airway pathogens, and could be a potential immunomodulatory target to attenuate Staphylococcus aureus-related immunopathology.
ICOS differentially regulates effector T cells in secondary lymphoid organs and inflamed tissues.
we identified ICOS as a key costimulatory pathway that controls induction and maintenance of collagen-induced arthritis
this study shows that ICOS protects against mortality from acute lung injury through activation of IL-5(+) type 2 innate lymphocytes
Binding of NUFIP2 to Roquin promotes recognition and regulation of ICOS mRNA.
Data show that inducible T cell co-stimulator (ICOS) deficient mice have a significant increase in the population of IL-17-producing Vgamma2+ gammadelta T cells in the thymus, spleen, lymph nodes and skin and exhibit exacerbated sensitization responses to 2,4-dinitrofluorobenzene.
Our results indicate that ICOS regulates the ontogeny and homeostasis of B6.Sle1 lupus prone mouse T follicular helper (TFH) cells and influences the function of TFH cells during aberrant germinal center B cell responses
The expression of ICOSL in the cornea and the ICOS-mediated induction of Foxp3+ CD4+ regulatory T cells may contribute to successful corneal allograft survival.
this study reveals a previously unknown ICOS-TBK1 signaling pathway that specifies the commitment of germinal center follicular helper T cells
IFNAR1-deficiency accelerated humoral immune responses and parasite control by boosting ICOS-signalling in two non-lethal murine models of malaria
Fecal levels of Crp1 were significantly decreased in mice with graft-versus-host disease and were correlated with intestinal flora diversity.
IL-10 production by CD4(+) YFP(+) T cells is controlled systemically during malaria infection through IL-27 receptor signaling that is supported after CD4(+) T cell priming by ICOS signaling.
these data suggest ICOS is not required for follicular Th cell induction during Plasmodium chabaudi chabaudi AS infection
the ICOS signaling has a regulatory effect on CD154/CD40 expressions, and may play an important role in the hepatic egg granuloma formation of schistosomiasis.
data identify a role of ICOS in innate immunity and indicate that not only cytokines, but also costimulatory pathways such as those involving ICOS, can contribute to regulate the ILC2 pool.
results indicate that ILC2s might interact with B7RP-1-expressing DCs in allergic inflammatory lung, and ICOS signaling can positively regulate the protease allergen-induced ILC2 activation followed by eosinophil infiltration into the lungs
these data show a role for IL-6 in promoting "effector" Tregs accrual with age likely through maintenance of ICOS expression.
These data demonstrate the importance of ICOS signals specifically for memory CD4(+) T-cell formation, while highlighting the potential of therapeutically targeting this pathway.
ICOS and Bcl6-dependent pathways maintain a CD4 T cell population with memory-like properties during tuberculosis.
Circulating follicular helper T cells exhibit reduced ICOS expression and impaired function in narcolepsy type 1 patients.
PD-1 and ICOS on peripheral CD3(+)CD8(+) Tem and Tfh cells may be involved in the development of rheumatoid arthritis (RA) and may be an indicator of RA activity.
The leptin upregulated RA CD4(+)CXCR5(+)ICOS(+) T cells via increased IL-6 by activation of STAT1 and STAT3.
Interactions between the ICOS rs10932029 genotype and hepatitis B virus A1762T or A1762T/G1764A mutations could decrease the risk of liver cirrhosis in hepatitis b virus infection.
CXCR5 and ICOS expression identifies a CD8 T-cell subset with TFH features in Hodgkin lymphomas.
our findings demonstrated that polymorphisms in the 3'-UTRs of genes in the ICOS/CD28-ICOSL pathway may influence the efficacy and occurrence of adverse events of capecitabine-based chemotherapy in advanced colon cancer patients
ICOSL-ICOS signaling promoted Treg differentiation from CD4(+) T cells through activation of the phosphoinositide 3-kinase-Akt pathway. MSCs primed with Interleukin-1beta significantly induced Tregs through ICOSL upregulation. We demonstrated that the Treg-inducing activity of MSCs is proportionate to their basal ICOSL expression.
ICOS (-693A/G) gene polymorphism is associated with Colon Cancer Risk.
The altered soluble (s)PD1 and sICOS serum levels in the different Hepatitis b (HBV) groups may reflect the dysregulation of T cell activation, and may be associated with the HBV pathological process.
we have found an association between chronic spontaneous urticaria and polymorphisms in the ICOS gene. To our knowledge, this report is the fi rst to suggest and initially indicate a possible association of the ICOS rs6726035 TT genotype with CSU pathogenesis.
Suggest that genetic polymorphisms of ICOS function as sex-dependent risk factors for development of acute rejection in an Iranian kidney transplant population.
OX40 and ICOS act in a cooperative, nonredundant manner to maximize and prolong the T follicular helper cell response that is generated after acute virus infection
High ICOS expression is associated with colorectal neoplasms.
This is the first replication association study of rs4325730 upstream of ICOS with AIH in the Japanese population and rs4325730G is a risk allele.
the induction pathway of ICOS(+) Foxp3(+) cells was analyzed
ratio of ICOS(+) /PD-1(+) Tfh cells and the frequency of IL-21(+) Tfh cells may be indicators for evaluating the idiopathic membranous nephropathy development
The ICOS and ICOSL SNPs examined do not have an apparent effect on the disease susceptibility and prognosis of autoimmune thyroid diseases.
Follicular helper T cells differentiation is a multistage process involving BCL6 and other transcription factors, cytokines, and costimulation through ICOS and several other molecules. (Review)
novel deletion mutation in Pakistani siblings presents in childhood with a combined Immunodeficiency associated with an enteritis, hepatitis and impaired antiviral immunity
The protein encoded by this gene belongs to the CD28 and CTLA-4 cell-surface receptor family. It forms homodimers and plays an important role in cell-cell signaling, immune responses, and regulation of cell proliferation.
inducible T-cell costimulator
, inducible T-cell co-stimulator
, CD28 and CTLA-4-like protein
, CD28-related protein 1
, activation-inducible lymphocyte immunomediatory molecule
, inducible costimulator