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Chicken Polyclonal SOD2 Primary Antibody pour ELISA, ICC - ABIN151490
Dumont, Wille, Stack, Calingasan, Beal, Lin: Reduction of oxidative stress, amyloid deposition, and memory deficit by manganese superoxide dismutase overexpression in a transgenic mouse model of Alzheimer's disease. dans FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2009
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Rat (Rattus) Polyclonal SOD2 Primary Antibody pour WB - ABIN152304
Siu, Alway: Id2 and p53 participate in apoptosis during unloading-induced muscle atrophy. dans American journal of physiology. Cell physiology 2005
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Human Polyclonal SOD2 Primary Antibody pour IF (p), IHC (p) - ABIN737361
Khan, Chen, Wan, Tania, Xu, Chen, Zhang: Regulatory effects of resveratrol on antioxidant enzymes: a mechanism of growth inhibition and apoptosis induction in cancer cells. dans Molecules and cells 2013
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Human Polyclonal SOD2 Primary Antibody pour IHC (fro), IHC (p) - ABIN151468
Dumont, Stack, Elipenahli, Jainuddin, Gerges, Starkova, Yang, Starkov, Beal: Behavioral deficit, oxidative stress, and mitochondrial dysfunction precede tau pathology in P301S transgenic mice. dans FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2011
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Rat (Rattus) Polyclonal SOD2 Primary Antibody pour WB - ABIN152303
Shi, So, Man, Vanhoutte: Oxygen-derived free radicals mediate endothelium-dependent contractions in femoral arteries of rats with streptozotocin-induced diabetes. dans British journal of pharmacology 2007
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Human Polyclonal SOD2 Primary Antibody pour IHC, IHC (p) - ABIN4355070
Lindfors, Nilsson, Garcia-Roves, Zuberi, Karimi, Donahue, Roopenian, Mulder, Uhlén, Ekström, Davisson, Hökfelt, Schalling, Johansen: Hypothalamic mitochondrial dysfunction associated with anorexia in the anx/anx mouse. dans Proceedings of the National Academy of Sciences of the United States of America 2011
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Chicken Polyclonal SOD2 Primary Antibody pour IP, IHC - ABIN361648
Adachi, Ohta, Yamada, Futenma, Kato, Hirano: Quantitative analysis of extracellular-superoxide dismutase in serum and urine by ELISA with monoclonal antibody. dans Clinica chimica acta; international journal of clinical chemistry 1993
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Chicken Polyclonal SOD2 Primary Antibody pour ICC, IF - ABIN361656
Gao, Flores, Leff, Bose, McCord: Synthesis and anti-inflammatory activity of a chimeric recombinant superoxide dismutase: SOD2/3. dans American journal of physiology. Lung cellular and molecular physiology 2003
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Human Polyclonal SOD2 Primary Antibody pour ICC, IF - ABIN4355072
Davis, Collier, Leonardo, Seifert, Ajmo, Pennypacker: Leukemia Inhibitory Factor Protects Neurons from Ischemic Damage via Upregulation of Superoxide Dismutase 3. dans Molecular neurobiology 2016
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Mouse (Murine) Polyclonal SOD2 Primary Antibody pour ICC, IHC - ABIN1078543
Zhao, Li, Han, Yue, Shi, Wang, Guo, Lu: DDAH1 deficiency promotes intracellular oxidative stress and cell apoptosis via a miR-21-dependent pathway in mouse embryonic fibroblasts. dans Free radical biology & medicine 2016
Prion (Montrer PRNP Anticorps) propagation exacerbates an apoptotic pathway whereby mitochondrial dysfunction follows mislocalisation of SOD2 to cytosolic caspases permitting its degradation.
Depletion of superoxide dismutases promotes muscular and neuronal ROS (Montrer ROS1 Anticorps) accumulation which may have a significant effect on age-dependent impairment of the Drosophila adults.
Paraquat exposure, but not Sod2 knockdown, resulted in increased carbonylated protein relative abundance.
The functional SOD1 (Montrer SOD1 Anticorps) and SOD2 genes knockout and their overexpression in neurons and glial tissue increase the sensitivity of Drosophila melanogaster to oxidative stress conditions.
ocytes lacking maternal SOD2 protein develop into adults just like normal SOD2-containing oocytes suggesting that maternal SOD2-mediated protection against mitochondrial ROS (Montrer ROS1 Anticorps) is not essential for oocyte viability
Muscles appear to be more sensitive to superoxide attack relative to the neurons and such overt phenotypes observed in SOD2-deficient animals can be directly attributed to the muscle.
Overexpression of Cu,ZnSOD (Montrer SOD1 Anticorps) and MnSOD in transgenic Drosophila.
Ablation of mitochondrial SOD2 through expression of a GAL4 (Montrer LGALS4 Anticorps)-regulated, inverted-repeat Sod2 RNA-interference causes increased endogenous oxidative stress, loss of respiratory chain & TCA cycle components,& early-onset mortality in young adults.
Effects of overexpression of copper-zinc and manganese superoxide dismutases, catalase, and thioredoxin reductase genes on longevity.
The chromosomal deficiency Df(2R)017 significantly up-regulated MnSOD mRNA by 1.7-fold. Deficiency in four other genomic intervals, Df(1)ct-J4, Df(2L)BSC4, Df(3L)66C-G28 and Df(3R)Scr (Montrer SCRIB Anticorps), down-regulated MnSOD expression.
SOD1 (Montrer SOD1 Anticorps) and SOD2 provide independent protection to compartment-specific protein iron-sulfur clusters against attack by superoxide generated under oxidative stress
review: discuss how H2O2 formation in mitochondria depends on and is controlled by MnSOD
SOD2 gene polymorphism may modulate biochemical responses to a 12-week swimming training.
Study shows an association of the polymorphism of MnSOD 47C/T in the Algerian population with type 1 diabetes without complications.
MnSOD is the major superoxide scavenger in mitochondria, whose activity is regulated by SIRT3 (Montrer SIRT3 Anticorps)-mediated deacetylation, particularly at the Lys68 site, and can be enhanced by a SIRT3 (Montrer SIRT3 Anticorps) coumarin derivative.
Heavy alcohol drinking down-regulates ALDH2 (Montrer ALDH2 Anticorps) gene expression level. Heavy smoking up-regulates SOD2 gene expression level in patients with head and neck squamous cell carcinoma.
This is the first report evaluating the role of SOD2 in native and T351-mutated BCR-ABL (Montrer ABL1 Anticorps)-expressing cells and in a large cohort of chronic myeloid leukemia (Montrer BCL11A Anticorps) patients. In leukemic cells silenced for SOD2 expression a specific down-regulation of the expression of PRDX2 (Montrer PRDX2 Anticorps) gene was found.
The investigated MnSOD and Catalase (Montrer CAT Anticorps) polymorphisms do not predispose to the development of alcoholic Chronic Pancreatitis.
Allele-specific interaction between GPX1 (Montrer GPX1 Anticorps) and MnSOD affects the levels of Bcl2 (Montrer BCL2 Anticorps), Sirt3 (Montrer SIRT3 Anticorps) and E-cadherin (Montrer CDH1 Anticorps).
Studies suggest that both SOD3 (Montrer SOD3 Anticorps) and SOD2 superoxide dismutases are regulated by oxidative stress and redox-dependent signaling mechanisms.
Data confirmed that SOD2 is important in the development and prognosis of salivary adenoid cystic carcinoma (SACC); that SOD2 deregulation is related to migration and invasion in SACC; and that the SOD2-dependent production of intracellular H2O2 promotes the migration and invasion of SACC, which involves the ERK-Slug signaling pathway.
The results showed that 60-min ischemia of the porcine uterus conducted at the mid-secretory estrous phase caused decreased HIF-1alpha (Montrer HIF1A Anticorps) and increased SOD-2 gene expression.
show that ischemia markedly potentiated the expression of arginase-1 (Montrer ARG1 Anticorps), and also induced the SOD2 in the wound tissue.
Exposure to follicular fluid transiently increased the transcript levels of IL8 (Montrer IL8 Anticorps) and PTGS2 (Montrer PTGS2 Anticorps), and decreased the expression of SOD2, GPX3 (Montrer GPX3 Anticorps), DAB2 (Montrer DAB2 Anticorps), and NR3C1 (Montrer NR3C1 Anticorps). TNF (Montrer TNF Anticorps) and IL6 (Montrer IL6 Anticorps) levels were also decreased while those of NAMPT (Montrer NAMPT Anticorps) were unaffected.
Data suggest that during ectogenesis of blastocysts infected with bovine Herpesvirus type 5, expression of SOD2 (Mn) remains high indicating intense mitochondrial activity; this effect may be involved in inhibition of apoptosis in infected embryos.
expression profile of SOD2 in follicles: oocytes (SOD2 restricted to ooplasm (Montrer NLRP5 Anticorps)); cumulus cells (expressed some SOD2); follicular fluid (small follicles show increased amounts of SOD2 in comparison with large follicles)
Shear stress influences spatial variations in vascular Mn-SOD expression with implications for LDL nitration.
The expression of SOD1 (Montrer SOD1 Anticorps) and SOD2 through the course of the estrous cycle is reported.
Heart mitochondrial nucleoids contained SOD2, aortic endothelial cell mitochondrial nucleoids did not
SOD2 expression is ATM (Montrer ATM Anticorps)- and RelA (Montrer NFkBP65 Anticorps)-dependent, ATM (Montrer ATM Anticorps) knockdown renders cells sensitive to pro-oxidant exposure, and SOD mimetics partially rescue this sensitivity. Mice with germline deletion of Atm (Montrer ATM Anticorps) fail to develop mature mammary glands, but using a conditional knockout approach, we determined that Atm (Montrer ATM Anticorps) deletion significantly diminished the expression of Sod2.
Enhanced mitochondrial oxidative stress under hyperlipidemic conditions in aging induces plaque instability, in part by increasing smooth muscle cell apoptosis, necrotic core expansion, and matrix degradation in Sod2/ApoE (Montrer APOE Anticorps) knockout mice.
Deletion of SOD2 increases mitochondrial reactive oxygen species but does not influence platelet function.
This study reveals a novel model regulating cardiomyocyte apoptosis which is composed of miR (Montrer MLXIP Anticorps)-23a and MnSOD.
data showed that Klotho (Montrer KL Anticorps) protects Tac (Montrer IL2RA Anticorps)-induced oxidative stress by negatively regulating the PI3K/AKT (Montrer AKT1 Anticorps) pathway and subsequently enhancing FoxO3a (Montrer FOXO3 Anticorps)-mediated MnSOD expression.
Moderate MnSOD and/or catalase (Montrer CAT Anticorps) overexpression in desmin (Montrer DES Anticorps)-null hearts leads to a marked decrease in intracellular reactive oxygen species, ameliorates mitochondrial and other ultrastructural defects, minimizes myocardial degeneration and leads to a significant improvement of cardiac function.
this study shows that differential protein acetylation assists import of excess SOD2 into mitochondria and mediates SOD2 aggregation associated with cardiac hypertrophy in the murine SOD2-tg heart
SOD2 overexpression blocks maternal diabetes-induced oxidative stress and ER stress, and reduces the incidence of NTDs in embryos exposed to maternal diabetes
the regulation of mitochondrial ROS (Montrer ROS1 Anticorps) by SOD2 and Sirt3 (Montrer SIRT3 Anticorps) plays an important role in fine-tuning the Osteoclast differentiation program.
treatment with Mito-TEMPO, a mitochondrial-specific superoxide scavenger, recovered mitochondrial fission-fusion imbalance and blunted mitochondrial superoxide production, and reduced the IDH2 (Montrer IDH2 Anticorps) knockdown-induced decrease in MnSOD expression, eNOS (Montrer NOS3 Anticorps) phosphorylation and NO production in endothelial cells
This gene is a member of the iron/manganese superoxide dismutase family. It encodes a mitochondrial protein that forms a homotetramer and binds one manganese ion per subunit. This protein binds to the superoxide byproducts of oxidative phosphorylation and converts them to hydrogen peroxide and diatomic oxygen. Mutations in this gene have been associated with idiopathic cardiomyopathy (IDC), premature aging, sporadic motor neuron disease, and cancer. Alternate transcriptional splice variants, encoding different isoforms, have been characterized.
, manganese superoxide dismutase
, Mn Sod
, Mn superoxide dismutase
, superoxide dismutase 2, mitochondrial
, superoxide dismutase [Mn], mitochondrial
, Mn-superoxide dismutase
, mitochodnrial SOD
, mitochondrial Mn-superoxide dismutase 2
, mitochondrial MnSOD
, mitochondrial superoxide dismutase 2
, superoxide dismutase
, superoxide dismutase 2
, superoxide dismutase 2 (Mn)
, superoxide dismutase-2
, indophenoloxidase B
, manganese-containing superoxide dismutase
, mangano-superoxide dismutase
, superoxide dismutase (Mn type)
, manganous superoxide dismutase
, manganese SOD
, Superoxide dimutase 2, mitochondrial