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An analysis of eight structures of mitochondrial bc(1) with bound quinol oxidation (Q(o)) site inhibitors revealed that the presence of inhibitors causes a bidirectional repositioning of the cd1 helix in the cytochrome b subunit.
increased superoxide release from complex III and mitochondrial membrane hyperpolarisation may initiate the cytosolic calcium increase underlying hypoxic pulmonary vasoconstriction
mt-Cytb mutations were studied in a mouse cultured cell model. Complex III assembly was impeded and caused a severe reduction in the amount of Complex I.
The heteroplasmy level of some mutations in gene MT-CYB among women with asymptomatic atherosclerosis
The effects of mutation in mitochondrially encoded cytochrome b are reviewed.
Mitochondrial mutation m.15804T>C in the mtCYB gene in a family with fibromyalgia is associated with NLRP3-inflammasome activation.
These results indicate that miR-151a-5p may participate in the regulation of cellular respiration and ATP production through targeting Cytb.
This study suggests that, in part, polymorphisms in the MT-ATP6 and MT-CYB genes may contribute to the unexpected fertilization failure.
The polymorphisms of CYTB as a very useful DNA marker were significantly different between different geographical Uyghur
Significant elevation of ERalpha and MTCYB transcript levels in premenopausal leiomyomas and its association with ERalpha, -397 CC genotype suggests the mitochondrial-mediated role of estrogen as the promoter of leiomyoma tumorigenesis.
Data show that homoplasmic G6709A (MT-CO1) and G14804A (MT-CYB) alterations cause amino acid changes in the highly conserved residues.
mitocryptide-2 (MCT-2) and its human homolog hMCT-2 are cryptides that activate neutrophils
The other proband had a non-synonymous G15221A mutation in the cytochrome b gene.
In a mitochondrial myopathy patient, a novel stop-codon mutation (G15761A) was found in the mitochondrial DNA (mtDNA)-encoded cytochrome b gene, resulting in loss of the last 41 amino acids of the protein and complex III deficiency
clinical/phenotypic variability of the G15498A mutation in mitochondrial DNA
biochemical and molecular genetic studies of a patient with both muscle and brain involvement and a severe reduction in the activities of both complexes I and III in skeletal muscle due to a novel mutation in the MTCYB gene
caspase 8-dependent cleavage of mitochondrial Cyt b and translocation of its C-terminal half into the cytoplasm occurred during FAS-induced apoptosis in both chicken and human cells
CytbI7T has a role in longevity related to caloric restriction
The m.15635T>C transition (S297P) was carried by a newborn who presented with a polyvisceral failure.
cytochrome B gene mutation induces mitochondrial proliferation and prevents apoptosis in human uroepithelial SV-HUC-1 cells.
Trx2 overexpression modulates the mRNA levels of the COX1 (cytochrome oxidase subunit I) and Cytb (cytochrome b), which are known to be regulated by GR and NF-kappaB.
Component of the ubiquinol-cytochrome c reductase complex (complex III or cytochrome b-c1 complex), which is a respiratory chain that generates an electrochemical potential coupled to ATP synthesis (By similarity).