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anti-Human Tumor Necrosis Factor Anticorps:
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Mouse (Murine) Polyclonal Tumor Necrosis Factor Primary Antibody pour CyTOF, ELISA (Capture) - ABIN4900489
Ramkhelawon, Hennessy, Ménager, Ray, Sheedy, Hutchison, Wanschel, Oldebeken, Geoffrion, Spiro, Miller, McPherson, Rayner, Moore: Netrin-1 promotes adipose tissue macrophage retention and insulin resistance in obesity. dans Nature medicine 2014
Show all 53 Pubmed References
Mouse (Murine) Polyclonal Tumor Necrosis Factor Primary Antibody pour WB - ABIN3043313
Zhang, Liu, Goldstein, Li, Ge, He, Fei, Wang, Ruiz: Role of the JAK/STAT signaling pathway in the pathogenesis of acute myocardial infarction in rats and its effect on NF-?B expression. dans Molecular medicine reports 2013
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Human Polyclonal Tumor Necrosis Factor Primary Antibody pour IHC (p), WB - ABIN4886747
Liu, Shan, Dong, Liu, Ma, Liu: Combined early fluid resuscitation and hydrogen inhalation attenuates lung and intestine injury. dans World journal of gastroenterology 2013
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Rat (Rattus) Polyclonal Tumor Necrosis Factor Primary Antibody pour WB - ABIN3042522
Qin, Ma, Yang, Hu, Zhou, Fu, Tian, Liu, Xu, Shen: A Triterpenoid Inhibited Hormone-Induced Adipocyte Differentiation and Alleviated Dexamethasone-Induced Insulin Resistance in 3T3-L1 adipocytes. dans Natural products and bioprospecting 2015
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Mouse (Murine) Polyclonal Tumor Necrosis Factor Primary Antibody pour ELISA, IHC (p) - ABIN3042817
Kan, Zhou, Jin, Yang: Effects of PDTC on NF-?B expression and apoptosis in rats with severe acute pancreatitis-associated lung injury. dans International journal of clinical and experimental medicine 2015
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Human Monoclonal Tumor Necrosis Factor Primary Antibody pour - ABIN1383955
Hervé, Racadot, Wijdenes, Flesch, Tiberghien, Bordigoni, Holler, Powles, Bourdeau, Wilmer: Monoclonal anti TNF alpha antibody in the treatment of acute GvHD refractory both to corticosteroids and anti IL-2 R antibody. dans Bone marrow transplantation 1991
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Human Polyclonal Tumor Necrosis Factor Primary Antibody pour ICC, IF - ABIN446837
Kwon, Jung, Hwang, Choi: Evaluation of Bronchiolar and Alveolar Cell Injuries Induced by Short- and Long-term Exposure to Sidestream Smoke. dans Korean journal of pathology 2012
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Human Polyclonal Tumor Necrosis Factor Primary Antibody pour IF (p), IHC (p) - ABIN677318
Morishita, Oku, Horie, Tonari, Kida, Okubo, Sugiyama, Takai, Hara, Ikeda: Systemic simvastatin rescues retinal ganglion cells from optic nerve injury possibly through suppression of astroglial NF-?B activation. dans PLoS ONE 2014
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Human Polyclonal Tumor Necrosis Factor Primary Antibody pour ELISA, ICC - ABIN6269443
Feng, Cui, Gao, Li, Li, Jiang, Tian, Wang, Cui, Cui: Resveratrol attenuates neuronal autophagy and inflammatory injury by inhibiting the TLR4/NF-κB signaling pathway in experimental traumatic brain injury. dans International journal of molecular medicine 2016
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Human Polyclonal Tumor Necrosis Factor Primary Antibody pour IHC (p), IHC - ABIN153474
Roth, Städler, Lemann, Hösli, Simon, Simon: Distinct eosinophil cytokine expression patterns in skin diseases - the possible existence of functionally different eosinophil subpopulations. dans Allergy 2011
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suggest that cardamonin inhibits TNF-alpha, IL-6 (Montrer IL6 Anticorps) and NO production and downregulates iNOS (Montrer NOS2 Anticorps) expression and NF-kappaB (Montrer NFKB1 Anticorps) activation.
Genomic DNA samples extracted from the blood of 107 malaria-infected patients and 190 uninfected controls were analyzed, and a significant difference in the genotypic (p = 2.20E-16) and allelic frequencies (p = 2.20E-16) of the TNF-alpha (snp rs1800629) polymorphism was found.
Data suggest that oxidative stress in PCOS (polycystic ovary syndrome) is associated with ovarian inflammation; biomarkers of oxidative stress [IL6 (Montrer IL6 Anticorps), interleukin-6 (Montrer IL6 Anticorps); IL8 (Montrer IL8 Anticorps), interleukin-8 (Montrer IL8 Anticorps); TNFA, tumor necrosis factor-alpha] are up-regulated in follicular fluid of women with PCOS as compared to control subjects.
regulation of OSCAR by TNF-alpha and receptor activator of NF kappa beta ligand (RANKL (Montrer TNFSF11 Anticorps)) in pre-osteoclasts/osteoclasts
TNF-alpha -238G>A polymorphism was involved in susceptibility to spinal tuerculosis, but not to pulmonary tuberculosis in Chinese polulation.
The baseline concentrations of IL-6 (Montrer IL6 Anticorps) and IL-1beta (Montrer IL1B Anticorps) were significantly higher in the Major Depressive Disorder group than those in the control group. There was no significant difference of TNF-alpha between the two groups.
The -221G>C polymorphism of MBL2 (Montrer MBL2 Anticorps), the -159C>T polymorphism of CD14 (Montrer NDUFA2 Anticorps) and the TNF-857 polymorphism of TNF-a are risk factors for spinal spinal tuberculosis (TB) and may be involved in the development of spinal TB in the Chinese population. These factors are indicators of susceptibility to spinal TB and require clinical attention.
effect of IL-22 (Montrer IL22 Anticorps) on Intestinal Epithelial Cells responses may not be in inducing CXCL8 (Montrer IL8 Anticorps) by itself, but in enhancing TNF-alpha- and IL-1 (Montrer IL1A Anticorps)-induced CXCL8 (Montrer IL8 Anticorps) secretion to augment the contribution of IECs to local inflammatory responses.
Pro-inflammatory proteins S100A9 (Montrer S100A9 Anticorps) and tumor necrosis factor-alpha suppress erythropoietin (Montrer EPO Anticorps) elaboration in myelodysplastic syndromes
Bio informatics analysis of 982 lung cancer patients revealed that higher expression of TNF- alpha was associated with low risk of cancer progression while overexpression of IGF-1 (Montrer IGF1 Anticorps) was correlated with high risk. Collectively, these results reveal that the cytokines in the tumor microenvironment differentially modulate radiation therapy through a variety of signaling mechanisms.
Results demonstrate a critical role for the TRPM2 (Montrer TRPM2 Anticorps) channel in Abeta42-induced microglial activation and generation of TNF-alpha: PKC (Montrer PKC Anticorps)/NOX-mediated generation of ROS (Montrer ROS1 Anticorps) and activation of PARP-1 (Montrer PARP1 Anticorps) are required for Abeta42-induced TRPM2 (Montrer TRPM2 Anticorps) channel activation and, furthermore, the PYK2 (Montrer PTK2B Anticorps)/MEK (Montrer MDK Anticorps)/ERK (Montrer EPHB2 Anticorps) signaling pathway as a positive feedback mechanism downstream of TRPM2 (Montrer TRPM2 Anticorps) channel activation facilitates further activation of PARP-1 (Montrer PARP1 Anticorps) and TRPM2 (Montrer TRPM2 Anticorps) ...
TNFalpha may act reciprocally with DRA, leading to the development of intestinal inflammation.
TNF-alpha plays a pivotal role in the development of nonalcoholic fatty liver disease and progression to nonalcoholic steatohepatitis.
Cross-fostering and conditional knockout experiments indicated that a TNF-alpha deficit in the maternal brain, rather than in the hematopoietic system, and during gestation was responsible for the low-fear offspring phenotype.
In a retinitis pigmentosa mouse model, TrkC (Montrer NTRK3 Anticorps) activity generates phosphorylated Erk (Montrer EPHB2 Anticorps), which upregulates glial TNF-alpha, causing selective neuronal death.
genome-wide knockdown of 19 ribosomal proteins resulted in decreased IL-10 (Montrer IL10 Anticorps) and increased TNF-alpha production.
We conclude that one of the possible regulatory mechanisms of TNF in mechanical orofacial hyperalgesia involves upregulation of the nociceptor TRPV1 (Montrer TRPV1 Anticorps)
The work highlighted the modulatory role of miR (Montrer MLXIP Anticorps)-105 in TNF-alpha-induced epithelial-mesenchymal transition and promoting colorectal cancer metastasis.
This study demonstrated that TNF-alpha genetic deletion ameliorates the amyloid phenotype of the 5XFAD mouse model of AD. 5XFAD/TNF-alpha-/- mice exhibit significantly decreased amyloid deposition and reduced levels of AbetaPP-CTFs and amyloid-beta protein.
Data suggest that expression of Tnfa in adipocytes can be regulated by dietary fatty acids; here, polyunsaturated fatty acids regulate Tnfa expression via alteration in methylation of Tnfa promoter in rats fed polyunsaturated fatty acids (safflower oil versus coconut/olive oil) and in mouse adipocyte cell line incubated with polyunsaturated fatty acid (linoleic acid versus palmitic/oleic acids).
Data suggest that luteolytic factors (such as TNFa, interferon gamma (Montrer IFNG Anticorps), and PGF2a) control expression of MMP1 (Montrer MMP1 Anticorps), other matrix metalloproteinases, and tissue inhibitors of metalloproteinase in cultured luteal cells.
These results suggest that polymorphism of the TNFalpha-824 A>G gene and mTNFalpha protein expression play an important role in the pathogenesis of enzootic bovine leukosis.
These results suggest that the endometrium might lower the TNF concentration in the blastocyst by (1) regulating TNF secretion into the uterine fluid and (2) inducing decreased TNF and TNFR2 (Montrer TNFRSF1B Anticorps) mRNA transcription in the embryo.
There was a significant association between the proviral load and a low frequency of the G/G genotype of TNF-alpha at position -824.
SNP in the TNF-alpha gene affects immune function and reproductive performance in dairy cows.
Studied genotypic and expression profiling of partial TNF-alpha gene and its association with mastitis susceptibility in 129 crossbred cattle.
Messenger RNA and protein levels of prostaglandin (PG) E synthase (PGES (Montrer PTGES Anticorps)), PGF2alpha receptor (PGFR), tumor necrosis factor-alpha (TNF) and Fas (Montrer FAS Anticorps) were found to be higher in the corpus luteum of pregnancy than in corpus luteum of the cycle.
TNF-alpha up-regulates NaV1.7 (Montrer SCN9A Anticorps) mRNA in both adrenal chromaffin cells and dorsal root ganglia (DRG) neurons, highlighting the peripheral nociceptive mechanism of TNF-alpha
These results provide evidence for a high prevalence of subclinical endometritis in repeat breeding cows as well as the involvement of TNFalpha and iNOS (Montrer NOS2 Anticorps) pathways in the regulation of this pathological condition.
Exposure to follicular fluid transiently increased the transcript levels of IL8 (Montrer IL8 Anticorps) and PTGS2 (Montrer PTGS2 Anticorps), and decreased the expression of SOD2 (Montrer SOD2 Anticorps), GPX3 (Montrer GPX3 Anticorps), DAB2 (Montrer DAB2 Anticorps), and NR3C1 (Montrer NR3C1 Anticorps). TNF and IL6 (Montrer IL6 Anticorps) levels were also decreased while those of NAMPT (Montrer NAMPT Anticorps) were unaffected.
these findings provide the direct evidence that ADAM17 (Montrer ADAM17 Anticorps) cleaves porcine TNFalpha, which represents a new view for identifying potential therapeutic targets in anti-porcine reproductive and respiratory syndrome virus therapy
TMZ pretreatment effectively reduced the myocardial damage caused by CME via inhibiting the PDCD4 (Montrer PDCD4 Anticorps)/NF-kappaB (Montrer NFKB1 Anticorps)/ TNF-alpha pathway in cardiomyocytes.
TNF-alpha, secreted from activated Monocytes, mediates the downregulation of OTX2 (Montrer OTX2 Anticorps) and essential retinal pigment epithelium genes.
TNF-alpha induced MMP-13 (Montrer MMP13 Anticorps) expression by condylar cells might be involved in the degradation of the juvenile condyle.
TNF-alpha was able to promote theca interna cell proliferation. Our results suggest that TNF-alpha might play a role in hyperandrogenism, cortex thickness, and the increased ovary volume observed in polycystic ovaries.
This study showed that the -791(C-->T) mutation of the TNF-alpha gene could be considered an important potential genetic marker of enterotoxigenic Escherichia coli F18 (Montrer MAMLD1 Anticorps) resistance.
Both Nsp1beta and Nsp11 of porcine reproductive and respiratory syndrome virus were demonstrated to be responsible for the inhibitory effect on TNF-alpha production in pulmonary alveolar macrophages.
Study presents evidence demonstrating a single species of exotoxin ApxI, derived from A. pleuropneumoniae serotype 10, induces the expression and production of proinflammatory cytokines IL-1beta (Montrer IL1B Anticorps), IL-8 (Montrer IL8 Anticorps) and TNF-alpha in porcine alveolar macrophages.
the role of miR (Montrer MYLIP Anticorps)-181a in adipocyte differentiation by regulation of TNF-alpha
The NOS (Montrer NOS Anticorps) inhibitor, L-NMMA, significantly suppressed the combined effects of HT and CORM-2 on TNFalpha-triggered NFkappaBp65 phosphorylation as well as decreased cell viability.
Mecp2 is required for tnfa expression during zebrafish development and inflammation.
Using in vivo, time-lapse imaging we show that as centrally-projecting pioneer axons from dorsal root ganglia (DRG (Montrer COL4A1 Anticorps)) enter the spinal cord, they initiate expression of the cytokine TNFalpha. This induction coincides with ensheathment of these axons by associated glia via a TNF receptor 2 (TNFR2 (Montrer TNFRSF1B Anticorps))-mediated process.
The results reveal a crucial role for TNFalpha/TNFR2 (Montrer TNFRSF1B Anticorps) axis in the protection of the skin against DUOX1 (Montrer DUOX1 Anticorps)-mediated oxidative stress.
proinflammatory cytokines IL-1 (Montrer IL1A Anticorps)/TNFalpha trigger a novel antiviral mechanism involving AID to regulate host cell permissiveness to HBV infection.
TNF alpha is the first molecule identified that is produced by dying retinal neurons and is necessary to induce Muller glia to proliferate in the zebrafish retinal regeneration response.
The data suggest that Mycobacterium marinum SecA2 modulates adaptive immunity to promote granuloma stability, perhaps through induction of tumor necrosis factor alpha.
The data suggest that during infectious pancreatic necrosis virus infection, the expression of cytokines and metalloproteinases might be initiated through the TNFalpha/NF-kappaB (Montrer NFKB1 Anticorps)-mediated pathway.
Infectious pancreatic necrosis virus triggers two death pathways via up-stream induction of the pro-inflammatory cytokine TNFalpha.
Antisense knockdown of tnfa rescued hepatic steatosis and liver degeneration in dtp (Montrer AHCY Anticorps) larvae, whereas the overexpression of tnfa and the hepatic phenotype were unchanged in dtp (Montrer AHCY Anticorps) larvae.
TNF is not required for tuberculous granuloma formation, but maintains granuloma integrity indirectly by restricting mycobacterial growth within macrophages and preventing their necrosis.
These interactions result in the induction of the TNF signaling pathway, activation of apoptosis, and DNA-damage stress response.
Findings showed that both mucosal compartments harbor similar percentages of memory CD4 (Montrer CD4 Anticorps)(+) T cells and displayed comparable cytokine TNF-alpha responses to mitogenic stimulations prior to infection.
Primary role for IL-1beta (Montrer IL1B Anticorps) and TNF-alpha in the triggering of preterm labor associated with inflammation or infection.
The results obtained showed that IL-6 (Montrer IL6 Anticorps) expression in adipose tissue biopsies derived from animals with equine metabolic syndrome was enhanced while TNF-alpha levels of both groups were comparable.
Data indicate that tumor necrosis factor-alpha production is closely related to ovarian steroid actions.
These results indicate that in equine corpus luteum, cytokines TNF, IFNG (Montrer IFNG Anticorps) and FASL (Montrer FASL Anticorps) regulate nitric oxide activity, via eNOS (Montrer NOS3 Anticorps) expression modulation.
These data show the presence of cytokines TNF and IFNG (Montrer IFNG Anticorps), and their receptors, in the equine corpus luteum and indicate their potential involvement in regulation of luteal function.
The contribution of bronchial epithelium to airway inflammation, with focus on mRNA and protein expression of IL-6 (Montrer IL6 Anticorps), IL-10 (Montrer IL10 Anticorps) and TNF-alpha, in horses with recurrent airway obstruction during exacerbation and in remission is reported.
TUNEL staining was positively correlated with TNF-a protein expression. Our findings suggest that apoptosis can be induced in the vocal fold epithelium after 120min of modal intensity phonation. In contrast, shorter durations of vibration exposure do not result in apoptosis signaling.
Inflammatory factors such as TNF-alpha can stimulate MMP-2 (Montrer MMP2 Anticorps)/9 activity in corneal epithelium cells. This may be a potential manipulating mechanism of MMP expression in the pathogenesis of corneal diseases.
The JNK (Montrer MAPK8 Anticorps) pathway plays an important role in mechanical ventilation-stimulated TNF-alpha expression in alveolar macrophages, but the injury-stimulated IL-8 (Montrer IL8 Anticorps) expression may be regulated by other signaling pathways.
The TNFalpha-evoked Cl- current.
IL-1beta (Montrer IL1B Anticorps) and TNF-alpha expression increases significantly during acute lung injury. Ambroxol combined with low-dose heparin inhibits teh release of IL-1beta (Montrer IL1B Anticorps) and TNF-alpha.
Hypercapnia increased expression of TNFa and decreased expression of NFKB in acute lung injury models.
In the early stages of myocardial ischemia, bone marrow stem cells are mobilized and home to ischemic myocardium with a concomitant increase in expression of cytokines VEGF (Montrer VEGFA Anticorps) and TNFalpha.
Ammonium perchlorate can increase gene expressions of types I, III collagens, TGF-beta(1 (Montrer TGFB1 Anticorps)) and TNF-alpha in lung of rabbits.
TNFalpha may either be directly or indirectly involved in vascular damage following an embolic stroke. Moreover, TNFalpha may mediate some of the detrimental effects of tPA (Montrer PLAT Anticorps) on the vascular compartment
Data indicate that three single-nucleotide polymorphisms were identified in tumor necrosis factor-alpha (TNF-alpha) gene.
Macrophage- and neutrophil-derived TNF-alpha instructs skin langerhans cells to prime antiviral immune responses.
This gene encodes a multifunctional proinflammatory cytokine that belongs to the tumor necrosis factor (TNF) superfamily. This cytokine is mainly secreted by macrophages. It can bind to, and thus functions through its receptors TNFRSF1A/TNFR1 and TNFRSF1B/TNFBR. This cytokine is involved in the regulation of a wide spectrum of biological processes including cell proliferation, differentiation, apoptosis, lipid metabolism, and coagulation. This cytokine has been implicated in a variety of diseases, including autoimmune diseases, insulin resistance, and cancer. Knockout studies in mice also suggested the neuroprotective function of this cytokine.
, TNF, macrophage-derived
, TNF, monocyte-derived
, tumor necrosis factor ligand superfamily member 2
, tumor necrosis factor-alpha
, tumor necrosis factor (TNF superfamily, member 2)
, tumor necrosis factor alpha
, tumor necrosis factor
, tumor-necrosis factor
, tumour necrosis factor
, TNF alpha
, ATP-binding cassette, sub-family F (GCN20), member 1
, tumour necrosis factor alpha
, LPS-induced TNF-alpha factor homolog
, TNF-alpha factor
, lipid-induced TNF-alpha factor
, lipopolysaccharide-induced tumor necrosis factor-alpha factor homolog
, small integral membrane protein of lysosome/late endosome
, tissue necrosis factor-alpha
, tumor necrosis factor, alpha
, Tumor necrosis factor ligand superfamily member 2
, TNF-alpha 1