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Human Polyclonal ESRRA Primary Antibody pour ICC, IF - ABIN4309527
Silvennoinen, Rinnankoski-Tuikka, Vuento, Hulmi, Torvinen, Lehti, Kivelä, Kainulainen: High-fat feeding induces angiogenesis in skeletal muscle and activates angiogenic pathways in capillaries. dans Angiogenesis 2013
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Bat Polyclonal ESRRA Primary Antibody pour ChIP, IHC (p) - ABIN4309525
Fritah, Steel, Parker, Nikolopoulou, Christian, Carling, Parker: Absence of RIP140 reveals a pathway regulating glut4-dependent glucose uptake in oxidative skeletal muscle through UCP1-mediated activation of AMPK. dans PLoS ONE 2012
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ERRalpha is abundantly expressed in H(+)-pump-rich cells, a group of ionocytes responsible for H(+) secretion in the skin of developing embryos, and its expression is stimulated by acidic environments. Knockdown of ERRalpha impairs both basal and low pH-induced H(+) secretion in the yolk-sac (Montrer ADCY10 Anticorps) skin, which is accompanied by decreased expression of H(+)-secreting-related transporters.
Results propose Estrogen Receptor (Montrer ESR1 Anticorps)-Relatedalpha as a new regulator of morphogenetic movement during gastrulation, independently of cell fate determination.
The results identify TGFB1 (Montrer TGFB1 Anticorps) and ESRRA as likely transcriptional regulators of rumen epithelial development and energy metabolism, respectively, and provide targets for modulation of rumen development and function in the growing calf.
ERalpha (Montrer ESR1 Anticorps), is only expressed in the caruncle of the placenta.
ERalpha (Montrer ESR1 Anticorps) mRNA-expressing neurons (ERalpha (Montrer ESR1 Anticorps) neurons) were distributed in some structures of the telencephalon, diencephalon, mesencephalon, and hindbrain.
Findings suggest that PGF (Montrer PGF Anticorps)(2alpha) regulates luteolysis by ESR1 (Montrer ESR1 Anticorps) mRNA down-regulation.
the involvement of ERRalpha in the progression of cisplatin-induced AKI is probably through impaired mitochondrial dynamics
Localizing ESRRA binding sites in cortical chromatin, it has been shown that this nuclear receptor binds both differentially expressed energy-related and neurodevelopmental transcription factor genes during social challenge.
The Genomic Context and Corecruitment of SP1 Affect ERRalpha Coactivation by PGC-1alpha in Muscle Cells
findings highlight a MYC (Montrer MYC Anticorps)/ERRalpha pathway that contributes to physiological and pathological bone loss by integrating the MYC (Montrer MYC Anticorps)/ERRalpha axis to drive metabolic reprogramming during osteoclast differentiation
Study performed a structural and functional assessment of glutamatergic synapses on medium spiny neurons in the nucleus accumbens of Esrra-null mice, and discovered sex-specific alterations in miniature excitatory postsynaptic currents frequency and amplitude, reduced paired-pulse ratio, and reduction in glutamatergic synaptic vesicles consistent with altered functional connectivity
The nuclear receptor, estrogen-related receptor alpha (ERRalpha), was shown to regulate the expression of genes required for lactate utilization.
In bone, the effects of cholesterol, statin, and bisphosphonate on osteoclastogenesis require ERRalpha; and consequently, cholesterol-induced bone loss or bisphosphonate osteoprotection is lost in ERRalpha knockout mice.
We conclude that E2-induced ERRalpha expression in endothelium plays an important role for the E2-induced vasculoprotective effect
Exercise-induced changes in tumour LDH-B (Montrer LDHB Anticorps) and MCT1 (Montrer MCTS1 Anticorps) expression are modulated by oestrogen-related receptor alpha in breast cancer-bearing BALB/c mice
These results indicate that Esrra deficiency in the mouse brain impairs behavioral responses in multiple functional domains.
LC3B (Montrer MAP1LC3B Anticorps) and ESRRA might be a useful prognostic factor in patients with muscle-invasive bladder cancer. The co-expression of LC3B (Montrer MAP1LC3B Anticorps) and ESRRA might be a prognostic and therapeutic target for patients with bladder cancer.
These findings indicate that ERRalpha may serve as a novel molecular target for the treatment of endometrial cancer
Gls (Montrer GLS Anticorps) is a novel ERRalpha target gene.
can trigger the proliferation and migration of colorectal cancer cells via up regulation of IL-8 (Montrer IL8 Anticorps)
curcumin suppressed the ERRalpha gene expression through upregulation of miR (Montrer MLXIP Anticorps)-125a. Data from this study revealed a novel mechanism for curcumin-mediated apoptotic cell death, which involves tumor cell killing via activating miR (Montrer MLXIP Anticorps)-125a/ERRalpha pathway.
blocking the ERRalpha-controlled mitochondrial program largely inhibits the PLA2R1 (Montrer PLA2R1 Anticorps)-induced tumor-suppressive response. Together, our data document ERRalpha and its mitochondrial program as downstream effectors of the PLA2R1 (Montrer PLA2R1 Anticorps)-JAK2 (Montrer JAK2 Anticorps) pathway leading to oncosuppression.
results suggest that the decreased expression of miR (Montrer MLXIP Anticorps)-135a in metastatic tumors leads to elevated ERRalpha expression, resulting in increased cell invasion capacities.
HO-1 (Montrer HMOX1 Anticorps)-derived CO enhances mitochondrial biogenesis in astrocytes by activating L-type Ca(2 (Montrer CA2 Anticorps)+) channel-mediated PGC-1alpha/ERRalpha axis, leading to maintenance of astrocyte function and neuroprotection/recovery against damage of brain function
in ERalpha (Montrer ESR1 Anticorps) negative breast cancer, the low level of ERalpha (Montrer ESR1 Anticorps) reduced miR (Montrer MLXIP Anticorps)-497 expression, which promoted ERRalpha expression that enhanced cell proliferation, migration, and invasion by increasing MIF (Montrer AMH Anticorps) expression and MMP9 (Montrer MMP9 Anticorps) activity.
these data show that ERRalpha expression predicts response to tamoxifen treatment, and ERRalpha could be a biomarker of tamoxifen sensitivity and a prognostic factor in TNBC.
Results show that miR (Montrer MYLIP Anticorps)-125a inhibits porcine preadipocytes differentiation through targeting ERRa.
XCT790 significantly inhibited the expression of ERR alpha and lipid accumulation in porcine mature adipocytes.
The protein encoded by this gene is a nuclear receptor that is closely related to the estrogen receptor. This protein acts as a site-specific transcription regulator and has been also shown to interact with estrogen and the transcripton factor TFIIB by direct protein-protein contact. The binding and regulatory activities of this protein have been demonstrated in the regulation of a variety of genes including lactoferrin, osteopontin, medium-chain acyl coenzyme A dehydrogenase (MCAD) and thyroid hormone receptor genes. A processed pseudogene of ESRRA is located on chromosome 13q12.1.
, estrogen-related receptor 1
, steroid hormone receptor ERR1
, estrogen-related receptor alpha
, Steroid hormone receptor ERR1 (Estrogen-related receptor, alpha) (ERR-alpha) (Estrogen receptor-like 1)
, estrogen receptor related 1
, estrogen receptor-like 1
, nuclear receptor subfamily 3 group B member 1
, estrogen-related receptor, alpha
, orphan nuclear receptor
, estrogen-related nuclear receptor alpha