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anti-Human PLA2G16 Anticorps:
anti-Mouse (Murine) PLA2G16 Anticorps:
anti-Rat (Rattus) PLA2G16 Anticorps:
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study uncovers two competing processes triggered by Picornaviridae virus entry: activation of a pore-activated clearance pathway and recruitment of a PLA2G16 phospholipase to enable genome release
Demonstrate increased PLA2G16 expression activates the MAPK (Montrer MAPK1 Anticorps) pathway to enhance osteosarcoma metastasis.
our results revealed that H-rev107 (Montrer HRASLS Anticorps) is also involved in lipid accumulation in liver cells through the POR (Montrer POR Anticorps) pathway via its PLA2 (Montrer YWHAZ Anticorps) activity.
Results show that osteosarcoma patients with metastasis showed higher expression of PLA2G16 at both the mRNA and protein levels and shorter overall survival suggesting it as significant prognostic factor for poor outcome.
the flexible main loop of H-REV107, but not that of TIG3, is critical for its NTD to modulate its CTD in inducing cell death.
crystal structure of the HRASLS3-LRAT (Montrer LRAT Anticorps) chimeric enzyme in a thioester catalytic intermediate state revealed a major structural rearrangement accompanied by three-dimensional domain swapping dimerization not observed in native HRASLS (Montrer HRASLS Anticorps)
several mutant p53 (Montrer TP53 Anticorps) proteins bind the Pla2g16 promoter at E26 transformation-specific (ETS (Montrer ETS1 Anticorps)) binding motifs and knockdown of ETS2 (Montrer ETS2 Anticorps) suppressed mutant p53 (Montrer TP53 Anticorps) induction of Pla2g16.
Our study further suggests that the PLA/AT activity of H-rev107 may play an important role in H-rev107-mediated RAS suppression.
an alternate mechanism for AdPLA in promoting adipose tissue lipolysis that is not contingent on the release of arachidonic acid and that is compatible with its combined PLA(1 (Montrer POU2F3 Anticorps))/A(2) activity
Data show that acyl-modified forms of HRAS (Montrer HRAS Anticorps)-like tumor suppressors HRASLS2 (Montrer HRASLS2 Anticorps) and HRASLS3 mimicking lipolytic activity of lecithin retinol acyltransferase LRAT (Montrer LRAT Anticorps).
H-rev107 (Montrer HRASLS Anticorps), through PTGDS (Montrer PTGDS Anticorps), suppressed cell migration and invasion. Data suggest that the PGD2 (Montrer PTGDS Anticorps)-cAMP-SOX9 (Montrer SOX9 Anticorps) signal pathway might play an important role in H-rev107 (Montrer HRASLS Anticorps)-mediated cancer cell invasion in testes.
H-rev107 (Montrer HRASLS Anticorps) interferes with the biosynthesis of ether-type lipids and is responsible for the dysfunction of peroxisomes in H-rev107 (Montrer HRASLS Anticorps)-expressing cells.
identification of an Sp1 (Montrer SP1 Anticorps)/Sp3-binding GC-box required for the transcription of H-rev107 (H-rev107 (Montrer HRASLS Anticorps))
study found that AdPLA that was previously known as H-Rev-107 (Montrer HRASLS Anticorps)/HRASLS3 is an adipose-specific, calcium-dependent histidine PLA2 (Montrer PLA2G2A Anticorps)
HRASLS3 as one of the downstream effectors of PPARgamma (Montrer PPARG Anticorps) action in adipogenesis.
Single nucleotide polymorphisms in the adipose specific phospholipase A2 are associated with growth traits in cattle.
gene whose expression is suppressed by HRAS
HRAS-like suppressor 3
, H-rev 107 protein homolog
, group XVI phospholipase A1/A2
, group XVI phospholipase A2
, Ca-independent phospholipase A1/2
, HRAS-like suppressor 1
, adipose-specific PLA2
, adipose-specific phospholipase A2
, renal carcinoma antigen NY-REN-65
, H-rev 107 protein
, HRAS like suppressor 3
, LRAT-like protein 3
, Hras-revertant gene 107 (expression down-regulated in HRAS-transformed rat 208F fibroblasts)