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anti-Human FSHR Anticorps:
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Human Polyclonal FSHR Primary Antibody pour IF (p), IHC (p) - ABIN729508
Luo, Yin, Zhang, Yuan, Zhao, Luan, Zhang: Role of SDF-1/CXCR4 and cytokines in the development of ovary injury in chemotherapy drug induced premature ovarian failure mice. dans Life sciences 2017
Show all 2 Pubmed References
Human Polyclonal FSHR Primary Antibody pour ELISA, WB - ABIN560952
Ritter, Thuering, Saint Mezard, Luong-Nguyen, Seltenmeyer, Junker, Fournier, Susa, Morvan: Follicle-stimulating hormone does not impact male bone mass in vivo or human male osteoclasts in vitro. dans Calcified tissue international 2008
Human Polyclonal FSHR Primary Antibody pour WB - ABIN4909704
Wang, Wang, Zhu, Zhang, Sheng, Zhang, Han, Yuan, Weng: Seasonal expression of luteinizing hormone receptor and follicle stimulating hormone receptor in testes of the wild ground squirrels (Citellus dauricus Brandt). dans Acta histochemica 2018
INVESTIGATION OF STAT5A (Montrer STAT5A Anticorps), FSHR AND LHR (Montrer LHCGR Anticorps) GENE POLYMORPHISMS IN TURKISH INDIGENOUS CATTLE BREEDS
The expression of FSHR mRNA in granulosa cells was highest in small antral follicles, then decreased significantly as follicles increased in size, and was lowest in cysts.
transfer. We conclude that variation at these loci of the FSHR gene has no significant effect on pregnancy rates in Luxi cattle.
Specific alleles of the bovine FSHR gene are associated with variations in embryo yield and in the number of unfertilised oocytes.
This study evaluated the relationships among aromatase (Montrer CYP19A1 Anticorps), IGF-1 (Montrer IGF1 Anticorps), IGF2R (Montrer IGF2R Anticorps), and follicle stimulating hormone (FSH) receptor levels expressed in ovarian follicles of cattle selected for twin pregnancies.
granulosa cell clustering is accompanied by marked increases in FSHr, IGF-1r (Montrer IGF1R Anticorps), and p450 arom (Montrer CYP19A1 Anticorps) expression, and precedes induction and subsequent peak E2 production
Dominant follicles experience a reduction in FSH (Montrer BRD2 Anticorps) dependence (diminished expression of FSHR), but acquire increased LH dependence (enhanced expression of LHCGR (Montrer LHCGR Anticorps)) as they grow during the low FSH (Montrer BRD2 Anticorps) milieu of follicular waves.
FSHR is specifically regulated through androgen receptor (Montrer AR Anticorps) in granulosa cells
Heterozygous heifers showed a higher pregnancy rate (67 and 66% for LHR (Montrer LHCGR Anticorps) and FSHR genes, respectively), but no significant effects were observed for the genes studied (
Data show that double mutation of follicle-stimulating hormone receptor (fshr) and luteinizing hormone receptor (lhcgr (Montrer LHCGR Anticorps)) resulted in infertile males.
Data show for the first time in a vertebrate species that Leydig cells as well as Sertoli cells express the mRNAs for both fshr and lhcgr (Montrer LHCGR Anticorps).
Characterization of the first functional zebrafish (Danio rerio) gonadotropic hormone I receptor (follicle stimulating hormone receptor).
the novel homozygous FSHR variant observed in 2 siblings with Hypergonadotropic hypogonadism (Montrer LHCGR Anticorps) can expand the spectrum of FSHR mutations in humans.
Follicle-stimulating hormone receptor single nucleotide polymorphisms are not associated with male infertility.
the expression levels of placental FSHR mRNA and protein were significantly decreased in pregnancies complicated by preeclampsia.
Polymorphisms of the FSH receptor is associated with normal morphology and genetic maturation (metaphase II) oocytes in dependence of genotypic variation polymorphisms.
T cells redirected against FSHR(+) tumor cells with full-length FSH (Montrer BRD2 Anticorps) represent a promising therapeutic alternative against a broad range of ovarian malignancies, with negligible toxicity even in the presence of cognate targets in tumor-free ovaries
Findings of this study suggest a significant association between FSHR gene p. Thr307Ala or p. Asn680Ser coding sequence change and PCOS. The variant homozygote genotype results in a higher risk of PCOS.
The evaluation of sperm DNA fragmentation as a surrogate marker of sperm quality, and of the FSHR SNP rs6166 (p.N680S), might be useful to predict the response to FSH (Montrer BRD2 Anticorps) treatment in men with idiopathic infertility
Mouse chondrocytes and human articular cartilage express functional FSHR. Moreover, FSH (Montrer BRD2 Anticorps) can act on chondrocytes and cause genetic changes.
The mutation p.R59X in FSHR is causative for primary ovarian insufficiency by means of arresting folliculogenesis.
two mutations, V(221)G and T(449)N, in the extracellular domain and transmembrane helix 3, of FSHR, respectively, are reported.
These data highlight an important interdependency between the potent pro-inflammatory cytokine IL1A (Montrer IL1A Anticorps) and Fshr expression.
Study demonstrates expression of follicle-stimulating hormone receptor (FSHR) and a direct action of follicle-stimulating hormone on testicular stem/germ cells possibly mediated via alternatively spliced growth factor type 1 receptor FSHR3 in mice.
FSHR and LHR (Montrer LHCGR Anticorps) proteins are significantly upregulated in CCs (Montrer CCS Anticorps) surrounding oocytes arrested at the 2-cell stage, reflecting their developmental incompetence.
Triptorelin and cetrorelix induce immune responses and affect uterine development and expressions of genes and proteins of ESR1 (Montrer ESR1 Anticorps), LHR (Montrer LHCGR Anticorps), and FSHR
Brca1 (Montrer BRCA1 Anticorps)(GC-/-) models reveal that specific intra-follicular Brca1 (Montrer BRCA1 Anticorps) loss alone, or combined with cancer-promoting genetic (Trp53 (Montrer TP53 Anticorps) loss) and endocrine (high serum follicle-stimulating hormone) changes, was not sufficient to cause ovarian tumors.
Data (including date from knockout mice) suggest that Fshr is expressed early in pregnany in placenta and other extragonadal tissues of fetoplacental unit; expression is particularly strong at term.
Sertoli cell-specific expression of MTA2 (Montrer MTA2 Anticorps) is required for transcriptional regulation of FSHR gene during spermatogenesis.
By day 20 and in adult animals total AR or FSHR ablation significantly reduced Leydig cell numbers but Sertoli cell specific AR ablation had no effect.
The results demonstrate that gain-of-function mutations of the FSHR in mice bring about distinct and clear changes in ovarian function
The amount of dopamine d1 receptor (Montrer DRD1 Anticorps), dopamine D2 receptor (Montrer DRD2 Anticorps), and follicle stimulating hormone receptor (FSHr) mRNA were quantified in ovarian tissues in anestrous and mares expressing estrus during the breeding season are reported.
Activated TGF-beta (Montrer TGFB1 Anticorps) signaling rescued miR (Montrer MYLIP Anticorps)-143-reduced FSHR and intracellular signaling molecules, and miR (Montrer MYLIP Anticorps)-143-induced porcine granulosa cell apoptosis.
The results showed that polymorphisms in exon 10 of the FSHR gene had a significant effect on litter size traits of Wannan Black and Berkshire pigs. These results can be applied for marker-assisted selection in the 2 swine breeds
These results showed that an increased FSHR gene expression level was accompanied with an increase in histone H3K9 acetylation levels, suggesting that histone H3K9 acetylation could regulate the expression of the porcine FSHR gene.
248 F(2) animals from a Duroc and Meishan cross were genotyped for three FSHR SNPs at positions 74, 532 and 1166, and these were correlated with the phenotypes of litter size and corpus luteum number
findings suggest the FSH receptor may be involved in the early follicle formation in pigs, which begins during prenatal life.
FSHR is expressed in the hamster ovary starting from the fetal life to account for FSH (Montrer BRD2 Anticorps)-induced primordial follicle formation and cyclic AMP (Montrer TMPRSS5 Anticorps) production.
The protein encoded by this gene belongs to family 1 of G-protein coupled receptors. It is the receptor for follicle stimulating hormone and functions in gonad development. Mutations in this gene cause ovarian dysgenesis type 1, and also ovarian hyperstimulation syndrome. Alternative splicing results in multiple transcript variants.
follicle stimulating hormone receptor
, follicle-stimulating hormone receptor
, gonadotropic hormone I
, follicle-stimulating hormone receptor-like
, FSH receptor
, follitropin receptor