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anti-Mouse (Murine) IL1B Anticorps:
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Human Monoclonal IL1B Primary Antibody pour CyTOF, FACS - ABIN4899224
Yates, Burgess, Kocsis-Angle, Antal, Dority, Embury, Piotrkowski, Brunden: Amyloid beta and amylin fibrils induce increases in proinflammatory cytokine and chemokine production by THP-1 cells and murine microglia. dans Journal of neurochemistry 2000
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Human Monoclonal IL1B Primary Antibody pour CyTOF, FACS - ABIN4899223
Oliver, Lovric, Yu, Christou, Aiken, Cooper, Walsh: Development of a Novel Model for the Assessment of Dead-Space Management in Soft Tissue. dans PLoS ONE 2015
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Bacteria Polyclonal IL1B Primary Antibody pour ELISA, EM - ABIN269770
Petrovski, Ayna, Majai, Hodrea, Benko, Mádi, Fésüs: Phagocytosis of cells dying through autophagy induces inflammasome activation and IL-1β release in human macrophages. dans Autophagy 2011
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Dog (Canine) Polyclonal IL1B Primary Antibody pour IF (p), IHC (p) - ABIN728503
Zhong, Shu, Wang, Luo, Zhong, Ran, Zheng, Yin, Ling: Enhanced homing of mesenchymal stem cells to the ischemic myocardium by ultrasound-targeted microbubble destruction. dans Ultrasonics 2011
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Dog (Canine) Polyclonal IL1B Primary Antibody pour IHC, ELISA - ABIN1582289
Choi, Lim, Hwang, Lee, Cho, Kim: Anti-ischemic and anti-inflammatory activity of (S)-cis-verbenol. dans Free radical research 2010
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Mouse (Murine) Polyclonal IL1B Primary Antibody pour Neut, WB - ABIN223538
Scott, Ma, Viriyakosol, Terkeltaub, Liu-Bryan: Engagement of CD14 mediates the inflammatory potential of monosodium urate crystals. dans Journal of immunology (Baltimore, Md. : 1950) 2006
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Human Polyclonal IL1B Primary Antibody pour IHC (p), IHC - ABIN446969
Omran, Peng, Zhang, Xiang, Xue, Gan, Kong, Yin: Interleukin-1β and microRNA-146a in an immature rat model and children with mesial temporal lobe epilepsy. dans Epilepsia 2012
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Mouse (Murine) Monoclonal IL1B Primary Antibody pour IP, Neut - ABIN1176987
Fei, Gott, Edwards, Schuyler: Experimental hypersensitivity pneumonitis: in vitro effects of interleukin-2 and interferon-gamma. dans The Journal of laboratory and clinical medicine 1995
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Mouse (Murine) Polyclonal IL1B Primary Antibody pour ELISA, FACS - ABIN4324365
Triantafilou, Kar, Vakakis, Kotecha, Triantafilou: Human respiratory syncytial virus viroporin SH: a viral recognition pathway used by the host to signal inflammasome activation. dans Thorax 2012
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Human Monoclonal IL1B Primary Antibody pour FACS - ABIN4895764
Hakonarson, Whelan, Leiter, Kim, Chen, Campbell, Grunstein: T lymphocyte-mediated changes in airway smooth muscle responsiveness are attributed to induced autocrine release and actions of IL-5 and IL-1beta. dans The Journal of allergy and clinical immunology 2002
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This study reveals that proper levels of Il1b signaling and tissue inflammation, which are tuned by macrophages, play a crucial role in tissue regeneration.
Leukocyte expression of IL-1beta was detectable only following injury, which activated leukocytes throughout zebrafish embryos in a caspase (Montrer CASP3 Anticorps) dependent manner.
Embryo and larva leukocytes upregulate IL-1beta expression proportional to the dose of ultraviolet radiation exposure in an immune response at the organismal level.
findings reveal that the Il-1beta-Myd88 (Montrer MYD88 Anticorps) axis and NADPH oxidase (Montrer NOX1 Anticorps)-mediated ROS (Montrer ROS1 Anticorps) signaling are two independent pathways that differentially regulate neutrophil migration during sterile inflammation.
the expression levels of IL-1beta and TNF-alpha in high cholesterol diet (HCD)-fed zebrafish larvae
These results represent the first demonstration of processing and secretion of zebrafish IL-1beta in response to a pathogen.
Inhibition of signaling stimulated by both TNF (Montrer TNF Anticorps) and IL1beta synergizes with NF-kappaB (Montrer NFKB1 Anticorps) inhibition in eliminating leukemic stem cells.
Parenchymal polymorphonuclear myeloid-derived suppressor cell (PMN (Montrer TBCE Anticorps)-MDSC), have a positive correlation with IL1a (Montrer IL1A Anticorps), IL8 (Montrer IL8 Anticorps), CXCL5 (Montrer CXCL5 Anticorps), and Mip-1a (Montrer CCL3 Anticorps), suggesting they may attract PMN (Montrer TBCE Anticorps)-MDSC into the tumor
Chemokine receptor 2 (CCR2 (Montrer CCR2 Anticorps)(+)) monocytes invade the hippocampus between 1 and 3 d after SE. In contrast, only an occasional CD3 (Montrer CD3E Anticorps)(+) T lymphocyte was encountered 3 d after SE. The initial cellular sources of the chemokine (Montrer CCL1 Anticorps) CCL2 (Montrer CCL2 Anticorps), a ligand for CCR2 (Montrer CCR2 Anticorps), included perivascular macrophages and microglia. The induction of the proinflammatory cytokine IL-1beta was greater in FACS-isolated microglia than in brain-invading monocytes
hypernociception in experimental model of autoimmune encephalomyelitis may be a consequence of the increase in some cytokines in dorsal root ganglia, especially IL-1beta.
An OA model was established in mouse articular chondrocytes (MACs) treated by interleukin-1beta (IL-1beta).
The current study demonstrated that honey can stimulate or suppress the mRNA expression of some pro-inflammatory cytokines in mice brains. Furthermore, honey suppresses the TNF-alpha (Montrer TNF Anticorps) mRNA expression in the presence of T. gondii infection but it stimulates the IL-1beta and IL-6 (Montrer IL6 Anticorps) mRNA expression. Treatment of the mice with honey reduces parasite multiplication in the brain.
IL-1beta has a direct effect on NGAL (Montrer LCN2 Anticorps) production by tubular epithelial cells.
Following vasectomy, IL1alpha (Montrer IL1A Anticorps), IL1beta, IL1ra (Montrer IL1RN Anticorps), IL10 (Montrer IL10 Anticorps), and TNF-alpha (Montrer TNF Anticorps) may mediate immune reaction in whole epididymis, whereas IL6 (Montrer IL6 Anticorps) and TGF-beta1 (Montrer TGFB1 Anticorps) may mediate regionally different immune response primarily in the lower part of epididymis.
Elevations of CO2 cause oligomerization of the inflammasome components ASC (Montrer STS Anticorps), NLRP3 (Montrer NLRP3 Anticorps), caspase 1 (Montrer CASP1 Anticorps), thioredoxin interacting protein (Montrer TXNIP Anticorps), and calreticulin (Montrer CALR Anticorps) - a protein from endoplasmic reticulum, leading to IL-1beta synthesis. An increased production rate of MPs containing elevated amounts of IL-1beta persists for hours after short-term exposures to elevated CO2
dimerized or endogenous caspase-8 (Montrer CASP8 Anticorps) can also directly cleave IL-1beta into its biologically active form, in the absence of canonical inflammasome components.
our study has described the association between rs3783550 (IL-1A), rs3783546 (IL-1A) and rs2853550 (IL-1B) and AS risk and between a new haplotype, "TCG", of rs3783550, rs3783546 and rs2853550 and AS in Chinese Han population.
serum levels of IL-1beta were able to prognostically stratify our cohort of iDCM patients. The precise cut-off of IL-1beta associated with bad outcome in iDCM remains to be determined in larger, independent cohorts of patients.
Biological complications in implant-supported overdentures were found to be mostly associated with the presence of allele 2 of the IL1B gene.
High interleukin 1 beta expression is associated with neuroinflammation.
Data show that cyclic stretch activated the nucleotide-binding oligomerization domain-like receptor containing pyrin (Montrer MEFV Anticorps) domain 1 and 3 (NLRP1 (Montrer NLRP1 Anticorps) and NLRP3 (Montrer NLRP3 Anticorps)) inflammasomes and induced the release of IL-1beta and pyroptosis via a caspase-1 (Montrer CASP1 Anticorps)-related mechanism in human periodontal ligament cells (HPDLCs).
Our findings suggest that there is a prevalence of mutated allele of IL-1 (Montrer IL1A Anticorps) gene cluster in our population, which may be responsible for renal dysfunction.
Demonstrate that RARalpha (Montrer RARA Anticorps) was frequently elevated in gastric carcinoma and exerted oncogenic properties via positive feedback loop of IL-1beta/Akt (Montrer AKT1 Anticorps)/RARalpha (Montrer RARA Anticorps)/Akt (Montrer AKT1 Anticorps) signaling.
Icariin inhibits MMP1 (Montrer MMP1 Anticorps), MMP3 (Montrer MMP3 Anticorps) and MMP13 (Montrer MMP13 Anticorps) expression through MAPK (Montrer MAPK1 Anticorps) pathways in IL1betastimulated SW1353 chondrosarcoma cells
Sesamin showed anti-inflammatory effects in IL-1beta-stimulated chondrocytes by activating Nrf2 (Montrer GABPA Anticorps) signaling pathway.
In conclusion, the analyzed IL1A (Montrer IL1A Anticorps) -889 C>T, IL1B +3954 C>T, and IL6 (Montrer IL6 Anticorps) -174 G>C polymorphisms may be associated with the occurrence and development of human cytomegalovirus infection among studied patients.
This study showed that classical swine fever virus and p7 protein induced IL-1beta secretion and that p7 protein was a short-lived protein degraded by the proteasome.
local expression of IL-1beta and IL-8 (Montrer IL8 Anticorps) in non-bacterial thrombotic endocarditis, Staphylococcus aureus infective endocarditis, animals with S. aureus sepsis without endocarditis and controls
IL1B regulates expression of IL1R1 (Montrer IL1RN Anticorps) and IL1RAP (Montrer IL1RAP Anticorps) and stimulates expression of PTGS1 (Montrer PTGS1 Anticorps) and PTGS2 (Montrer PTGS2 Anticorps) that are considered to be the most rate-limiting enzymes for endometrial synthesis of prostaglandins during the peri (Montrer PLIN1 Anticorps)-implantation period of pregnancy in pigs.
the results presented here strongly suggest IL-1beta as a key molecule guiding tissue remodelling events after myocardial infarction.
The presence of embryos increased endometrial IL1B protein locally, while no differences regarding IL1R1 (Montrer IL1RN Anticorps) protein and IL1B and IL1R1 (Montrer IL1RN Anticorps) mRNA were detected.
For the inflammasome-dependent IL-1beta release, bovine monocytes require ATP in addition to a primary stimulus. This IL-1beta release depends on potassium efflux, but, in contrast to human and murine monocytes, does not require calcium influx or generation of reaction oxygen and is independent of the P2X7 receptor (Montrer P2RX7 Anticorps).
Role of TGF-beta1 (Montrer TGFB1 Anticorps) and TNF-alpha (Montrer TNF Anticorps) in IL-1beta mediated activation of proMMP-9 in pulmonary artery smooth muscle cells: involvement of an aprotinin sensitive protease.
Testicular IL-1 alpha (Montrer IL1A Anticorps) and IL-1 beta concentrations were highest in the early post-natal period; however, IL-1 (Montrer IL1A Anticorps) bioactivity and IL-6 (Montrer IL6 Anticorps) concentrations were greatest in the immediate pre-pubertal period.
Data describe the expression of IL-8 (Montrer IL8 Anticorps), IL-1beta and their respective receptors, CXCR1 (Montrer CXCR1 Anticorps) and IL-1R1 in bovine theca cells, and suggest that VEGF (Montrer VEGFA Anticorps) is associated with the IL system in theca cells in ovary.
Genes for IL-1alpha and IL-1beta are expressed and a functional IL-1R is present in bovine corpora lutea throughout luteal phase. IL-1alpha and IL-1beta may have different roles as regulating PGF (Montrer PGF Anticorps)(2alpha) and PGE (Montrer LIPF Anticorps)(2) production during luteal phase.
non-metalloproteinase mechanisms participate in IL-1 (Montrer IL1A Anticorps)-induced matrix degradation and loss of tissue material properties
dynamic compression stimulates cell proliferation and proteoglycan (Montrer Vcan Anticorps) synthesis in the presence of IL-1beta and/or inhibitors of the MAPKs and NFkappaB and AP-1 (Montrer JUN Anticorps) signalling pathways
These results indicate that activation of the intrinsic antistaphylococcal response in bovine endothelial cells (BEC), enhanced by TNF-alpha (Montrer TNF Anticorps) and IL-1beta, is effective to eliminate S. aureus and S. epidermidis.
the low friction of articular cartilage can be modified by TGF-beta1 (Montrer TGFB1 Anticorps) and IL-1beta treatment and that the friction coefficient depends on multiple factors, including superficial zone protein localization and surface roughness
Mild heat shock increased the production of inflammatory cytokines, IL-1beta and IL-6 (Montrer IL6 Anticorps) in rabbit cornea cells.
IL-1beta and TNF-alpha (Montrer TNF Anticorps) expression increases significantly during acute lung injury. Ambroxol combined with low-dose heparin inhibits teh release of IL-1beta and TNF-alpha (Montrer TNF Anticorps).
IL-1beta induced a significant reduction in the relative intrinsic activity of GLUT5 (Montrer SLC2A5 Anticorps) and in this decrease are involved NO signal pathways; blockage of D-fructose intestinal uptake by IL-1beta may play an essential role in the pathophysiology of septic shock.
The inhibitory effect of IL-1beta on D-galactose absorption across mucosal side of enterocyte could be mediated by the activation of several kinases and nuclear factor kappa B.
Glucosamine hydrochloride treatment can can partially decrease the expression levels of IL-1 beta and increase the expression levels of TGF-beta 1 (Montrer TGFB1 Anticorps), which delays the development of osteoarthritis.
In this study evidence is provided that exogenous PGF2alpha differentially modulates luteal expression of IL1B transcripts depending on luteal stage.
results revealed that a transient episode of raised-intensity phonation causes a significant increase in vocal fold inflammatory mRNA expression - IL-1beta,COX-2 (Montrer COX2 Anticorps), and TGFbeta1 (Montrer TGFB1 Anticorps)
Increased PGE2 production led to reduction in 5-LO (Montrer ALOX5 Anticorps) products in LPS (Montrer IRF6 Anticorps)-treated equine whole blood via IL-1b.
Results suggested that chemokine (Montrer CCL1 Anticorps) expression by cultured equine BECs following exposure to pulmonary hemorrhage conditions may contribute to the development of inflammatory airway disease in horses.
IL-1beta-induced up-regulation of matrix metalloproteinase 13 (Montrer MMP13 Anticorps) mRNA was blocked by all concentrations of geldanamycin tested
This study examined effects of in vitro exposure to solutions of hay (Montrer GTF2H5 Anticorps) dust, lipopolysaccharides, or beta-glucan on cytokine expression in pulmonary mononuclear cells isolated from healthy horses and horses with recurrent airway obstruction.
The protein encoded by this gene is a member of the interleukin 1 cytokine family. This cytokine is produced by activated macrophages as a proprotein, which is proteolytically processed to its active form by caspase 1 (CASP1/ICE). This cytokine is an important mediator of the inflammatory response, and is involved in a variety of cellular activities, including cell proliferation, differentiation, and apoptosis. The induction of cyclooxygenase-2 (PTGS2/COX2) by this cytokine in the central nervous system (CNS) is found to contribute to inflammatory pain hypersensitivity. This gene and eight other interleukin 1 family genes form a cytokine gene cluster on chromosome 2.
, IL-1 beta
, interleukin-1 beta
, preinterleukin 1 beta
, prointerleukin-1 beta
, Interleukin-1 beta
, precursor interleukin-1beta
, interleukin-1 beta proprotein
, interleukin 1 beta
, lymphocyte proliferation-potentiating factor