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anti-Mouse (Murine) IL1B Anticorps:
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Human Monoclonal IL1B Primary Antibody pour CyTOF, FACS - ABIN4899223
Yates, Burgess, Kocsis-Angle, Antal, Dority, Embury, Piotrkowski, Brunden: Amyloid beta and amylin fibrils induce increases in proinflammatory cytokine and chemokine production by THP-1 cells and murine microglia. dans Journal of neurochemistry 2000
Show all 15 Pubmed References
Bacteria Polyclonal IL1B Primary Antibody pour ELISA, EM - ABIN269770
Petrovski, Ayna, Majai, Hodrea, Benko, Mádi, Fésüs: Phagocytosis of cells dying through autophagy induces inflammasome activation and IL-1β release in human macrophages. dans Autophagy 2011
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Dog (Canine) Polyclonal IL1B Primary Antibody pour IF (p), IHC (p) - ABIN728503
Zhong, Shu, Wang, Luo, Zhong, Ran, Zheng, Yin, Ling: Enhanced homing of mesenchymal stem cells to the ischemic myocardium by ultrasound-targeted microbubble destruction. dans Ultrasonics 2011
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Mouse (Murine) Polyclonal IL1B Primary Antibody pour Neut, WB - ABIN223538
Scott, Ma, Viriyakosol, Terkeltaub, Liu-Bryan: Engagement of CD14 mediates the inflammatory potential of monosodium urate crystals. dans Journal of immunology (Baltimore, Md. : 1950) 2006
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Human Polyclonal IL1B Primary Antibody pour IHC (p), IHC - ABIN446969
Omran, Peng, Zhang, Xiang, Xue, Gan, Kong, Yin: Interleukin-1β and microRNA-146a in an immature rat model and children with mesial temporal lobe epilepsy. dans Epilepsia 2012
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Mouse (Murine) Monoclonal IL1B Primary Antibody pour IP, Neut - ABIN1176987
Fei, Gott, Edwards, Schuyler: Experimental hypersensitivity pneumonitis: in vitro effects of interleukin-2 and interferon-gamma. dans The Journal of laboratory and clinical medicine 1995
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Human Monoclonal IL1B Primary Antibody pour FACS - ABIN4895764
Hakonarson, Whelan, Leiter, Kim, Chen, Campbell, Grunstein: T lymphocyte-mediated changes in airway smooth muscle responsiveness are attributed to induced autocrine release and actions of IL-5 and IL-1beta. dans The Journal of allergy and clinical immunology 2002
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Human Monoclonal IL1B Primary Antibody pour FACS - ABIN4895765
Hussain, Kong, Jordan, Conrad, Madden, Fokt, Priebe, Heimberger: A novel small molecule inhibitor of signal transducers and activators of transcription 3 reverses immune tolerance in malignant glioma patients. dans Cancer research 2007
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Mouse (Murine) Polyclonal IL1B Primary Antibody pour ELISA, FACS - ABIN4324365
Triantafilou, Kar, Vakakis, Kotecha, Triantafilou: Human respiratory syncytial virus viroporin SH: a viral recognition pathway used by the host to signal inflammasome activation. dans Thorax 2012
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Rat (Rattus) Polyclonal IL1B Primary Antibody pour Func, ELISA - ABIN2474901
Marcelis, Kurdziel, Dondelinger: Small intestinal obstruction caused by extramedullary hemopoiesis and revealing myeloproliferative syndrome. dans Journal belge de radiologie 1992
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This study reveals that proper levels of Il1b signaling and tissue inflammation, which are tuned by macrophages, play a crucial role in tissue regeneration.
Leukocyte expression of IL-1beta was detectable only following injury, which activated leukocytes throughout zebrafish embryos in a caspase (Montrer CASP3 Anticorps) dependent manner.
Embryo and larva leukocytes upregulate IL-1beta expression proportional to the dose of ultraviolet radiation exposure in an immune response at the organismal level.
findings reveal that the Il-1beta-Myd88 (Montrer MYD88 Anticorps) axis and NADPH oxidase (Montrer NOX1 Anticorps)-mediated ROS (Montrer ROS1 Anticorps) signaling are two independent pathways that differentially regulate neutrophil migration during sterile inflammation.
the expression levels of IL-1beta and TNF-alpha in high cholesterol diet (HCD)-fed zebrafish larvae
These results represent the first demonstration of processing and secretion of zebrafish IL-1beta in response to a pathogen.
The present study demonstrated that IL-1b may induce ICAM-1 (Montrer ICAM1 Anticorps) expression, thus enhancing the cohesion between mesenchymal stem cells and endothelial progenitor cells via the p38 MAPK (Montrer MAPK14 Anticorps) signaling pathway.
SAG2A differentially modulates IL-1beta expression in resistant and susceptible murine peritoneal macrophages cells.
High IL-1beta expression is associated with experimental autoimmune encephalomyelitis.
tibias of botulin A toxin-treated and tail-suspended mice, which featured unloading and decreased bone mass, showed higher expression of IL-1beta, Lcn2 and Nos2, suggesting their pathophysiologic involvement in endothelial cell-osteoblast crosstalk.
HMGB1 (Montrer HMGB1 Anticorps)/IL-1beta complexes released after burn injuries can modulate immune responses
bone marrow-derived macrophages (BMM) and three murine macrophage cell lines, J774.1, J774A.1, and RAW264.7 were exposed to ATP or fibrous titanium dioxide (FTiO2) in the presence or absence of lipopolysaccharide (LPS (Montrer TLR4 Anticorps)), and the concentrations of IL-1beta and IL-6 (Montrer IL6 Anticorps) in both cell lysates and in the culture media were measured by immunoblotting to differentiate active form of IL-1beta from pro-IL-1beta.
The present study demonstrates a novel mechanism underlying LPS (Montrer TLR4 Anticorps)-induced innate immunity; that is, a secondary upregulation of IL-1beta-IL-1RI signaling is responsible for alveolar macrophages pyroptosis and augmented lung injury in response to LPS (Montrer TLR4 Anticorps).
IL-33 (Montrer IL33 Anticorps) may induce Th17 cell responses via IL-1beta and IL-6 (Montrer IL6 Anticorps) derived from IL-33 (Montrer IL33 Anticorps)-matured dendritic cells.
ESP of fifth-stage larval Angiostrongylus cantonensis stimulates astrocyte activation and IL-1beta and IL-6 (Montrer IL6 Anticorps) production through NF-kappaB (Montrer NFKB1 Anticorps) and the Shh (Montrer SHH Anticorps) signaling pathway.
we confirmed that Th1 (Montrer HAND1 Anticorps) cell-conditioned medium decreased Cx43 (Montrer GJA1 Anticorps) protein levels in mixed glial cell cultures. These findings suggest that Th1 (Montrer HAND1 Anticorps) cell-derived IFNg (Montrer IFNG Anticorps) activates microglia to release IL-1b that reduces Cx43 (Montrer GJA1 Anticorps) gap junctions in astrocytes. Thus, Th1 (Montrer HAND1 Anticorps)-dominant inflammatory states disrupt astrocytic intercellular communication and may exacerbate multiple sclerosis.
IL-1beta and CTHRC1 (Montrer CTHRC1 Anticorps) are upregulated in patients with Osteoarthritis.
our observations indicated that IL-1beta-mediated NF-kappaB (Montrer NFKB1 Anticorps) signaling augments the osteosarcoma cell growth via modulating MiR (Montrer MLXIP Anticorps)-376c/TGFA (Montrer TGFA Anticorps) axis
Insulin (Montrer INS Anticorps) resistance correlates positively with IL-1beta among non-diabetic hemodialysis patients.
The baseline concentrations of IL-6 (Montrer IL6 Anticorps) and IL-1beta were significantly higher in the Major Depressive Disorder group than those in the control group. There was no significant difference of TNF-alpha (Montrer TNF Anticorps) between the two groups.
The genetic association between the studied SNPs of the IL-1beta gene and recurrent apththous stomatitis remains controversial and requires further investigation.
The rs16944 TT genotype of Il-1 beta is associated with mesial temporal lobe epilepsy with hippocampal sclerosis development what may be explained by the higher IL-1beta levels produced by this genotype.
Our meta-analysis proved that IL-1beta + 3954C/T is associated with MI susceptibility, especially among Caucasian populations.
In this longitudinally monitored male population, observed effect of baseline central adiposity on future periodontitis progression is conditional on proinflammatory IL-1 (Montrer IL1A Anticorps) genetic variations.
In Pakistani population, an increased risk of gastric cancer development is associated with the carriage of IL-1B-511*T and IL-1RN (Montrer IL1RN Anticorps)*2 alleles
This study revealed that the P2X7R (Montrer P2RX7 Anticorps)/NLRP3 (Montrer NLRP3 Anticorps) pathway plays important roles in IL-1beta secretion and inhibition of Toxoplasma gondii proliferation in small intestinal epithelial cells.
This study showed that the presence of embryos in oviducts on days 2-3 after mating may influence the oviductal expression of the members of the IL-1beta system, determining the action of IL-1beta, which may be considered to be the earliest sign of pregnancy in pigs.
This study showed that classical swine fever virus and p7 protein induced IL-1beta secretion and that p7 protein was a short-lived protein degraded by the proteasome.
local expression of IL-1beta and IL-8 (Montrer IL8 Anticorps) in non-bacterial thrombotic endocarditis, Staphylococcus aureus infective endocarditis, animals with S. aureus sepsis without endocarditis and controls
IL1B regulates expression of IL1R1 (Montrer IL1RN Anticorps) and IL1RAP (Montrer IL1RAP Anticorps) and stimulates expression of PTGS1 (Montrer PTGS1 Anticorps) and PTGS2 (Montrer PTGS2 Anticorps) that are considered to be the most rate-limiting enzymes for endometrial synthesis of prostaglandins during the peri (Montrer PLIN1 Anticorps)-implantation period of pregnancy in pigs.
the results presented here strongly suggest IL-1beta as a key molecule guiding tissue remodelling events after myocardial infarction.
Mycobacterium bovis infected animals have a higher frequency of IFN-gamma (Montrer IFNG Anticorps) producing CD4 (Montrer CD4 Anticorps)(+) T cells and elevated plasma IL-1beta levels.
The presence of embryos increased endometrial IL1B protein locally, while no differences regarding IL1R1 (Montrer IL1RN Anticorps) protein and IL1B and IL1R1 (Montrer IL1RN Anticorps) mRNA were detected.
For the inflammasome-dependent IL-1beta release, bovine monocytes require ATP in addition to a primary stimulus. This IL-1beta release depends on potassium efflux, but, in contrast to human and murine monocytes, does not require calcium influx or generation of reaction oxygen and is independent of the P2X7 receptor (Montrer P2RX7 Anticorps).
Role of TGF-beta1 (Montrer TGFB1 Anticorps) and TNF-alpha (Montrer TNF Anticorps) in IL-1beta mediated activation of proMMP-9 in pulmonary artery smooth muscle cells: involvement of an aprotinin sensitive protease.
Testicular IL-1 alpha (Montrer IL1A Anticorps) and IL-1 beta concentrations were highest in the early post-natal period; however, IL-1 (Montrer IL1A Anticorps) bioactivity and IL-6 (Montrer IL6 Anticorps) concentrations were greatest in the immediate pre-pubertal period.
Data describe the expression of IL-8 (Montrer IL8 Anticorps), IL-1beta and their respective receptors, CXCR1 (Montrer CXCR1 Anticorps) and IL-1R1 in bovine theca cells, and suggest that VEGF (Montrer VEGFA Anticorps) is associated with the IL system in theca cells in ovary.
Genes for IL-1alpha and IL-1beta are expressed and a functional IL-1R is present in bovine corpora lutea throughout luteal phase. IL-1alpha and IL-1beta may have different roles as regulating PGF (Montrer PGF Anticorps)(2alpha) and PGE (Montrer LIPF Anticorps)(2) production during luteal phase.
non-metalloproteinase mechanisms participate in IL-1 (Montrer IL1A Anticorps)-induced matrix degradation and loss of tissue material properties
dynamic compression stimulates cell proliferation and proteoglycan (Montrer Vcan Anticorps) synthesis in the presence of IL-1beta and/or inhibitors of the MAPKs and NFkappaB and AP-1 (Montrer JUN Anticorps) signalling pathways
These results indicate that activation of the intrinsic antistaphylococcal response in bovine endothelial cells (BEC), enhanced by TNF-alpha (Montrer TNF Anticorps) and IL-1beta, is effective to eliminate S. aureus and S. epidermidis.
Mild heat shock increased the production of inflammatory cytokines, IL-1beta and IL-6 (Montrer IL6 Anticorps) in rabbit cornea cells.
IL-1beta and TNF-alpha (Montrer TNF Anticorps) expression increases significantly during acute lung injury. Ambroxol combined with low-dose heparin inhibits teh release of IL-1beta and TNF-alpha (Montrer TNF Anticorps).
IL-1beta induced a significant reduction in the relative intrinsic activity of GLUT5 (Montrer SLC2A5 Anticorps) and in this decrease are involved NO signal pathways; blockage of D-fructose intestinal uptake by IL-1beta may play an essential role in the pathophysiology of septic shock.
The inhibitory effect of IL-1beta on D-galactose absorption across mucosal side of enterocyte could be mediated by the activation of several kinases and nuclear factor kappa B.
Glucosamine hydrochloride treatment can can partially decrease the expression levels of IL-1 beta and increase the expression levels of TGF-beta 1 (Montrer TGFB1 Anticorps), which delays the development of osteoarthritis.
In this study evidence is provided that exogenous PGF2alpha differentially modulates luteal expression of IL1B transcripts depending on luteal stage.
results revealed that a transient episode of raised-intensity phonation causes a significant increase in vocal fold inflammatory mRNA expression - IL-1beta,COX-2 (Montrer COX2 Anticorps), and TGFbeta1 (Montrer TGFB1 Anticorps)
Increased PGE2 production led to reduction in 5-LO (Montrer ALOX5 Anticorps) products in LPS (Montrer IRF6 Anticorps)-treated equine whole blood via IL-1b.
Results suggested that chemokine (Montrer CCL1 Anticorps) expression by cultured equine BECs following exposure to pulmonary hemorrhage conditions may contribute to the development of inflammatory airway disease in horses.
IL-1beta-induced up-regulation of matrix metalloproteinase 13 (Montrer MMP13 Anticorps) mRNA was blocked by all concentrations of geldanamycin tested
This study examined effects of in vitro exposure to solutions of hay (Montrer GTF2H5 Anticorps) dust, lipopolysaccharides, or beta-glucan on cytokine expression in pulmonary mononuclear cells isolated from healthy horses and horses with recurrent airway obstruction.
The protein encoded by this gene is a member of the interleukin 1 cytokine family. This cytokine is produced by activated macrophages as a proprotein, which is proteolytically processed to its active form by caspase 1 (CASP1/ICE). This cytokine is an important mediator of the inflammatory response, and is involved in a variety of cellular activities, including cell proliferation, differentiation, and apoptosis. The induction of cyclooxygenase-2 (PTGS2/COX2) by this cytokine in the central nervous system (CNS) is found to contribute to inflammatory pain hypersensitivity. This gene and eight other interleukin 1 family genes form a cytokine gene cluster on chromosome 2.
, IL-1 beta
, interleukin-1 beta
, preinterleukin 1 beta
, prointerleukin-1 beta
, Interleukin-1 beta
, precursor interleukin-1beta
, interleukin-1 beta proprotein
, interleukin 1 beta
, lymphocyte proliferation-potentiating factor