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anti-Human Tumor Necrosis Factor Anticorps:
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Mouse (Murine) Polyclonal Tumor Necrosis Factor Primary Antibody pour CyTOF, ELISA (Capture) - ABIN4900489
Ramkhelawon, Hennessy, Ménager, Ray, Sheedy, Hutchison, Wanschel, Oldebeken, Geoffrion, Spiro, Miller, McPherson, Rayner, Moore: Netrin-1 promotes adipose tissue macrophage retention and insulin resistance in obesity. dans Nature medicine 2014
Show all 59 Pubmed References
Rat (Rattus) Polyclonal Tumor Necrosis Factor Primary Antibody pour WB - ABIN3042522
Zhang, Liu, Goldstein, Li, Ge, He, Fei, Wang, Ruiz: Role of the JAK/STAT signaling pathway in the pathogenesis of acute myocardial infarction in rats and its effect on NF-?B expression. dans Molecular medicine reports 2013
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Mouse (Murine) Polyclonal Tumor Necrosis Factor Primary Antibody pour WB - ABIN3043313
Liu, Shan, Dong, Liu, Ma, Liu: Combined early fluid resuscitation and hydrogen inhalation attenuates lung and intestine injury. dans World journal of gastroenterology 2013
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Mouse (Murine) Polyclonal Tumor Necrosis Factor Primary Antibody pour ELISA, IHC (p) - ABIN3042817
Qin, Ma, Yang, Hu, Zhou, Fu, Tian, Liu, Xu, Shen: A Triterpenoid Inhibited Hormone-Induced Adipocyte Differentiation and Alleviated Dexamethasone-Induced Insulin Resistance in 3T3-L1 adipocytes. dans Natural products and bioprospecting 2015
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Human Polyclonal Tumor Necrosis Factor Primary Antibody pour IHC (p), WB - ABIN4886747
Kan, Zhou, Jin, Yang: Effects of PDTC on NF-?B expression and apoptosis in rats with severe acute pancreatitis-associated lung injury. dans International journal of clinical and experimental medicine 2015
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Mouse (Murine) Polyclonal Tumor Necrosis Factor Primary Antibody pour WB - ABIN5518792
Zhang, Leng, Xue, Zhang, Wang, Kang: Effects of penehyclidine hydrochloride in small intestinal damage caused by limb ischemia-reperfusion. dans World journal of gastroenterology 2011
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Human Monoclonal Tumor Necrosis Factor Primary Antibody pour - ABIN1383955
Hervé, Racadot, Wijdenes, Flesch, Tiberghien, Bordigoni, Holler, Powles, Bourdeau, Wilmer: Monoclonal anti TNF alpha antibody in the treatment of acute GvHD refractory both to corticosteroids and anti IL-2 R antibody. dans Bone marrow transplantation 1991
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Human Polyclonal Tumor Necrosis Factor Primary Antibody pour ICC, IF - ABIN446837
Kwon, Jung, Hwang, Choi: Evaluation of Bronchiolar and Alveolar Cell Injuries Induced by Short- and Long-term Exposure to Sidestream Smoke. dans Korean journal of pathology 2012
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Human Polyclonal Tumor Necrosis Factor Primary Antibody pour IF (p), IHC (p) - ABIN677318
Morishita, Oku, Horie, Tonari, Kida, Okubo, Sugiyama, Takai, Hara, Ikeda: Systemic simvastatin rescues retinal ganglion cells from optic nerve injury possibly through suppression of astroglial NF-?B activation. dans PLoS ONE 2014
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Human Polyclonal Tumor Necrosis Factor Primary Antibody pour IHC (p), IHC - ABIN153474
Roth, Städler, Lemann, Hösli, Simon, Simon: Distinct eosinophil cytokine expression patterns in skin diseases - the possible existence of functionally different eosinophil subpopulations. dans Allergy 2011
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Impairment in TNF, IL-1beta (Montrer IL1B Anticorps), and IL-17 (Montrer IL17A Anticorps) production upon stimulation with mycobacterial antigens may contribute to the increased susceptibility to M. tuberculosis infection observed in HTLV-1 infected individuals.
LL was significantly negatively correlated with PGC-1alpha, TNF-alpha, and IL-6 mRNA expressions. PGC-1alpha mRNA expression levels in paraspinal muscles may be affected by lumbar kyphosis.
TNF-alpha-308G>A polymorphism affects the overall survival of cancer patients and is a potential therapeutic target for cancer.
Many inflammatory pathologies are now recognized to be driven by aberrant TNF-induced cell death, which, in most circumstances, depends on the kinase Receptor-interacting serine/threonine-protein kinase 1 (RIPK1 (Montrer RIPK1 Anticorps)). [review]
High TNF-alpha expression is associated with retinopathy of prematurity.
Tumour necrosis factor-alpha selectively reduces BMPR-II (Montrer BMPR2 Anticorps) transcription and mediates post-translational BMPR-II (Montrer BMPR2 Anticorps) cleavage via the sheddases, ADAM10 (Montrer ADAM10 Anticorps) and ADAM17 (Montrer ADAM17 Anticorps) in pulmonary artery smooth muscle cells.
Polymorphisms of IL-1betab and TNF-a are not a risk of ICC, but an individual with O. viverrini infection has an effect on all genotypes of the TNF-alpha gene that might promote intrahepatic cholangiocarcinoma . Primary prevention of intrahepatic cholangiocarcinoma in high-risk areas is based on efforts to reduce O. viverrini infection.
In placenta, when gestational age was controlled for, partial correlation revealed a significant positive correlation between TNF-alpha and MMP-9 (Montrer MMP9 Anticorps) only in the second trimester.
Study shows that in human endometrial stromal cells, high tumor necrosis factor levels negatively affect the insulin action through decreased adiponectin signaling and glucose transporter type 4 protein. This could explain the failures observed in endometrial function of obese women with polycystic ovary syndrome.
Three single nucleotide polymorphisms (SNPs) within P2X4R (Montrer P2RX4 Anticorps) and two SNPs within CAMKK2 (Montrer CAMKK2 Anticorps) influenced concentrations of TNFalpha in peripheral blood mononuclear cells, but these SNP did not associate with risk for HIV-associated sensory neuropathy in South Africans.
In vitro mild uncoupling rescued from TNF-induced endothelial permeability, disassembly of cell contacts and VE-cadherin cleavage by the matrix metalloprotease 9 (capital EM, Cyrilliccapital EM, Cyrilliccapital ER, Cyrillic9). The uncouplers prevented TNF-induced expression of MMP9 via inhibition of NFkappaB signaling.
macrophage-TNF-induced AKT (Montrer AKT1 Anticorps)/beta-catenin (Montrer CTNNB1 Anticorps) signalling in Lgr5 (Montrer LGR5 Anticorps)(+) hair follicle stem cells has a crucial role in promoting hair follicle cycling and neogenesis after wounding
Transmembrane TNF, TNFR2 (Montrer TNFRSF1B Anticorps) and TNFR1 (Montrer TNFRSF1A Anticorps) (indirectly) are critical for preventing inflammation during BCG (Montrer SLC11A1 Anticorps)-induced pleurisy in mice.
findings demonstrate a new role for TNFalpha as a key regulator of neutrophil trafficking into and within lymphatic system in vivo.
Our work suggested that TNF-alpha and TNF-R1 (Montrer TNFRSF1A Anticorps) are the major contributors of TNF signaling pathway in anesthesia-induced spinal cord neurotoxicity. Targeting TNF-alpha / TNF-R1 (Montrer TNFRSF1A Anticorps), not TNF-R2 (Montrer TNFRSF1B Anticorps) signaling pathway may be the key component to rescue or prevent anesthesia-induced apoptotic injury in spinal cord neurons.
observation from the present research work reveals that Quercetin suppressed the production of proinflammatory cytokines at different levels, such as TNF-alpha and IL-1beta (Montrer IL1B Anticorps), and inhibits the activation of I-kappaB phosphorylation, whereas the total content was not affected.
this is the first evidence to suggest that TET2 (Montrer TET2 Anticorps) mutations promote clonal dominance with aging by conferring TNFalpha resistance to sensitive bone marrow progenitors while also propagating such an inflammatory environment.
Elevated A20 (Montrer TNFAIP3 Anticorps) promotes TNF-induced and RIPK1 (Montrer RIPK1 Anticorps)-dependent intestinal epithelial cell death
M. tuberculosis and TNFalpha synergise to induce necroptosis in murine fibroblasts via RIPK1-dependent mechanisms and characterized by phosphorylation of Ser345 of the MLKL necroptosis death effector.
Our current study has demonstrated that in allergic airway disease (AAD) mice, intestinal dysbiosis (ID) caused increased nasal rubbing, sneezing, serum OVA specific IgE level and pro-inflammatory cytokine TNF-alpha in NALF and BALF. ID also inhibited miR (Montrer MLXIP Anticorps)-130a expression in AAD mice. Further molecular experiments indicated that miR (Montrer MLXIP Anticorps)-130a could specifically target and repress TNF-alpha mRNA expression.
Data suggest that luteolytic factors (such as TNFa, interferon gamma (Montrer IFNG Anticorps), and PGF2a) control expression of MMP1 (Montrer MMP1 Anticorps), other matrix metalloproteinases, and tissue inhibitors of metalloproteinase in cultured luteal cells.
These results suggest that polymorphism of the TNFalpha-824 A>G gene and mTNFalpha protein expression play an important role in the pathogenesis of enzootic bovine leukosis.
These results suggest that the endometrium might lower the TNF concentration in the blastocyst by (1) regulating TNF secretion into the uterine fluid and (2) inducing decreased TNF and TNFR2 (Montrer TNFRSF1B Anticorps) mRNA transcription in the embryo.
There was a significant association between the proviral load and a low frequency of the G/G genotype of TNF-alpha at position -824.
SNP in the TNF-alpha gene affects immune function and reproductive performance in dairy cows.
Studied genotypic and expression profiling of partial TNF-alpha gene and its association with mastitis susceptibility in 129 crossbred cattle.
Messenger RNA and protein levels of prostaglandin (PG) E synthase (PGES (Montrer PTGES Anticorps)), PGF2alpha receptor (PGFR), tumor necrosis factor-alpha (TNF) and Fas (Montrer FAS Anticorps) were found to be higher in the corpus luteum of pregnancy than in corpus luteum of the cycle.
TNF-alpha up-regulates NaV1.7 (Montrer SCN9A Anticorps) mRNA in both adrenal chromaffin cells and dorsal root ganglia (DRG) neurons, highlighting the peripheral nociceptive mechanism of TNF-alpha
These results provide evidence for a high prevalence of subclinical endometritis in repeat breeding cows as well as the involvement of TNFalpha and iNOS (Montrer NOS2 Anticorps) pathways in the regulation of this pathological condition.
Exposure to follicular fluid transiently increased the transcript levels of IL8 (Montrer IL8 Anticorps) and PTGS2 (Montrer PTGS2 Anticorps), and decreased the expression of SOD2 (Montrer SOD2 Anticorps), GPX3 (Montrer GPX3 Anticorps), DAB2 (Montrer DAB2 Anticorps), and NR3C1 (Montrer NR3C1 Anticorps). TNF and IL6 (Montrer IL6 Anticorps) levels were also decreased while those of NAMPT (Montrer NAMPT Anticorps) were unaffected.
these findings provide the direct evidence that ADAM17 (Montrer ADAM17 Anticorps) cleaves porcine TNFalpha, which represents a new view for identifying potential therapeutic targets in anti-porcine reproductive and respiratory syndrome virus therapy
TMZ pretreatment effectively reduced the myocardial damage caused by CME via inhibiting the PDCD4 (Montrer PDCD4 Anticorps)/NF-kappaB (Montrer NFKB1 Anticorps)/ TNF-alpha pathway in cardiomyocytes.
TNF-alpha, secreted from activated Monocytes, mediates the downregulation of OTX2 (Montrer OTX2 Anticorps) and essential retinal pigment epithelium genes.
TNF-alpha induced MMP-13 (Montrer MMP13 Anticorps) expression by condylar cells might be involved in the degradation of the juvenile condyle.
TNF-alpha was able to promote theca interna cell proliferation. Our results suggest that TNF-alpha might play a role in hyperandrogenism, cortex thickness, and the increased ovary volume observed in polycystic ovaries.
This study showed that the -791(C-->T) mutation of the TNF-alpha gene could be considered an important potential genetic marker of enterotoxigenic Escherichia coli F18 (Montrer MAMLD1 Anticorps) resistance.
Both Nsp1beta and Nsp11 of porcine reproductive and respiratory syndrome virus were demonstrated to be responsible for the inhibitory effect on TNF-alpha production in pulmonary alveolar macrophages.
Study presents evidence demonstrating a single species of exotoxin ApxI, derived from A. pleuropneumoniae serotype 10, induces the expression and production of proinflammatory cytokines IL-1beta (Montrer IL1B Anticorps), IL-8 (Montrer IL8 Anticorps) and TNF-alpha in porcine alveolar macrophages.
the role of miR (Montrer MYLIP Anticorps)-181a in adipocyte differentiation by regulation of TNF-alpha
The NOS inhibitor, L-NMMA, significantly suppressed the combined effects of HT and CORM-2 on TNFalpha-triggered NFkappaBp65 phosphorylation as well as decreased cell viability.
Mecp2 is required for tnfa expression during zebrafish development and inflammation.
Using in vivo, time-lapse imaging we show that as centrally-projecting pioneer axons from dorsal root ganglia (DRG (Montrer COL4A1 Anticorps)) enter the spinal cord, they initiate expression of the cytokine TNFalpha. This induction coincides with ensheathment of these axons by associated glia via a TNF receptor 2 (TNFR2 (Montrer TNFRSF1B Anticorps))-mediated process.
The results reveal a crucial role for TNFalpha/TNFR2 (Montrer TNFRSF1B Anticorps) axis in the protection of the skin against DUOX1 (Montrer DUOX1 Anticorps)-mediated oxidative stress.
proinflammatory cytokines IL-1 (Montrer IL1A Anticorps)/TNFalpha trigger a novel antiviral mechanism involving AID to regulate host cell permissiveness to HBV infection.
TNF alpha is the first molecule identified that is produced by dying retinal neurons and is necessary to induce Muller glia to proliferate in the zebrafish retinal regeneration response.
The data suggest that Mycobacterium marinum SecA2 modulates adaptive immunity to promote granuloma stability, perhaps through induction of tumor necrosis factor alpha.
The data suggest that during infectious pancreatic necrosis virus infection, the expression of cytokines and metalloproteinases might be initiated through the TNFalpha/NF-kappaB (Montrer NFKB1 Anticorps)-mediated pathway.
Infectious pancreatic necrosis virus triggers two death pathways via up-stream induction of the pro-inflammatory cytokine TNFalpha.
Antisense knockdown of tnfa rescued hepatic steatosis and liver degeneration in dtp (Montrer AHCY Anticorps) larvae, whereas the overexpression of tnfa and the hepatic phenotype were unchanged in dtp (Montrer AHCY Anticorps) larvae.
TNF is not required for tuberculous granuloma formation, but maintains granuloma integrity indirectly by restricting mycobacterial growth within macrophages and preventing their necrosis.
These interactions result in the induction of the TNF signaling pathway, activation of apoptosis, and DNA-damage stress response.
Findings showed that both mucosal compartments harbor similar percentages of memory CD4 (Montrer CD4 Anticorps)(+) T cells and displayed comparable cytokine TNF-alpha responses to mitogenic stimulations prior to infection.
Primary role for IL-1beta (Montrer IL1B Anticorps) and TNF-alpha in the triggering of preterm labor associated with inflammation or infection.
The results obtained showed that IL-6 (Montrer IL6 Anticorps) expression in adipose tissue biopsies derived from animals with equine metabolic syndrome was enhanced while TNF-alpha levels of both groups were comparable.
Data indicate that tumor necrosis factor-alpha production is closely related to ovarian steroid actions.
These results indicate that in equine corpus luteum, cytokines TNF, IFNG (Montrer IFNG Anticorps) and FASL (Montrer FASL Anticorps) regulate nitric oxide activity, via eNOS (Montrer NOS3 Anticorps) expression modulation.
These data show the presence of cytokines TNF and IFNG (Montrer IFNG Anticorps), and their receptors, in the equine corpus luteum and indicate their potential involvement in regulation of luteal function.
The contribution of bronchial epithelium to airway inflammation, with focus on mRNA and protein expression of IL-6 (Montrer IL6 Anticorps), IL-10 (Montrer IL10 Anticorps) and TNF-alpha, in horses with recurrent airway obstruction during exacerbation and in remission is reported.
TUNEL staining was positively correlated with TNF-a protein expression. Our findings suggest that apoptosis can be induced in the vocal fold epithelium after 120min of modal intensity phonation. In contrast, shorter durations of vibration exposure do not result in apoptosis signaling.
Inflammatory factors such as TNF-alpha can stimulate MMP-2 (Montrer MMP2 Anticorps)/9 activity in corneal epithelium cells. This may be a potential manipulating mechanism of MMP expression in the pathogenesis of corneal diseases.
The JNK (Montrer MAPK8 Anticorps) pathway plays an important role in mechanical ventilation-stimulated TNF-alpha expression in alveolar macrophages, but the injury-stimulated IL-8 (Montrer IL8 Anticorps) expression may be regulated by other signaling pathways.
The TNFalpha-evoked Cl- current.
IL-1beta (Montrer IL1B Anticorps) and TNF-alpha expression increases significantly during acute lung injury. Ambroxol combined with low-dose heparin inhibits teh release of IL-1beta (Montrer IL1B Anticorps) and TNF-alpha.
Hypercapnia increased expression of TNFa and decreased expression of NFKB in acute lung injury models.
In the early stages of myocardial ischemia, bone marrow stem cells are mobilized and home to ischemic myocardium with a concomitant increase in expression of cytokines VEGF (Montrer VEGFA Anticorps) and TNFalpha.
Ammonium perchlorate can increase gene expressions of types I, III collagens, TGF-beta(1 (Montrer TGFB1 Anticorps)) and TNF-alpha in lung of rabbits.
TNFalpha may either be directly or indirectly involved in vascular damage following an embolic stroke. Moreover, TNFalpha may mediate some of the detrimental effects of tPA (Montrer PLAT Anticorps) on the vascular compartment
Data indicate that three single-nucleotide polymorphisms were identified in tumor necrosis factor-alpha (TNF-alpha) gene.
Macrophage- and neutrophil-derived TNF-alpha instructs skin langerhans cells to prime antiviral immune responses.
This gene encodes a multifunctional proinflammatory cytokine that belongs to the tumor necrosis factor (TNF) superfamily. This cytokine is mainly secreted by macrophages. It can bind to, and thus functions through its receptors TNFRSF1A/TNFR1 and TNFRSF1B/TNFBR. This cytokine is involved in the regulation of a wide spectrum of biological processes including cell proliferation, differentiation, apoptosis, lipid metabolism, and coagulation. This cytokine has been implicated in a variety of diseases, including autoimmune diseases, insulin resistance, and cancer. Knockout studies in mice also suggested the neuroprotective function of this cytokine.
, TNF, macrophage-derived
, TNF, monocyte-derived
, tumor necrosis factor ligand superfamily member 2
, tumor necrosis factor-alpha
, tumor necrosis factor (TNF superfamily, member 2)
, tumor necrosis factor alpha
, tumor necrosis factor
, tumor-necrosis factor
, tumour necrosis factor
, TNF alpha
, ATP-binding cassette, sub-family F (GCN20), member 1
, tumour necrosis factor alpha
, tumor necrosis factor, alpha
, Tumor necrosis factor ligand superfamily member 2
, TNF-alpha 1