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Activation of DPF3a upon hypertrophic stimuli in cardiac hypertrophy switches cardiac fetal gene expression from being silenced by HEY to being activated by BRG1.
identified and characterized human HES-like (HESL), rat hesl, and rainbow trout hesl genes by using bioinformatics
the combination of gain- and loss-of-function experiments in the developing midbrain showed co-operative roles for Mgn and Mash1 genes in determining GABAergic identity.
the prethalamic factors Dlxs upregulated by Ascl1/Helt deficiency play unique roles in regulating thalamic progenitor specification.
Helt acts as a transcriptional repressor to regulate neuronal differentiation and/or identity
identification of a novel mouse bHLH factor, termed Heslike, that has Hes1-like bHLH domain and transcriptional repressor activity; Heslike is coexpressed with the bHLH factor Mash1 in brain regions that give rise to GABAergic neurons
Mgn is a single copy gene on mouse chromosome 8 and encodes a 27kDa protein that functions in the nucleus.
Homozygous Mgn-null mice die between the second and the fifth postnatal week of age, and show a complete depletion of Gad65 and Gad67 expression in the superior colliculus and a reduction in the inferior colliculus.
Helt determines GABAergic over glutamatergic fate, by repressing Ngn genes; Helt and Ngns are commonly used in the mesencephalon for determination of the GABAergic versus glutamatergic transmitter phenotype.
Transcriptional repressor which binds preferentially to the canonical E box sequence 5'-CACGCG-3' (By similarity).
HES/HEY-like transcription factor
, Hey-like transcription factor
, Hey-like transcriptional repressor
, Megane bHLH factor
, hairy and enhancer of split-related protein HELT
, bHLH transcription factor megane
, mesencephalic GABAergic neurons enhancer of split related protein
, protein megane
, HES/HEY-like transcription factor, pseudogene 1
, Her/Hey-like protein
, hairy and enhancer of split-related protein helt