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anti-Human Cullin 5 Anticorps:
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Human Polyclonal Cullin 5 Primary Antibody pour ICC, IF - ABIN439651
Weems, Slaughter, Unruh, Hall, McLaird, Gilmore, Washburn, Florens, Yasukawa, Aso, Conaway, Conaway: Assembly of the Elongin A Ubiquitin Ligase Is Regulated by Genotoxic and Other Stresses. dans The Journal of biological chemistry 2015
cullin-5 and cullin-2 (Montrer CUL2 Anticorps) play a role in the development of neuromuscular junction and the female germ line of Drosophila
Data show that the follicular epithelium that normally encapsulates single germ line cysts develops aberrantly in cul-5 mutant.
cul-5 plays multiple roles in cell fate specification and synapse formation during Drosophila development.
CUL5 may be an important part of the mechanism for thalidomide-dependent inhibition.
These findings report genetic variants possibly associated with susceptibility to HIV-1 infection (CUL5 rs11212495, rs7103534, rs7117111) and partial viral load control (APOBEC3G (Montrer APOBEC3G Anticorps) rs2294367).
structural model suggests that the diverse FIV and HIV-1 Vifs use conserved residues for Cullin 5 binding, FIV Vif (Montrer BTG1 Anticorps) binds Cullin 5 independently of zinc, in contrast to HIV-1 Vif (Montrer BTG1 Anticorps).
these findings identify Cul-5 as a signaling component that connects an LPS (Montrer IRF6 Anticorps)-activated TLR4 (Montrer TLR4 Anticorps)-MyD88 (Montrer MYD88 Anticorps) complex to TRAF6 (Montrer TRAF6 Anticorps) for efficient activation of NF-kappaB (Montrer NFKB1 Anticorps)
CUL5 neddylation may allosterically tune polyubiquitin (Montrer UBB Anticorps) chain length and topology.
Study found that CUL5 expression was significantly decreased in both endometrioid adenocarcinomas with the more aggressive serous type displaying a higher reduction. Its mRNA and protein overexpression in endometrial cancer cells resulted in decreased cell proliferation.
Studies indicate that Cullin-RING E3 ubiquitin ligases (CRL (Montrer IL31RA Anticorps)) are key players of the ubiquitylation pathway.
These results provide important information on the assembly of the Vif (Montrer BTG1 Anticorps)-CUL5-E3 ubiquitin ligase (Montrer MUL1 Anticorps) and identify a new viV binding interface with CBF-beta (Montrer CBFB Anticorps) at the C-terminus of HIV-1 Vif (Montrer BTG1 Anticorps).
Data indicate that cell surface adhesion molecules VCAM-1 (Montrer VCAM1 Anticorps) and P-selectin (Montrer SELP Anticorps) play some roles in mechanical stretch-induced HL-60 cell adhesion to mouse common carotid arteries.
N-terminal mutants of HIV-1 vif (Montrer BTG1 Anticorps) that demonstrated reduced Cul5 binding were also unable to degrade APOBEC3G (Montrer APOBEC3G Anticorps) as well as APOBEC3F (Montrer APOBEC3F Anticorps).
neural precursor cell expressed developmentally downregulated (Montrer NEDD8 Anticorps) protein 8 (Nedd8 (Montrer NEDD8 Anticorps)) modification of Cul-5 is required for its interaction with TRAF6 (Montrer TRAF6 Anticorps).
Our results show that endogenous Cul5 suppresses epithelial cell transformation by several pathways, including inhibition of Src (Montrer SRC Anticorps)-Cas (Montrer CTNND1 Anticorps)-induced ruffling through SOCS6 (Montrer SOCS6 Anticorps)
The expression of cullin5 on retinal cells and reduction in the percentage of dead retinal cells induced by L-glutamate (Montrer GRIN1 Anticorps) suggest that cullin5 has neuroprotective properties in retinal cells.
SOCS (Montrer CISH Anticorps) box acts as an independent binding domain capable of recruiting elonginBC and cullin5 to promote E3 ligase formation.
The role of Cul5 is to degrade disabled homolog 1 (Dab1 (Montrer DAB1 Anticorps)) and thus control neuron migration speed and neuron insertion at the top of the cortical plate.
These results suggest that, when Src (Montrer SRC Anticorps) is activated by mutation or physiological mechanisms, its effects are limited by Culllin 5, which downregulates active Src (Montrer SRC Anticorps) and its phosphorylated substrates.
data imply the existence of two distinct subclasses of SOCS (Montrer CISH Anticorps) proteins with a high affinity for Cullin5, the E3 ligase scaffold, possibly reflecting complete dependence upon ubiquitination for suppression of cytokine signalling
The study results suggest that VACM-1 may regulate endothelial AQP1 concentration both in vivo and in vitro.
oocyte maturation from pachytene exit and MPK-1 (Montrer MAPK1 Anticorps) activation are redundantly controlled by the RBX-2-CUL-5- and RBX-1 (Montrer RBX1 Anticorps)-CUL-2 (Montrer CUL2 Anticorps)-based complexes.
neuropeptide arginine vasopressin receptor\; may have a role in the central nervous system
Vasopressin-activated calcium-mobilizing receptor (VACM-1)
, cullin 5
, Vasopressin-activated calcium-mobilizing receptor-1
, Cullin-5 (vasopressin-activated calcium-mobilizing receptor-1)
, vasopressin-activated calcium-mobilizing receptor 1
, vasopressin-activated calcium-mobilizing receptor protein
, vasopressin-activated calcium-mobilizing protein