Il n’y a pas de produits dans votre liste de comparaison.
Votre panier est vide.
Afficher tous les synonymes
Sélectionnez vos espèces d'intérêt
TUSC3 accelerates cancer growth and induces the epithelial-mesenchymal transition in non-small-cell lung cancer cells by upregulating claudin-1.
miR-UL112-3p exerts its oncogene function by directly targeting TUSC3 in Glioblastoma.
study demonstrated an oncogenic role of TUSC3 in Non-small cell lung cancer and showed that dis-regulation of TUSC3 may affect tumour cell invasion and migration through possible involvement in the Hedgehog (Hh) signalling pathway.
TUSC3 may act as an oncogene in the progression of colorectal cancer.
TUSC3 can function both as an oncogene and as a tumor suppressor. (Review)
our data indicate that miR-132 induces temozolomide resistance and promotes the formation of cancer stem cell phenotypes by targeting TUSC3 in glioblastoma.
This paper supports the previous clinical descriptions of the condition caused by TUSC3 mutations and describes the seventh family with mutations in this gene, thus contributing to the genetic spectrum of mutations. This is the first report of a family from the Arabian peninsula with this form of Intellectual disability .
SOX2 regulates the proliferation, migration and invasiveness of breast cancer cells through miR-181a-5p and miR-30e-5p which modulate TUSC3 protein levels
Decreased Tumor Suppressor Candidate 3 Predicts Poor Prognosis of Patients with Esophageal Squamous Cell Carcinoma
TUSC3 regulates proliferation and invasion of glioblastoma cells by inhibiting the activity of the Akt signaling pathway.
decreased immunological TUSC3 staining is a factor prognostic of poor survival in pancreatic cancer patients.
The TUSC3 gene is associated with mental retardation in the Qinba mountain area in China; the sixth exon of the TUSC3 gene may contribute to the risk of developing the disease.
Report frequencies of short tandem repeat markers linked to TUSC3 (MRT7) or NSUN2 (MRT5) genes used for homozygosity mapping of recessive intellectual disability.
TUSC3 loss alters the ER stress response and accelerates prostate cancer growth in vivo
Loss of TUSC3 alters the molecular response to endoplasmic reticulum stress.
IGFII and N33 methylation status may be related to gastric carcinogenesis.
TUSC3 increases glycosylation efficiency for a subset of human glycoproteins by slowing glycoprotein folding.
Homozygous deletion in TUSC3 causes syndromic intellectual disability.
TUSC3 is a tumor suppressor gene in ovarian cancer.
Genotyping and linkage analysis excluded linkage of the GRIK2 gene and TUSC3 gene with mental retardation.
This gene is a candidate tumor suppressor gene. It is located within a homozygously deleted region of a metastatic prostate cancer. The gene is expressed in most nonlymphoid human tissues including prostate, lung, liver, and colon. Expression was also detected in many epithelial tumor cell lines. Two transcript variants encoding distinct isoforms have been identified for this gene.
tumor suppressor candidate 3
, Putative prostate cancer tumor suppressor
, magnesium uptake/transporter TUSC3
, oligosaccharyltransferase 3 homolog A
, putative prostate cancer tumor suppressor