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Calcium Channel, Voltage-Dependent, beta 2 Subunit (CACNB2) Peptide

CACNB2 Reactivité: Mammifères Hôte: Synthetic BP, WB, IHC
N° du produit ABIN939158
  • Antigène Tous les produits CACNB2
    CACNB2 (Calcium Channel, Voltage-Dependent, beta 2 Subunit (CACNB2))
    Type de proteíne
    Synthetic
    Origine
    Mammifères
    Source
    • 29
    Synthetic
    Application
    Blocking Peptide (BP), Western Blotting (WB), Immunohistochemistry (IHC)
    Séquence
    HLADYLEAYW KATHPPSSSL PNPLLSRTLA TSSLPLSPTL ASNSQGSQGD
    Attributs du produit
    A synthetic peptide for use as a blocking control in assays to test for specificity of CACNB2 antibody,
    Alternative Names: CACNB2 control peptide, CACNB2 antibody Blocking Peptide, Anti-CACNB2 Blocking Peptide, Calcium Channel Voltage-Dependent Beta 2 Subunit Blocking Peptide, CACNLB2 Blocking Peptide, FLJ23743 Blocking Peptide, MYSB Blocking Peptide, CACNB2, CACNB-2, CACNB 2, CACNB-2 Blocking Peptide, CACNB 2 Blocking Peptide
  • Indications d'application
    Optimal conditions should be determined by the investigator
    Restrictions
    For Research Use only
  • Format
    Lyophilized
    Reconstitution
    Add 100 µL of distilled water for a final peptide concentration is 1 mg/mL.
    Buffer
    PBS
    Conseil sur la manipulation
    Avoid repeated freeze/thaw cycles.
    Stock
    -20 °C
    Stockage commentaire
    Store at -20 °C long term.
  • Antigène
    CACNB2 (Calcium Channel, Voltage-Dependent, beta 2 Subunit (CACNB2))
    Synonymes
    CACNLB2 Peptide, CAVB2 Peptide, MYSB Peptide, AW060387 Peptide, CAB2 Peptide, Cavbeta2 Peptide, Cchb2 Peptide, Cacnlb2 Peptide, Cacnb2 Peptide, CACNB2 Peptide, CACNB2.2 Peptide, cacnb2a Peptide, im:7141271 Peptide, si:dkey-32m20.2 Peptide, calcium voltage-gated channel auxiliary subunit beta 2 Peptide, calcium channel, voltage-dependent, beta 2 subunit Peptide, calcium channel, voltage-dependent, beta 2b Peptide, CACNB2 Peptide, Cacnb2 Peptide, cacnb2b Peptide
    Sujet
    CACNB2 contributes to the function of the calcium channel by increasing peak calcium current, shifting the voltage dependencies of activation and inactivation, modulating G protein inhibition and controlling the alpha-1 subunit membrane targeting. Defects in CACNB2 are the cause of Brugada syndrome type 4 (BRS4).
    Poids moléculaire
    68 kDa
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