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anti-Human TGFB1 Anticorps:
anti-Mouse (Murine) TGFB1 Anticorps:
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Various Species Monoclonal TGFB1 Primary Antibody pour CyTOF, ELISA (Capture) - ABIN4900861
Phillips: Rapid analysis of inflammatory cytokines in cerebrospinal fluid using chip-based immunoaffinity electrophoresis. dans Electrophoresis 2004
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Various Species Monoclonal TGFB1 Primary Antibody pour CyTOF, ELISA (Capture) - ABIN4900860
Rafei, Wu, Annabi, Lejeune, François, Galipeau: A GMCSF and IL-15 fusokine leads to paradoxical immunosuppression in vivo via asymmetrical JAK/STAT signaling through the IL-15 receptor complex. dans Blood 2007
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Human Polyclonal TGFB1 Primary Antibody pour IF (p), IHC (p) - ABIN724685
Kou, Hu, Yao, Wang, Shen, Kang, Hong: Transforming growth factor-?1 promotes Treg commitment in nasal polyposis after intranasal steroid treatment. dans Inflammation research : official journal of the European Histamine Research Society ... [et al.] 2013
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Human Monoclonal TGFB1 Primary Antibody pour FACS - ABIN4898892
Yamauchi, Ueki, Konno, Ito, Takeda, Nakamura, Nishikawa, Moritoki, Omokawa, Saga, Hirokawa: The effect of hepatocyte growth factor on secretory functions in human eosinophils. dans Cytokine 2016
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Human Monoclonal TGFB1 Primary Antibody pour FACS - ABIN4897280
Azukizawa, Döhler, Kanazawa, Nayak, Lipp, Malissen, Autenrieth, Katayama, Riemann, Weih, Berberich-Siebelt, Lutz: Steady state migratory RelB+ langerin+ dermal dendritic cells mediate peripheral induction of antigen-specific CD4+ CD25+ Foxp3+ regulatory T cells. dans European journal of immunology 2011
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Human Polyclonal TGFB1 Primary Antibody pour WB - ABIN223608
Tamaki, Hatano, Taura, Tada, Kodama, Nitta, Iwaisako, Seo, Nakajima, Ikai, Uemoto: CHOP deficiency attenuates cholestasis-induced liver fibrosis by reduction of hepatocyte injury. dans American journal of physiology. Gastrointestinal and liver physiology 2008
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Human Monoclonal TGFB1 Primary Antibody pour CyTOF, FACS - ABIN4900169
Boswell, Sharif, Alisa, Pereira, Williams, Behboudi et al.: Induction of latency-associated peptide (transforming growth factor-β(1)) expression on CD4+ T cells reduces Toll-like receptor 4 ligand-induced tumour necrosis factor-α production in a transforming ... dans Immunology 2011
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Mouse (Murine) Monoclonal TGFB1 Primary Antibody pour CyTOF, FACS - ABIN4899119
Lee, Park, Woo, Park, Kang et al.: RhoA/phosphatidylinositol 3-kinase/protein kinase B/mitogen-activated protein kinase signaling after growth arrest-specific protein 6/mer receptor tyrosine kinase engagement promotes epithelial cell ... dans The Journal of pharmacology and experimental therapeutics 2014
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Human Monoclonal TGFB1 Primary Antibody pour ELISA, FACS - ABIN4359062
Makboul, Makboul, Abdelhafez, Hassan, Youssif: Evaluation of the effect of fractional CO2 laser on histopathological picture and TGF-β1 expression in hypertrophic scar. dans Journal of cosmetic dermatology 2014
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Human Monoclonal TGFB1 Primary Antibody pour EIA, Func - ABIN114558
Crawford, Stellmach, Murphy-Ullrich, Ribeiro, Lawler, Hynes, Boivin, Bouck: Thrombospondin-1 is a major activator of TGF-beta1 in vivo. dans Cell 1998
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TGF-beta1 regulated pAKT (Montrer AKT1 Anticorps) and IFNgamma expressions were associated with epithelial cell survival in rhesus macaque colon explants and suggest a potential role of mucosal TGF-beta1 in regulating intestinal homeostasis and EC integrity.
These findings suggest that FBLN5 (Montrer FBLN5 Anticorps) may interfere with choroidal neovascularization by downregulating VEGF (Montrer VEGFA Anticorps), CXCR4 (Montrer CXCR4 Anticorps), and TGFB1 expression and inhibiting choroidl endothelial cell proliferation.
SIV infection of rhesus macaques results in the emergence of IL-17 (Montrer IL17A Anticorps)-expressing cells during the acute phase. This subpopulation appears at day 14 postinfection concomitantly with an increase in TGF-beta and IL-18 (Montrer IL18 Anticorps) expression.
SIV-infected macaques exhibiting progression to AIDS displayed greater expression of TGF-beta and indoleamine 2,3 dioxygenase in CD8 (Montrer CD8A Anticorps)+ T cells from mesentric lymph nodes.
transforming growth factor beta (TGFbeta) is required for hematopoietic progenitor cell specification. The requirement for TGFbeta is two fold and sequential: autocrine via Tgfbeta1a and Tgfbeta1b produced in the endothelial cells themselves, followed by a paracrine input of Tgfbeta3 from the notochord, suggesting that the former programs the hemogenic endothelium and the latter drives endothelial-to-hematopoietic tran...
The fine tuning of TGF-beta signaling derives from positive and negative control by Ldb2a (Montrer LDB2 Anticorps).
TGFbeta1a regulates zebrafish posterior lateral line formation via Smad5 (Montrer SMAD5 Anticorps) mediated pathway.
PCSK7 (Montrer PCSK7 Anticorps) is essential for zebrafish development and regulates the expression and proteolytic cleavage of TGFbeta1a.
TGFbeta signaling orchestrates the beneficial interplay between scar-based repair and cardiomyocyte-based regeneration to achieve complete heart regeneration.
TGFbeta1 plays a role in zebrafish keratocyte migration.
data presented show that Zili (Montrer PIWIL2 Anticorps) suppresses TGF-beta signaling by physically associating with Smad4 (Montrer SMAD4 Anticorps) and preventing the formation of Smad2 (Montrer SMAD2 Anticorps)/3/4 and Smad1 (Montrer SMAD1 Anticorps)/5/9/4 complexes
TGF-beta1 acts at multiple sites, including LH receptor (Montrer LHCGR Anticorps), 20beta-HSD (Montrer HAL Anticorps) and membrane progestin receptor-beta (Montrer PAQR8 Anticorps), to inhibit zebrafish oocyte maturation
These data suggest Pez (Montrer PTPN14 Anticorps) plays a crucial role in organogenesis by inducing TGFbeta and epithelial-mesenchymal transition.
Data show that Rock2 (Montrer ROCK2 Anticorps) acts as a negative regulator of the TGFbeta signaling pathway.
These data indicate that TGF-beta signaling is crucial for the function of the transition zone, which in turn may affect the regulation of cilia length.
R-Smads are the key components of TGFbeta beta signals in germ layer induction. SCP3 (Montrer SYCP3 Anticorps) serves as a vegetally enriched, intrinsic factor (Montrer GIF Anticorps) to ensure a prepared status of Smads for their activation.
the present in vitro system, which permits not only the cell contraction-mediated cell sorting but also the TGF-b-directed mesodermal induction such as cartilage formation, may fairly reflect the embryogenesis in vivo.
Loss of XTgfbi impaired blastopore formation and dorsal tissue morphogenesis.
TGF-beta signaling has a role in nuclear localization of transcription factor Smad4 (Montrer SMAD4 Anticorps)
sortilin (Montrer SORT1 Anticorps) negatively regulates TGF-beta signaling by diverting trafficking of precursor proteins to the lysosome during transit through the biosynthetic pathway
our findings suggest that tumor extracellular vesicle (EV)-educated mesenchymal stem cells promote osteosarcoma progression and provide the basis for testing IL6 (Montrer IL6 Anticorps)- and TGFbeta-blocking agents as new therapeutic options for osteosarcoma patients.
Lipopolysaccharides-mediated TLR4 (Montrer TLR4 Anticorps) signaling enhances TGF-beta response through downregulating BAMBI (Montrer BAMBI Anticorps) during prostatic hyperplasia.
This work additionally suggests ILEI (Montrer FAM3C Anticorps) mediates TGF-beta1-dependent Epithelial-to-mesenchymal transitions (EMTs) via the extracellular regulated protein kinases (ERKs) and protein kinase B (Akt (Montrer AKT1 Anticorps)) signalling pathways.
conclude that Lipin-1 (Montrer LPIN1 Anticorps) can antagonize HSC (Montrer FUT1 Anticorps) activation through the inhibition of TGF-beta/SMAD (Montrer SMAD1 Anticorps) signaling and that resveratrol may affect Lipin-1 (Montrer LPIN1 Anticorps) gene induction and contribute to the inhibition of TGF-beta-mediated hepatic fibrogenesis
SARS (Montrer SARS Anticorps) coronavirus papain-like protease significantly triggered Egr-1 (Montrer EGR1 Anticorps) dependent activation of TGF-beta1 promoter via reactive oxygen species/p38 MAPK (Montrer MAPK14 Anticorps)/STAT3 (Montrer STAT3 Anticorps) pathway.
TGF-beta1 has a role in stimulating the migration of type II endometrial cancer cells by activating SMAD (inducing SMAD 2/3 phosphorylation and knocking down SMAD4) and ERK 1/2, which down-regulates PTEN
overview of primary cilia-mediated regulation of receptor tyrosine kinase (Montrer RET Anticorps) (RTK- PDGFRa (Montrer PDGFRA Anticorps) and PDGFRb (Montrer PDGFRB Anticorps)) and transforming growth factor beta (TGF-beta) signaling [review]
The present study indicates that TGFB1 variants have subtype-specific roles in BC and may switch from tumor suppressor to promoter during tumor development, consistent with TGFbeta1 dual role in BC pathogenesis.
the expression of TGF-beta1 was related to shorter survival time and rapid progression for gastric cancer patients
Results suggest that high TGF-beta1 expression promotes a poor prognosis in hepatocellular carcinoma patients. [meta-analysis]
Activated TGF-beta signaling rescued miR (Montrer MYLIP Anticorps)-143-reduced FSHR (Montrer FSHR Anticorps) and intracellular signaling molecules, and miR (Montrer MYLIP Anticorps)-143-induced porcine granulosa cell apoptosis.
TGF-beta1-induced epithelial-myofibroblast plasticity is FAK (Montrer PTK2 Anticorps)- dependent, whereas TGF-beta1-induced apoptosis is favored when FAK (Montrer PTK2 Anticorps) signaling is inhibited.
this study shows that TGF-beta1 protects intestinal integrity and influences Smad (Montrer SMAD1 Anticorps) and MAPK (Montrer MAPK1 Anticorps) signal pathways in intestinal epithelium cells after TNF-alpha (Montrer TNF Anticorps) challenge
this study shows that anemonin may ameliorate LPS (Montrer IRF6 Anticorps)-induced intestinal injury and improve restoration by regulating the TGF-b1 and EGFR (Montrer EGFR Anticorps) signaling pathways
The results indicated that TGF-beta-1 was associated with the restoration of intestinal morphology and barrier function following weaning stress.
Data (including data from in vitro and in vivo experiments) suggest that day 14 elongated conceptus secretes proteins that up-regulate TGFbeta1 mRNA and TGFbeta1 expression in endometrium; TGFbeta1 may be important during pregnancy maintenance.
The effects of semen, spermatozoa in extender, or extender alone on the expression of TGF-beta1, IL-10 (Montrer IL10 Anticorps), and IL-6 (Montrer IL6 Anticorps) in ovarian follicles are reported.
Dietary (1,3/1,6)-beta-D-glucan reduced the mRNA expression of transforming growth factor (TGF) beta2 (Montrer TGFB2 Anticorps) and tended to reduce the mRNA expression of TGF-beta1 in lung tissue of neonatal piglet.
TGF-beta1, via TGF-beta1 receptor I and p38 MAPK (Montrer MAPK14 Anticorps) signaling, reduces CFTR (Montrer CFTR Anticorps) expression to impair CFTR (Montrer CFTR Anticorps)-mediated anion secretion, which would likely compound the effects associated with mild CFTR (Montrer CFTR Anticorps) mutations and ultimately would compromise male fertility.
High yield isolation of BMP-2 (Montrer BMP2 Anticorps) from bone and in vivo activity of a combination of BMP-2 (Montrer BMP2 Anticorps)/TGF-beta1.
TGF-beta1 stimulated lubricin (Montrer PRG4 Anticorps) secretion by superficial zone chondrocytes at all densities with twice-a-week TGF-beta treatment. It is noteworthy that the daily treatment of TGF-beta1 increased lubricin (Montrer PRG4 Anticorps) much higher compared with twice-a-week treatment.
hypoxia increased the expression of platelet-derived growth factor (PDGF (Montrer PDGFA Anticorps)) and transforming growth factor-beta1 (TGF-beta1) and decreased the expression of neprilysin (NEP (Montrer MME Anticorps)), which contributed to the hypoxia-induced Endothelial-to-mesenchymal transition of pulmonary artery endothelial cells.
TGF-beta1 modulates the expression of syndecan-4 (Montrer SDC4 Anticorps) in cultured vascular endothelial cells in a biphasic manner.
Taken together, Staphylococcus aureus induces TGF-beta1 and bFGF (Montrer FGF2 Anticorps) expression through the activation of AP-1 (Montrer JUN Anticorps) and NF-kappaB (Montrer NFKB1 Anticorps) in bovine mammary gland fibroblasts.
The results identify TGFB1 and ESRRA (Montrer ESRRA Anticorps) as likely transcriptional regulators of rumen epithelial development and energy metabolism, respectively, and provide targets for modulation of rumen development and function in the growing calf.
the combined treatment with TGF-beta1 and BMP-7 (Montrer BMP7 Anticorps) or treatment first with TGF-beta1 followed by BMP-7 (Montrer BMP7 Anticorps) was more effective than other treatment groups in both chondrogenic differentiation and SZP (Montrer PRG4 Anticorps) secretion.
Tenascin-X (Montrer TNXB Anticorps) promotes activation of latent TGF-beta1 and subsequent epithelial to mesenchymal transition in mammary epithelial cells.
a detailed computational model for TGF-beta signalling that incorporates elements of previous models together with crosstalking between Smad1 (Montrer SMAD1 Anticorps)/5/8 and Smad2 (Montrer SMAD2 Anticorps)/3 channels through a negative feedback loop dependent on Smad7 (Montrer SMAD7 Anticorps).
Endogenous TGF-beta1 became more bioactive following activation of the transgene protein product in chondrocytes.
A novel peptide, P2K, regulating TGF-beta1 signaling had an anabolic effect on bovine intervertebral disc cells and rabbit degenerated discs.
These data suggest that the interplay between cell-matrix adhesion and intercellular adhesion is an important determinant for some aspects of TGFbeta1-induced epithelial-mesenchymal transition via alphaSMA (Montrer ACTA2 Anticorps) expression induction.
Excessive activation of TGFbeta by spinal instability causes vertebral endplate sclerosis and intervertebral disk degeneration.
study reveals a critical mechanism by which TGFbeta controls TH17 cell differentiation and uncovers the SKI (Montrer SKI Anticorps)-SMAD4 (Montrer SMAD4 Anticorps) axis as a potential therapeutic target for treating TH17-related diseases
A Smad3 (Montrer SMAD3 Anticorps)-PTEN regulatory loop controls proliferation and apoptotic responses to TGF-beta in mouse endometrium.
TGFss through the Alk1 (Montrer ACVRL1 Anticorps)/TgfssR2 receptor axis is acting on endothelial cells to produce hematopoiesis.
The adoptive transfer of NK1.1(-) CD4 (Montrer CD4 Anticorps)(+) NKG2D (Montrer KLRK1 Anticorps)(+) cells suppressed DSS (Montrer PMP22 Anticorps)-induced colitis largely dependent on TGF-beta. Thus, NK1.1(-) CD4 (Montrer CD4 Anticorps)(+) NKG2D (Montrer KLRK1 Anticorps)(+) cells exhibited immune regulatory functions, and this T cell subset could be developed to suppress inflammation in clinics.
The present study describes the ability of color-coded intravital imaging to demonstrate the efficacy of a TGF-beta inhibitor to target stroma in an orthotopic mouse model of pancreatic cancer.The present study describes the ability of color-coded intravital imaging to demonstrate the efficacy of a TGF-beta inhibitor to target stroma in an orthotopic mouse model of pancreatic cancer.
The ratio of full-length versus cleaved uPAR (Montrer PLAUR Anticorps) as analysed by Western blotting and its regulation was assessed by addition of different protease inhibitors and transforming growth factor - beta1 (TGF-beta1). The role of uPAR (Montrer PLAUR Anticorps) cleavage in cell proliferation and migration was analysed using real-time cell analysis and invasion was assessed using the myoma invasion model
Hepatic Stellate Cells express iron-transport proteins. Holotransferrin (iron) activates HSC (Montrer FUT1 Anticorps) fibrogenesis and the TGF-beta pathway, whereas iron depletion by chelation reverses this, suggesting that this could be a useful adjunct therapy for patients with fibrosis.
The results show that lnc-Spry1 (Montrer SPRY1 Anticorps) could act as an early mediator of TGF-beta signaling and reveal different roles for a long noncoding RNA in modulating transcriptional and posttranscriptional gene expression.
Data indicate that cardiac contractility modulation (CCM) therapy exerted protective effects against myocardial fibrosis potentially by inhibiting TGF-beta1/Smad3 (Montrer SMAD3 Anticorps) signaling pathway in chronic heart failure .
study suggested that TGF-beta1/Smad3/smad7 is a major pathway which plays an important role in the regulation of the IUA and specific inhibitor of Smad3 (SIS3) may provide a new therapeutic strategy for IUA.
cell therapy promoted TGF-beta1 expression in nucleus pulposus, leading to anti-inflammatory effects via the inhibition of NF-kappaB (Montrer NFKB1 Anticorps), and the amelioration of disc degradation.
that prenatal tracheal occlusion increases TGF-beta/Rho kinase (Montrer ROCK1 Anticorps) pathway, myofibroblast differentiation, and matrix deposition in neonatal rabbit and human congenital diaphragmatic hernia lungs
observation. Based on the above results, we conclude that TGF-beta1-immobilized PLGA-gelatin scaffold seeded with ASCs considerably enhances the quality of the tissue-engineered cartilage, therefore, advancing the field of cartilage tissue engineering
Smad7 (Montrer SMAD7 Anticorps) plays a crucial role in antagonizing epithelial-mesenchymal transition induced by TGFbeta signaling and is a Notch (Montrer NOTCH1 Anticorps) signaling target in limbal epithelial stem cells.
the role of IL-10 in inhibited allograft rejection may be associated with CD4+CD25+ regulatory T cells and IL-10, and may be independent of TGF-b
After NOTCH1 (Montrer NOTCH1 Anticorps) knockdown and TGFB1 stimulation, rabbit mesenchymal stem cells expressed higher levels of proteoglycan (Montrer Vcan Anticorps) and collagen II.
TGF-beta1 gene transcription significantly correlates with the surgical vaginal and dermal wound closure rate.
TGF beta is involved in the pathogenesis of tympanosclerosis.
There was significantly higher expression of TGF-beta1 and MMP-9 (Montrer MMP9 Anticorps) in nasal mucosa of experimental allergic rhinitis guinea pigs than in controls.
Within the limitations of the study design, production of COMP (Montrer COMP Anticorps) during healing of skin wounds does not appear to be influenced by wound type or anatomic site, nor does it appear to be correlated with TGF-beta1 concentrations.
Peritoneal TGF-beta(1) concentration was higher in horses with severe gastrointestinal diseases, in horses with an altered peritoneal fluid, and in nonsurvivors.
This gene encodes a member of the transforming growth factor beta (TGFB) family of cytokines, which are multifunctional peptides that regulate proliferation, differentiation, adhesion, migration, and other functions in many cell types. Many cells have TGFB receptors, and the protein positively and negatively regulates many other growth factors. The secreted protein is cleaved into a latency-associated peptide (LAP) and a mature TGFB1 peptide, and is found in either a latent form composed of a TGFB1 homodimer, a LAP homodimer, and a latent TGFB1-binding protein, or in an active form composed of a TGFB1 homodimer. The mature peptide may also form heterodimers with other TGFB family members. This gene is frequently upregulated in tumor cells, and mutations in this gene result in Camurati-Engelmann disease.
TGF-beta 1 protein
, latency-associated peptide
, transforming growth factor beta-1
, transforming growth factor, beta 1 (Camurati-Engelmann disease)
, transforming growth factor-beta 1
, transforming growth factor beta 1
, transforming growth factor-beta
, transforming growth factor beta1
, transforming growth factor-beta-1
, tgf beta
, tgf-beta 5
, transforming gorwth factor-Beta5
, transforming growth factor-B5
, transforming growth factor-beta 5
, TGF-beta 1
, regulatory protein
, transforming growth factor, beta-1
, transforming growth factor, beta 1
, transforming growth factor, beta-induced, 68kDa
, transforming growth factor beta-1-like
, transforming growth factor beta 4