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anti-Human MAPK3 Anticorps:
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Human Polyclonal MAPK3 Primary Antibody pour WB - ABIN1881527
Munshi, Wu, Mukhopadhyay, Ottaviano, Sassano, Koblinski, Platanias, Stack et al.: Differential regulation of membrane type 1-matrix metalloproteinase activity by ERK 1/2- and p38 MAPK-modulated tissue inhibitor of metalloproteinases 2 expression controls transforming growth ... dans The Journal of biological chemistry 2004
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Human Polyclonal MAPK3 Primary Antibody pour ICC, IHC (p) - ABIN3044377
Li, Zhu, Liu, Liu, Wang, Xiong, Shen, Hu, Zheng: ZFX knockdown inhibits growth and migration of non-small cell lung carcinoma cell line H1299. dans International journal of clinical and experimental pathology 2013
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Chicken Monoclonal MAPK3 Primary Antibody pour IF, IP - ABIN967700
Ackerley, Grierson, Brownlees, Thornhill, Anderton, Leigh, Shaw, Miller: Glutamate slows axonal transport of neurofilaments in transfected neurons. dans The Journal of cell biology 2000
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Chicken Monoclonal MAPK3 Primary Antibody pour IF, IP - ABIN967701
Aguirre-Ghiso, Liu, Mignatti, Kovalski, Ossowski: Urokinase receptor and fibronectin regulate the ERK(MAPK) to p38(MAPK) activity ratios that determine carcinoma cell proliferation or dormancy in vivo. dans Molecular biology of the cell 2001
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Chicken Monoclonal MAPK3 Primary Antibody pour IF, IP - ABIN967952
Boulton, Cobb: Identification of multiple extracellular signal-regulated kinases (ERKs) with antipeptide antibodies. dans Cell regulation 1991
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Chicken Polyclonal MAPK3 Primary Antibody pour ICC, FACS - ABIN361833
Boulton, Gregory, Cobb: Purification and properties of extracellular signal-regulated kinase 1, an insulin-stimulated microtubule-associated protein 2 kinase. dans Biochemistry 1991
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Human Polyclonal MAPK3 Primary Antibody pour WB - ABIN2801963
McLaughlin, Kumar, McDonnell, Van Horn, Lee, Livi, Young: Identification of mitogen-activated protein (MAP) kinase-activated protein kinase-3, a novel substrate of CSBP p38 MAP kinase. dans The Journal of biological chemistry 1996
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Mouse (Murine) Polyclonal MAPK3 Primary Antibody pour IHC, WB - ABIN3020725
Fan, Zhang, Hu, Li, Zhang: Activation of AKT/ERK confers non-small cell lung cancer cells resistance to vinorelbine. dans International journal of clinical and experimental pathology 2014
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Human Polyclonal MAPK3 Primary Antibody pour IF (p), IHC (p) - ABIN744143
Zhao, Zhang, Liu, Zhang, Hao, Li, Chen, Shen, Tang, Min, Meng, Wang, Yi, Zhang: Hydrogen Sulfide and/or Ammonia Reduces Spermatozoa Motility through AMPK/AKT Related Pathways. dans Scientific reports 2016
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Human Monoclonal MAPK3 Primary Antibody pour IP, WB - ABIN967398
Rossomando, Payne, Weber, Sturgill: Evidence that pp42, a major tyrosine kinase target protein, is a mitogen-activated serine/threonine protein kinase. dans Proceedings of the National Academy of Sciences of the United States of America 1989
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Apocynin may act as a novel molecular candidate to protect against vascular smooth muscle cell osteogenic switching/vascular calcification through suppressing ERK1/2 (Montrer MAPK1/3 Anticorps) pathway.
GHRH (Montrer GHRH Anticorps) agonist MR-409 increase phosphorylation of AKT (Montrer AKT1 Anticorps) and ERK1/2 (Montrer MAPK1/3 Anticorps) in dermal fibroblasts.
telomere stability is under direct control of one of the major pro-oncogenic signaling pathways (RAS/RAF (Montrer RAF1 Anticorps)/MEK (Montrer MAP2K1 Anticorps)/ERK (Montrer EPHB2 Anticorps)) via TRF2 (Montrer TERF2 Anticorps) phosphorylation.
Data suggest that inability of lithium, an anti-manic agent, to regulate circadian rhythms in cells from patient with bipolar disorder reflects reduced ERK1/2 (Montrer MAPK1/3 Anticorps) activity and MAP kinase (Montrer MAPK1 Anticorps) signaling through ELK1 (Montrer ELK1 Anticorps). (ERK (Montrer EPHB2 Anticorps) = extracellular signal-related kinase; ELK1 (Montrer ELK1 Anticorps) = ETS-domain protein (Montrer ELK3 Anticorps) ELK1 (Montrer ELK1 Anticorps))
Report differential expression of EZH2 (Montrer EZH2 Anticorps) protein in small cell and aggressive B-cell non-Hodgkin lymphomas and differential regulation of EZH2 (Montrer EZH2 Anticorps) expression by p-ERK1/2 (Montrer MAPK1/3 Anticorps) and MYC (Montrer MYC Anticorps) in aggressive B-cell lymphomas.
Data indicate that PD0325901 inhibited extracellular signal-regulated kinases ERK1/2 (Montrer MAPK1/3 Anticorps) phosphorylation.
Findings suggest that ERK1/2 (Montrer MAPK1/3 Anticorps)-mediated Cdk2 (Montrer CDK2 Anticorps)/cyclin A (Montrer CCNA2 Anticorps) signaling pathway is involved in 7-hydroxy-5,4'-dimethoxy-2-arylbenzofuran (Ary) - induced G1/S-phase arrest.
Endoplasmic reticulum stress contributes to nefazodone-induced toxicity in HepG2 cells and ERK1/2 (Montrer MAPK1/3 Anticorps) signaling pathway plays an important role.
ERK1 Directly Interacts With JNK1 (Montrer MAPK8 Anticorps) Leading to Regulation of JNK1 (Montrer MAPK8 Anticorps)/c-Jun (Montrer JUN Anticorps) Activity and Cell Transformation.
egulation of apoptosis was far more sensitive than regulation of proliferation. IGF1 (Montrer IGF1 Anticorps) and insulin (Montrer INS Anticorps) activated PKB (Akt/PKB (Montrer AKT1 Anticorps)) rapidly and consistently maintained its phosphorylation. Activation of ERK1/2 (Montrer MAPK1/3 Anticorps) was only observed in response to IGF1 (Montrer IGF1 Anticorps).
North American ginseng inhibits myocardial NOX2 (Montrer CYBB Anticorps)-ERK1/2 (Montrer MAPK1/3 Anticorps)-TNF-alpha (Montrer TNF Anticorps) signaling pathway and improves cardiac function in endotoxemia, suggesting that NA ginseng may have the potential in the prevention of clinical sepsis.
NF-alpha1 is critical for regulating antiproliferation and cell fate determination, through differentiating embryonic stem cells to GFAP (Montrer GFAP Anticorps)-positive astrocytes for normal neurodevelopment.
These findings suggested that USP14 induces NF-kappaB (Montrer NFKB1 Anticorps) activity and ERK1/2 (Montrer MAPK1/3 Anticorps) phosphorylation triggered by microbial infection.
Cortical neuron-specific deletion of extracellular signal-regulated kinases Erk1 or Erk2 (Montrer MAPK1 Anticorps) significantly increased the duration of wakefulness.
pERK1/2 is a regulator of CD44 (Montrer CD44 Anticorps) expression, and increased CD44 (Montrer CD44 Anticorps) expression leads to a pro-sclerotic and migratory parietal epithelial cell phenotype in focal segmental glomerulosclerosis.
mmLDL increased the serum concentrations and expression of ICAM-1 (Montrer ICAM1 Anticorps) and VCAM-1 (Montrer VCAM1 Anticorps) by activating the ERK1/2 (Montrer MAPK1/3 Anticorps) pathway, resulting in the expression of ETB (Montrer EDNRB Anticorps) receptors and the enhancement of contractile function in vascular smooth muscle.
Angiotensin II regulates dendritic cells through activation of p65 NF-kappaB (Montrer NFkBP65 Anticorps), ERK1, ERK2 (Montrer MAPK1 Anticorps) and STAT1 (Montrer STAT1 Anticorps) pathways.
MAPK3/1 participates in primordial follicle activation through mTORC1-KITL (Montrer KITLG Anticorps) signaling.
At low oxLDL levels LOX-1 (Montrer OLR1 Anticorps) activates the protective Oct-1 (Montrer POU2F1 Anticorps)/SIRT1 (Montrer SIRT1 Anticorps) pathway, while at higher levels of the lipoprotein switches to the thrombogenic ERK1/2 (Montrer MAPK1/3 Anticorps) pathway.
Studies indicate that progesterone receptor (Montrer PGR Anticorps) transgenic (Pgrcre/+) mitogen inducible gene 6 (Mig (Montrer CXCL9 Anticorps)-6over) phosphatase and tensin homolog (Montrer PTEN Anticorps) protein (Ptenf/f) knockout mice exhibited an increase of phospho-ERK1/2 (Montrer MAPK1/3 Anticorps) and its target genes.
ERK1/2 (Montrer MAPK1/3 Anticorps)-Akt1 (Montrer AKT1 Anticorps) crosstalk regulates arteriogenesis in mice and zebrafish.
eena (Montrer SH3GL1 Anticorps) plays an important role in the development of the myeloid cell through activation of the ERK1/ERK2 (Montrer MAPK1 Anticorps) pathway
ERK1 and ERK2 (Montrer MAPK1 Anticorps) target common and distinct gene sets, confirming diverse roles for these kinases during embryogenesis; for ERK1 different specific genes involved in dorsal-ventral patterning and subsequent embryonic cell migration were identified.
These results demonstrate that induction of Hsp70 (Montrer HSPA1A Anticorps) in response to heat stress is dependent on ERK (Montrer MAPK1 Anticorps) activation in Pac2 (Montrer PSMG2 Anticorps) cells.
Data define distinct roles for ERK1 and ERK2 (Montrer MAPK1 Anticorps) in developmental cell migration processes during zebrafish embryogenesis.
MAPK3/1 is involved in luteinizing hormone-mediated decrease of C-type natriuretic peptide (Montrer NPPC Anticorps) and this process is related to the EGFR (Montrer EGFR Anticorps) and MAPK3/1 signal pathways
Chronic hypoxia induces Egr-1 (Montrer EGR1 Anticorps) via activation of ERK1/2 (Montrer MAPK1/3 Anticorps) and contributes to pulmonary vascular remodeling.
ER Ca(2 (Montrer CA2 Anticorps)+) release enhances eNOS (Montrer NOS3 Anticorps) Ser (Montrer SIGLEC1 Anticorps)-635 phosphorylation and function via ERK1/2 (Montrer MAPK1/3 Anticorps) activation.
Thrombospondin 1 (Montrer THBS1 Anticorps), fibronectin (Montrer FN1 Anticorps), and vitronectin (Montrer VTN Anticorps) are differentially dependent upon RAS, ERK1/2 (Montrer MAPK1/3 Anticorps), and p38 (Montrer MAPK14 Anticorps) for induction of vascular smooth muscle cell chemotaxis.
results suggest that Nav1.7-Ca2+ influx-protein kinase C-alpha pathway activated ERK1/ERK2 and p38, which increased phosphorylation of glycogen synthase kinase-3beta, decreasing tau phosphorylation
These data suggest that Gab1 (Montrer GAB1 Anticorps)-ERK1/2 (Montrer MAPK1/3 Anticorps) binding and their nuclear translocation play a crucial role in Egr-1 (Montrer EGR1 Anticorps) nuclear accumulation.
data demonstrate that hypoxia-induced adventitial fibroblast proliferation requires activation and interaction of PI3K, Akt (Montrer AKT1 Anticorps), mTOR (Montrer FRAP1 Anticorps), p70S6K (Montrer RPS6KB1 Anticorps), and ERK1/2 (Montrer MAPK1/3 Anticorps).
This study demonstrates for the first time that cyclic mechanical stretch induces the proliferation of bovine satellite cells and suppresses their myogenic differentiation through the activation of ERK (Montrer MAPK1 Anticorps).
findings indicate that exposure to DHEA, at concentrations found in human blood, causes vascular endothelial proliferation by a plasma membrane-initiated activity that is Gi/o and ERK1/2 (Montrer MAPK1/3 Anticorps) dependent.
Results suggest that estrogen receptors and the ERK1/2 (Montrer MAPK1/3 Anticorps) signaling pathway are involved in the anti-apoptotic action of LY117018 in vascular endothelial cells.
Early activation of MAPK p44/42 is involved in deoxynivalenol -induced disruption of intestinal barrier function and tight junction network signaling.
Pseudorabies virus glycoprotein gE-mediated ERK 1/2 phosphorylation also occurs in epithelial cells and in these cells, gE-mediated ERK 1/2 signaling is associated with degradation of the pro-apoptotic protein Bim (Montrer BCL2L11 Anticorps).
Treatment with ERK (Montrer MAPK1 Anticorps) inhibitors or ERK1/2 (Montrer MAPK1/3 Anticorps) knockdown significantly suppressed porcine epidemic diarrhea virus progeny production.
This study reveals a new function of the gE glycoprotein of pseudorabies virus and suggests that pseudorabies virus, through activation of ERK1/2 (Montrer MAPK1/3 Anticorps) signaling, has a substantial impact on T cell behavior.
CSF2 (Montrer CSF2 Anticorps) stimulates proliferation of trophectoderm cells by activation of the PI3K-and ERK1/2 (Montrer MAPK1/3 Anticorps) MAPK (Montrer MAPK1 Anticorps)-dependent MTOR (Montrer FRAP1 Anticorps) signal transduction cascades.
PGRN (Montrer GRN Anticorps) inhibits adipogenesis in porcine preadipocytes partially through ERK (Montrer MAPK1 Anticorps) activation mediated PPARgamma (Montrer PPARG Anticorps) phosphorylation.
Porcine circovirus type 2 (PCV2) might induce autophagy via the AMPK (Montrer PRKAA1 Anticorps)/ERK (Montrer MAPK1 Anticorps)/TSC2 (Montrer TSC2 Anticorps)/mTOR (Montrer FRAP1 Anticorps) signaling pathway in the host cells, representing a pivotal mechanism for PCV2 pathogenesis
Data show that proinflammatory cytokines induction was ERK1/2 (Montrer MAPK1/3 Anticorps) and JNK1 (Montrer MAPK8 Anticorps)/2 dependent.
Saccharomyces cerevisiae inhibits the Enterotoxigenic Escherichia coli-induced expression of pro-inflammatory transcripts and this inhibition was associated to a decrease of ERK1/2 (Montrer MAPK1/3 Anticorps) and p38 MAPK (Montrer MAPK14 Anticorps) phosphorylation
ERK1 phosphorylation in response to Insulin-like Growth Factor-1 (Montrer IGF1 Anticorps) does not require activation of the Insulin-like Growth Factor-1 receptor tyrosine kinase (Montrer IGF1R Anticorps)
The results suggest that the MPK-1 (Montrer MAPK1 Anticorps)/ERK (Montrer MAPK1 Anticorps) regulatory network, including FBF-1 (Montrer FBF1 Anticorps), FBF-2, and LIP-1 (Montrer CENPJ Anticorps), controls the number of sperm by regulating the timing of the sperm-oocyte switch in C. elegans.
Cek2 (Montrer FGFR3 Anticorps) has a cryptic role in cell-wall biogenesis and its role is not entirely redundant to Cek1.
knockdown of SUV420H1 (Montrer SUV420H1 Anticorps) reduced phosphorylated ERK1 and total ERK1 proteins, and interestingly suppressed ERK1 at the transcriptional level
Secreted aspartic protease-mediated proteolytic cleavage of Msb2 is required for activation of the Cek1 mitogen activated protein kinase (Montrer MAPK1 Anticorps) pathway in response to environmental cues.
The authors propose that a Msb2, Cek1 and Ace2 signalling pathway addresses PMT genes as downstream targets and that different modes of regulation have evolved for PMT1 and PMT2/PMT4 genes.
Msb2 is involved in the transmission of the signal toward Cek1 mediated by the Cdc42 (Montrer CDC42 Anticorps) GTPase (Montrer RACGAP1 Anticorps).
constitutively active (CA)-MPK3 crosses with summ1 and summ2, two known suppressors of mpk4 (Montrer MAPK4 Anticorps), resulted in a partial reversion of the CA-MPK3 phenotypes.
that MPK3/MPK6 (Montrer MAPK6 Anticorps) phosphorylate and destabilize ICE1, which negatively regulates CBF (Montrer CEBPZ Anticorps) expression and freezing tolerance in plants
Changes in PUB22 Ubiquitination Modes Triggered by MITOGEN-ACTIVATED PROTEIN KINASE3 Dampen the Immune Response
MPK3 role in ultraviolet induced stomatal closure
Study propose that the pathogen-responsive MPK3/MPK6 (Montrer MAPK6 Anticorps) cascade and ABA are two essential signaling pathways that control, respectively, the organic acid metabolism and ion channels, two main branches of osmotic regulation in guard cells that function interdependently to control stomatal opening/closure.
Data report that MPK3/MPK6 and their substrate ERF6 promote the biosynthesis of IGSs and the conversion of I3G to 4MI3G, a target of PEN2/PEN3-dependent chemical defenses in plant immunity.
Data show that the protein kinases MPK3 and MPK6 (Montrer MAPK6 Anticorps) can both interact with SPOROCYTELESS/NOZZLE (SPL (Montrer SGPL1 Anticorps)) in vitro and in vivo and can phosphorylate the SPL (Montrer SGPL1 Anticorps) protein in vitro.
MKK4 (Montrer MAP2K4 Anticorps), MKK5 (Montrer MAP2K5 Anticorps), MKK7 (Montrer MAP2K7 Anticorps), and MKK9, are responsible for the activation of MPK3 and MPK6 (Montrer MAPK6 Anticorps) by melatonin, indicating that melatonin-mediated innate immunity is triggered by MAPK (Montrer MAPK1 Anticorps) signaling through MKK4 (Montrer MAP2K4 Anticorps)/5/7/9-MPK3/6 cascades.
Phosphatase AP2C1, as well as AP2C1-targeted MPK3 and MPK6 (Montrer MAPK6 Anticorps), are important regulators of plant-nematode interaction, where the co-ordinated action of these signalling components ensures the timely activation of plant defence.
Results demonstrated the contribution of MPK3 and MPK6 (Montrer MAPK6 Anticorps) to riboflavin-induced resistance.
The protein encoded by this gene is a member of the MAP kinase family. MAP kinases, also known as extracellular signal-regulated kinases (ERKs), act in a signaling cascade that regulates various cellular processes such as proliferation, differentiation, and cell cycle progression in response to a variety of extracellular signals. This kinase is activated by upstream kinases, resulting in its translocation to the nucleus where it phosphorylates nuclear targets. Alternatively spliced transcript variants encoding different protein isoforms have been described.
MAP kinase isoform p44
, MAPK 1
, extracellular signal-regulated kinase 1
, extracellular signal-related kinase 1
, insulin-stimulated MAP2 kinase
, microtubule-associated protein 2 kinase
, MAP kinase 3
, p44 MAP kinase
, pp42/MAP kinase
, mitogen-activated protein kinase 3
, MAP kinase 12
, MAPK 12
, extracellular signal-regulated kinase 6
, mitogen-activated protein kinase 12
, stress-activated protein kinase 3
, MAP kinase 1
, MAPK 3
, mitogen-activated 3
, mitogen-activated protein kinase 1
, extracellular signal-regulated kinase-1
, likely protein kinase