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anti-Mouse (Murine) BCL6 Anticorps:
anti-Human BCL6 Anticorps:
anti-Rat (Rattus) BCL6 Anticorps:
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Human Monoclonal BCL6 Primary Antibody pour IHC, WB - ABIN2668887
García, García, Maestre, Lucas, Sánchez-Verde, Romero-Chala, Piris, Roncador: Genetic immunization: a new monoclonal antibody for the detection of BCL-6 protein in paraffin sections. dans The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society 2005
Show all 2 Pubmed References
Human Polyclonal BCL6 Primary Antibody pour IF (p), IHC (p) - ABIN749843
Szabó, Gáspár, Dajnoki, Papp, Fábos, Szegedi, Zeher: Expansion of circulating follicular T helper cells associates with disease severity in childhood atopic dermatitis. dans Immunology letters 2017
Human Polyclonal BCL6 Primary Antibody pour ELISA, ICC - ABIN6260236
Huang, Zhang, Hou, Wang, Liu, Zhang, Chen, Zhu: LncRNA AK023391 promotes tumorigenesis and invasion of gastric cancer through activation of the PI3K/Akt signaling pathway. dans Journal of experimental & clinical cancer research : CR 2018
Human Polyclonal BCL6 Primary Antibody pour WB - ABIN4283623
Margalit, Amram, Amariglio, Simon, Shaklai, Granot, Minsky, Shimoni, Harmelin, Givol, Shohat, Oren, Rechavi: BCL6 is regulated by p53 through a response element frequently disrupted in B-cell non-Hodgkin lymphoma. dans Blood 2006
BCL6 forms a complex with BCL6 corepressor (BCoR (Montrer BCOR Anticorps)) on the promoters of selected Notch (Montrer NOTCH1 Anticorps) target genes such as enhancer of split related 1.
Bcl-6, expressed in abundance in Tfr (Montrer TFRC Anticorps) cells, inhibits CD25 (Montrer IL2RA Anticorps) expression and IL-21 (Montrer IL21 Anticorps)-mediated inhibition of CD25 (Montrer IL2RA Anticorps) is Bcl-6 dependent.
Together these results identify BCL6 as a potential driver of ETV6 (Montrer ETV6 Anticorps)-RUNX1 (Montrer RUNX1 Anticorps)-mediated leukemogenesis, which could involve loss of BTG1 (Montrer BTG1 Anticorps)-dependent suppression of ETV6 (Montrer ETV6 Anticorps)-RUNX1 (Montrer RUNX1 Anticorps) function.
this study shows that IL4 (Montrer IL4 Anticorps) and IL21 (Montrer IL21 Anticorps) cooperate to induce the high Bcl6 protein level required for germinal center formation
Although BCL6 controls follicular helper T cells activity in humans and mice, the role of miR (Montrer MLXIP Anticorps)-31 is restricted to human follicular helper T cell differentiation, reflecting a species specificity of the miR (Montrer MLXIP Anticorps)-31 action.
These findings indicate a role of the interaction between TH2-promoting factors and Bcl6 in promoting appropriate IL-4 (Montrer IL4 Anticorps) production in mTH2 (Montrer NUDT15 Anticorps) cells and suggest that chronic allergic diseases involve the TH2-promoting factor-mediated functional breakdown of Bcl6, resulting in allergy exacerbation.
Bioinformatics analysis and the dual-luciferase reporter assay demonstrated that miR (Montrer MLXIP Anticorps)-10b could target the 3'-untranslated regions of B cell lymphoma 6 (Bcl6) which is an important regulator of osteoblast differentiation.
Def6 (Montrer DEF6 Anticorps) limits proliferation of T follicular helper cells in mice via alteration of mTORC1 signaling and inhibition of Bcl6 expression.
The balance between CD4 (Montrer CD4 Anticorps)(+) cytotoxic T cell and follicular helper T(Tfh) differentiation heavily depends on the class of infecting virus and is jointly regulated by the Tfh-related transcription factors Bcl6 and Tcf7 (Montrer TCF7 Anticorps) (encoding TCF-1 (Montrer HNF1A Anticorps)) and by the expression of the inhibitory receptors PD-1 (Montrer PDCD1 Anticorps) and LAG3 (Montrer LAG3 Anticorps).
Study provides evidence for an essential role of Bcl6 in the complex regulation of gene transcription during early adipogenesis. The action of Bcl6 is at least partially mediated by the direct transcriptional activation of STAT1 (Montrer STAT1 Anticorps).
These data describe a novel regulatory mechanism through which STAT3 (Montrer STAT3 Anticorps) and the Ikaros (Montrer IKZF1 Anticorps) zinc finger transcription factors Aiolos (Montrer IKZF3 Anticorps) and Ikaros (Montrer IKZF1 Anticorps) cooperate to regulate Bcl-6 expression.
our exploratory study suggests that EOMES, BCL6 and GZMB gene expression are aberrant within the PB T cell transcriptome of HT patients. The association of this transcription signature with the heterogeneity of HT and disease control is suggested.
Cryptic t(3;8)(q27;q24) and/or MYC (Montrer MYC Anticorps)-BCL6 linkage associated with MYC (Montrer MYC Anticorps) expression by immunohistochemistry is frequent in multiple-hit B-cell lymphomas
BCL6 overexpression in SHR reduced blood pressure, NLRP3 expression and inflammation in the renal cortex of SHR
Aberrant CD10 (Montrer MME Anticorps) and BCL6 expression defines a subset of MCLs with higher mean Ki-67 (Montrer MKI67 Anticorps) index and higher prevalence of MUM1 (Montrer IRF4 Anticorps) expression
Double-hit lymphoma (DHL) is an aggressive form of DLBCL with an unmet treatment need, in which MYC (Montrer MYC Anticorps) rearrangement is present with either BCL2 (Montrer BCL2 Anticorps) or BCL6 rearrangement
BCL6 is a growth promoting factor in glioblastoma and glioma.
IFN gamma induced (Montrer SAMHD1 Anticorps) upregulation of BCL6 was dependent on the classical STAT1 (Montrer STAT1 Anticorps) signaling pathway, and affected both major BCL6 variants. Interestingly, although IFN alpha induced stronger STAT1 (Montrer STAT1 Anticorps) phosphorylation than IFN gamma (Montrer IFNG Anticorps), it only slightly upregulated BCL6 in multiple myeloma lines.
Findings demonstrate that BCL6 expression is downregulated by miR (Montrer MLXIP Anticorps)-519d which targets its 3 '-UTR (Montrer UTS2R Anticorps). Also, BCL6 mediates the repression of miR (Montrer MLXIP Anticorps)-519d on cell proliferation and invasive capability of gastric cancer cells.
In Pakistani population, the frequency of GCB (Montrer GBA Anticorps) type DLBCL [diffuse large B cell lymphoma ]expressing CD10 (Montrer MME Anticorps) and BCL6 is 37.5%, and non- GCB (Montrer GBA Anticorps) type DLBCL [diffuse large B cell lymphoma ] expressing MUM1 (Montrer IRF4 Anticorps) is 62.5%.
The protein encoded by this gene is a zinc finger transcription factor and contains an N-terminal POZ domain. This protein acts as a sequence-specific repressor of transcription, and has been shown to modulate the transcription of START-dependent IL-4 responses of B cells. This protein can interact with a variety of POZ-containing proteins that function as transcription corepressors. This gene is found to be frequently translocated and hypermutated in diffuse large-cell lymphoma (DLCL), and may be involved in the pathogenesis of DLCL. Alternatively spliced transcript variants encoding different protein isoforms have been found for this gene.
B-cell CLL/lymphoma 6
, B-cell CLL/lymphoma 6 (zinc finger protein 51)
, B-cell lymphoma 6 protein
, zinc finger protein 51
, B-cell leukemia/lymphoma 5
, B-cell leukemia/lymphoma 6
, B-cell lymphoma 6 protein homolog
, B-cell lymphoma 5 protein
, B-cell lymphoma 6 protein transcript
, cys-his2 zinc finger transcription factor
, lymphoma-associated zinc finger gene on chromosome 3
, protein LAZ-3
, zinc finger and BTB domain-containing protein 27
, zinc finger transcription factor BCL6S