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Human Polyclonal CRP Primary Antibody pour ICC, IF - ABIN258345
Tabuchi, Inoue, Usui-Kataoka, Kobayashi, Teramoto, Takasugi, Shikata, Yamamura, Ando, Nishida, Kasahara, Kume, Lopez, Mitsudo, Nobuyoshi, Yasuda, Kita, Makino, Matsuura: The association of C-reactive protein with an oxidative metabolite of LDL and its implication in atherosclerosis. dans Journal of lipid research 2007
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Human Monoclonal CRP Primary Antibody pour IF, ELISA - ABIN191156
Cook, Boy, Flowers, Daroca: The influence of high-altitude living on body iron. dans Blood 2005
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Human Monoclonal CRP Primary Antibody pour ELISA (Capture), IF - ABIN191157
Silaste, Rantala, Alfthan, Aro, Witztum, Kesäniemi, Hörkkö: Changes in dietary fat intake alter plasma levels of oxidized low-density lipoprotein and lipoprotein(a). dans Arteriosclerosis, thrombosis, and vascular biology 2004
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Human Monoclonal CRP Primary Antibody pour IF, ELISA - ABIN191158
Tarkkinen, Palenius, Lövgren: Ultrarapid, ultrasensitive one-step kinetic immunoassay for C-reactive protein (CRP) in whole blood samples: measurement of the entire CRP concentration range with a single sample dilution. dans Clinical chemistry 2002
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A significant association was found between high hs-CRP and Lp-PLA2 levels and carotid stenosis (adjusted odds ratio [OR]: 2.39; 95% confidence interval [CI]: 1.13-5.09), but this combination was not associated with carotid plaques (OR: 2.62, 95% CI: 0.93-7.38). The results suggested that the combination of hs-CRP and Lp-PLA2 were better predictors than either protein alone with regard to carotid atherosclerosis.
there is a trans-cardiac decrease in CRP in non-STEMI; CRP uptake is not limited to the infarcted myocardium but is also prominent in regions adjacent to injured myocardium
considering erythrocyte sedimentation rate, CRP and albumin as additional markers to fecal calprotectin significantly improved diagnostic performance
This meta-analysis suggests that C-reactive protein to albumin ratio may be a powerful prognostic indicator for colorectal cancer prognosis.
In aortic stenosis patients, hs-CRP at baseline is an independent predictor of post-transcatheter aortic valve implantation mortality.
After uncomplicated total knee arthroplasty CRP showed a gradual rise to peak at 48 hours, then fell gradually, remaining elevated at 4 days and higher than baseline level at 2 weeks.
an elevated CRP/ALB ratio on intensive care unit admission was an independent risk factor for 30-day mortality rate.
Cardiac biomarker hsCRP can be used as the most reliable cardiac biomarker.
Elevated serum hs-CRP was associated with future Premature Ventricular Contraction in participants without history of myocardial infarction or stroke. Elevated hs-CRP was an independent predictor of Premature Ventricular Contraction in Chinese population, especially in men.
visceral adipose tissue is a source of CRP in acute inflammation
There was a significant difference in the proportions of people with elevated C-reactive protein (CRP) in schizophrenia and healthy control .
increased serum levels of hepcidin and C-reactive protein in brucellosis can be considered a diagnostic biomarker of inflammation and active disease
We also discuss the impact of independent factors and C-reactive protein genetic polymorphisms on baseline plasma C-reactive protein levels
Our study shows, for the first time, that a different genetic association related with gender could contribute to AMD risk. As a consequence, the risk of female gender in the different CRP levels and A69S SNP frequencies could be taken into consideration to the established risk relationship of high levels of CRP and its association with risk A69S genotype.
Measurement of serum high-sensitivity C reactive protein (hs-CRP) in addition to the CURB-65 model improved the clinical usefulness in predicting ICU admission and mortality in communityacquired pneumonia (CAP) patients.
CRP levels on admission to hospital after first myocardial infarction are associated with an increased risk of heart failure.
High CRP/prealbumin expression is associated with recurrence in gastric cancer.
Persistently elevated C-reactive protein levels independently predicted the development of atrial fibrillation in a Korean population.
The observation of protective and susceptible association of crp - 757T > C polymorphism may be useful for better management and prophylaxis of periodontitis.
High CRP expression is associated with postoperative complications in pancreatic, hepatic and gastric tumors.
Enhanced external counterpulsation decreased C-reactive protein and increased flow-mediated dilation in hypercholesterolemia.
serum C-reactive protein correlates with macrophage accumulation and coronary artery disease in hypercholesterolemic pigs
CRP inhibits endothelium-dependent NO-mediated dilation in coronary arterioles by producing superoxide from NAD(P)H oxidase via p38 kinase activation.
The present study shows that elevated levels of serum CRP and IL-10 were associated with porcine circovirus type 2-infected piglets that subsequently developed severe PMWS. (postweaning multisystemic wasting syndrome)
These data suggest that CRP might play a pathogenic role in the development of myointimal hyperplasia in polytetrafluoroethylene grafts.
myocardial infarction induced proinflammatory gene and protein expression in peripheral blood mononuclear cells of tissue factor cyclo-oxygenase-2, monocyte chemoattractant protein-1 and CRP
CRP stimulates superoxide production and the subsequent formation of peroxynitrite from basal released nitric oxide compromises PGI(2) synthesis.
Data show that during posttranplant obliterative bronchiolitis development, CRP is localized in inflammatory cells, myofibroblasts and endothelial cells probably as a part of the local inflammatory response.
this study shows that C-reactive protein impairs dendritic cell development, maturation, and function
CRP is pathogenic in type-2 diabetes (T2DN). CRP may promote CD32b- NF-kappaB signaling to mediate renal inflammation; whereas, CRP may enhance renal fibrosis in T2DN via CD32b-Smad3-mTOR signaling.
FcgammaRIIb and FcgammaRIII are involved in the CRP-induced expression of MMPs and TIMP-1 and the CRP-induced phosphorylation of p38, whereas the FcgammaR-independent pathway may regulate the CRP-induced MMP-11 expression and the CRP-induced ERK1/2 phosphorylation.
C-reactive protein may promote acute kidney injury by impairing tubular epithelial cell regeneration via the CD32-Smad3-p27-driven inhibition of the CDK2/cyclin E complex.
These results suggested that CRP/oxidized low density lipoprotein/beta 2-Glycoprotein I complexes may induce lipid accumulation and inflammation in macrophages via the p38/MAPK and NFkappaB signaling pathways.
these data unexpectedly identify CBS as the major recognition site of mCRP and suggest that this motif may be exploited to tune the proinflammatory actions of mCRP.
CRP decreased adiponectin expression and multimerization, while CRP-induced decline in adiponectin might be mediated through the PI3K/Akt pathway.
mCRP may be responsible for promoting dementia after ischaemia and mCRP clearance could inform therapeutic avenues to reduce the risk of future dementia.
Effects of C-Reactive Protein on the Cardiac Differentiation of Mouse Embryonic Stem Cells
These data indicated that CRP may have a direct role on osteoclast and osteoblast differentiation via toll-like receptor signaling pathways.
Data suggest that serum amyloid P (SAP) activates CD209 DC-SIGN to regulate the innate immune system differently from C-reactive protein (CRP), and that DC-SIGN is a target for antifibrotics.
the acute phase protein C-reactive protein (CRP), a ligand for Fc receptors on phagocytes, enhances antibody-mediated platelet destruction by phagocytes in vivo
CRP has therapeutic potential against cancer through decreasing the accumulation of M2 macrophages and angiogenesis within tumors.
Orphan nuclear receptor Errgamma induces C-reactive protein gene expression through induction of ER-bound Bzip transmembrane transcription factor CREBH.
The interactions of CRP and the BBB are a two-phase process, with increased paracellular permeability at a high dose that enables its entry into the CNS and serves to induce reactive gliosis and impair CNS function.
Radiation combined injury-induced alterations of corticosterone, CRP, C3, IgM, and PGE2 cause homeostatic imbalance and may contribute to reduced survival.
In mice with photochemically induced thrombosis, acrolein produced at the locus of infarction increased the level of IL-6 and then CRP in plasma.
CRP directly inhibits tumoral lymphangiogenesis and lymph node metastasis in mice.
Which processes in glucose homeostasis are altered by CRP? Data suggest that CRP attenuates insulin-induced skeletal muscle blood flow; CRP contributes to insulin resistance via IgG receptor IIB-mediated inhibition of glucose delivery to muscle.
CRP is not only a biomarker, but also a mediator in DKD.
data show, for the first time, a significant increase of c-reactive protein (CRP) in the blood of calves 1 d after colostrum intake.
Rabbit CRP acute-phase system binds phosphocholine and phosphoethanolamine.
IL-6 augments mechanical strain-induced CRP synthesis in vessels via the stretch-activated channel-NF-kappaB pathway.
CRP antagonism of eNOS is mediated by coupling of FcgammaRI to FcgammaRIIB by Src kinase and activation of inositol 5'-phosphatase 1, and consistent with this mechanism, both FcgammaRI and FcgammaRIIB are required for CRP to blunt endothelial repair in vivo.
Metformin attenuated the progression of aortic atherosclerosis via down-regulation of serum C-reactive protein.
During abdominal aortic aneurysm formation, increased serum hsCRP levels derive from aneurysmal arteries with degenerating elastic lamina.
crystal of C-reactive protein diffracted to 2.3 A resolution and belonged to space group R3, with unit-cell parameters a = b = 114.7, c = 61.0 A
The protein encoded by this gene belongs to the pentaxin family. It is involved in several host defense related functions based on its ability to recognize foreign pathogens and damaged cells of the host and to initiate their elimination by interacting with humoral and cellular effector systems in the blood. Consequently, the level of this protein in plasma increases greatly during acute phase response to tissue injury, infection, or other inflammatory stimuli.
, pentraxin 1
, C-reactive protein, petaxin related
, C-reactive protein member of the pentraxin family
, C-reactive protein, member of the pentraxin family
, amyloid P component, serum
, serum amyloid P component
, pentraxin-related C-reactive protein
, uncharacterized protein LOC751795