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14-3-3epsilon has a critical role in cellular metabolism involving either molecular crosstalk with the EcR or direct interaction with metabolic proteins
The Khc73 stalk/14-3-3 (Montrer YWHAZ Anticorps)/NudE pathway defines a physical connection that coordinates the activities of multiple motor proteins to precisely position the spindle.
role of 14-3-3epsilon in germ cell migration
14-3-3epsilon acts as a biochemical control point for axon guidance in Drosophila, silencing Plexin A repulsive axon guidance and regulating a semaphorin repulsion/integrin adhesion switch.
We conclude that 14-3-3epsilon is required for Rab11 (Montrer RAB11A Anticorps)-positive vesicle function, which in turn enables antimicrobial peptide (Montrer cAMP Anticorps) secretion during an innate immune response.
Hpo (Montrer GFER Anticorps) signaling inhibited Yki (Montrer YAP1 Anticorps) nuclear localization and activity by phosphorylating Yki (Montrer YAP1 Anticorps) and both isoforms of 14-3-3 (Montrer YWHAZ Anticorps), 14-3-3varepsilon and 14-3-3zeta (Montrer YWHAZ Anticorps), regulate Yki (Montrer YAP1 Anticorps) activity through modulating its subcellular localization.
Drosophila 14-3-3/PAR-5 (Montrer YWHAZ Anticorps) is an essential mediator of PAR-1 (Montrer F2R Anticorps) function in axis formation.
PAR-1 (Montrer F2R Anticorps) phosphorylates Bazooka/PAR-3 (Montrer PARD3 Anticorps) on two conserved serines to generate 14-3-3 (Montrer YWHAZ Anticorps) binding sites. This inhibits formation of the Bazooka (Montrer PARD3 Anticorps)/PAR-6 (Montrer PARD6A Anticorps)/aPKC complex by blocking Bazooka (Montrer PARD3 Anticorps) oligomerization and binding to aPKC.
Serves as a central modulator of forkhead transcription factor FoxO (Montrer FOXO Anticorps) activity in the regulation of growth, cell death and longevity in vivo.
The expression of epsilon isoforms of 14-3-3 protein (Montrer YWHAE Anticorps) was identified at substantial levels in the first instar larva, upregulated in the second instar larva, and the highest levels were maintained in the late stage of larva, the pupa, and the adult.
Positively regulates Ras-mediated pathways. Acts downstream or parallel to Raf, but upstream of nuclear factors in Ras signaling. Three mutants have been isolated, that suppress the rough eye phenotype caused by mutated Ras1 (sev-Ras1 v12). Inhibits yki activity by restricting its nuclear localization.
, suppressor of Ras85D 3-9
, 14-3-3 protein
, 14-3-3 protein epsilon
, 14-3-3 epsilon
, 14-3-3 epsilon protein
, tyrosine 3-monooxygenase/tryptophan 5-monooxygenase activation protein epsilon polypeptide