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anti-Human C-MYC Anticorps:
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Chicken Monoclonal C-MYC Primary Antibody pour ChIP, CyTOF - ABIN152253
Locker, Dowle, Ellis, Elston, Blamey, Sikora, Evan, Robins: c-myc oncogene product expression and prognosis in operable breast cancer. dans British journal of cancer 1989
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Human Monoclonal C-MYC Primary Antibody pour FACS, IHC (p) - ABIN302017
Veracini, Simon, Richard, Schraven, Horejsi, Roche, Benistant: The Csk-binding protein PAG regulates PDGF-induced Src mitogenic signaling via GM1. dans The Journal of cell biology 2008
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Human Monoclonal C-MYC Primary Antibody pour FACS, IHC (p) - ABIN302092
Wang, Campoli, Ko, Luo, Ferrone: Enhancement of scFv fragment reactivity with target antigens in binding assays following mixing with anti-tag monoclonal antibodies. dans Journal of immunological methods 2004
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All Species Monoclonal C-MYC Primary Antibody pour FACS, IP - ABIN2749043
Persson, Hennighausen, Taub, DeGrado, Leder: Antibodies to human c-myc oncogene product: evidence of an evolutionarily conserved protein induced during cell proliferation. dans Science (New York, N.Y.) 1984
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Human Polyclonal C-MYC Primary Antibody pour ELISA, ICC - ABIN6263457
Tang, Peng, Huang, Xie, Chen, Shen, Gao, You, Xie, Chen: Neoisoliquiritigenin Inhibits Tumor Progression by Targeting GRP78-β- catenin Signaling in Breast Cancer. dans Current cancer drug targets 2018
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Human Monoclonal C-MYC Primary Antibody pour FACS, IHC (p) - ABIN536092
Fujiwara, Poikonen, Aleman, Valtavaara, Saksela, Mayer: A single-chain antibody/epitope system for functional analysis of protein-protein interactions. dans Biochemistry 2002
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Human Monoclonal C-MYC Primary Antibody pour IHC (fro), IF - ABIN2477762
Quant, Woo: Normal values of eye position in the Chinese population of Hong Kong. dans Optometry and vision science : official publication of the American Academy of Optometry 1992
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Human Monoclonal C-MYC Primary Antibody pour IF, IHC (p) - ABIN533219
Huang, Bredemeyer, Walker, Bassing, Sleckman: Dynamic regulation of c-Myc proto-oncogene expression during lymphocyte development revealed by a GFP-c-Myc knock-in mouse. dans European journal of immunology 2008
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Human Monoclonal C-MYC Primary Antibody pour IF, WB - ABIN3201011
Khanna, Böckelman, Hemmes, Junttila, Wiksten, Lundin, Junnila, Murphy, Evan, Haglund, Westermarck, Ristimäki: MYC-dependent regulation and prognostic role of CIP2A in gastric cancer. dans Journal of the National Cancer Institute 2009
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Menin functions as an oncogenic regulatory factor that is critical for MYC-mediated gene transcription.
High c-myc expression is associated with colorectal cancer.
Melatonin disturbs SUMOylation-mediated crosstalk between c-Myc and nestin (Montrer NES Anticorps) via MT1 (Montrer MT1A Anticorps) activation and promotes the sensitivity of paclitaxel in brain cancer stem cells.
FBP1 (Montrer FBP1 Anticorps) modulates the sensitivity of pancreatic cancer cells to BET inhibitors by decreasing the expression of c-Myc. These findings highlight FBP1 (Montrer FBP1 Anticorps) could be used as a therapeutic niche for patient-tailored therapies
miR135a directly bound to UCA1 and the 3' untranslated region of cmyc, and UCA1 competed with cmyc for miR135a binding.
MYC directly regulates DANCR and plays important role in cancer cell proliferation.
In this review, we provide support to the hypothesis that the cooperation of c-Myc with transcriptional cofactors mediates c-Myc-induced cellular functions. We produce evidence that recently identified cofactors are involved in c-Myc control of survival mechanisms of cancer cells
4-chlorobenzoyl berbamine (CBBM (Montrer OPN1MW Anticorps)) inhibits the JAK2 (Montrer JAK2 Anticorps)/STAT3 (Montrer STAT3 Anticorps) pathway, leading to reduced c-Myc transcription. Collectively, these findings suggest that CBBM (Montrer OPN1MW Anticorps) could be a promising lead compound for treatment of c-Myc-driven diffuse large B cell lymphoma.
Results revealed that C-MYC protein is highly expressed in colon cancer tissues, mainly in the cell nucleus and was identified as a direct target for mir (Montrer MLXIP Anticorps)-184. C-MYC appeared to participate in cell cycle regulation and malignant transformation to colon cancer.
MACC1 (Montrer MACC1 Anticorps) and c-Myc are highly expressed in serum and tumor tissues of EC patients. Both are correlated with TNM (Montrer ODZ1 Anticorps) stage, primary infiltration, and lymph node or distal metastasis.
Ouabain-induced proliferation might be attributed, at least in part, to decrease of intracellular free calcium and increase of c-myc mRNA expression, and that may be directly or indirectly involved in regulation of blood pressure.
report the isolation of complete coding regions of rabbit SOX2, KLF4, C-MYC and NANOG, which encode transcription factors that play crucial regulatory roles during early mammalian embryonic development
c-Myc is essential for tumor initiation, maintenance, and metastasis.
Genomic characterization of Emu-Myc mouse lymphomas identifies Bcor (Montrer BCOR Anticorps) as a Myc cooperative tumor-suppressor gene.
The data supports an indispensable role for Mule in cardiac homeostasis through the regulation of mitochondrial function via maintenance of Pgc-1alpha and Pink1 (Montrer PINK1 Anticorps) expression and persistent negative regulation of c-Myc.
MYC binding is enriched at neuroendocrine genes in tumor cells and loss of MYC reduces ductal-neuroendocrine lineage heterogeneity, while deregulated MYC expression in KRAS mutants increases this phenotype.
Although either BCR (Montrer BCR Anticorps) or CD40 (Montrer CD40 Anticorps) ligation induced c-Myc in naive B cells, both signals were required to highly induce c-Myc, a critical mediator of GC B (Montrer NPR2 Anticorps) cell survival and cell cycle reentry.
Myc is a component that links neuromesodermal progenitors maintenance and pre-somitic mesoderm maturation during the body axis elongation stages of mouse embryogenesis.
Myc potentiates the Wnt (Montrer WNT2 Anticorps)/beta-catenin (Montrer CTNNB1 Anticorps) signalling pathway, which cooperates with the transcriptional regulatory network in sustaining embryonic stem cell self-renewal.
Although mnt heterozygosity clearly slowed lymphomagenesis in vavP-MYC10 and Emu-myc mice, the change(s) in cellular properties responsible for this effect remain to be identified.
clusters of enhancers, such as BENC in the myc gene, form highly combinatorial systems that allow precise control of gene expression across normal cellular hierarchies and which also can be hijacked in malignancies
Conditional deletion of Myc in hyaloid vascular endothelial cells suppressed both proliferation and cell death.
Methylparabens exposure increased malformations, LPO, apoptosis, ccnd1 and myca expressions, and decreased GST activities and NO levels compared with the control group.
Apoptosis was also observed with myca expression; introduction of homozygous tp53 (Montrer TP53 Anticorps)(-/-) mutation into the myca transgenic fish reduced apoptosis and accelerated tumor progression.
MYC down-regulation induces mitochondrial apoptosis in T lymphoblasts.
These findings not only reveal a novel role of Mad1 (Montrer MXD1 Anticorps) in regulating developmental cell death but also suggest that a balance of Mad and Myc controls cell fate determination during adult organ development.
Thyroid hormone (Montrer PTH Anticorps) activates protein arginine methyltransferase 1 expression by directly inducing c-Myc transcription during Xenopus intestinal stem cell development.(
c-Myc has a direct role in the control of DNA replication
Findings support a model in which Myc, Twist and Slug/Snail2 function in a regulatory circuit within lateral plate mesoderm that directs normal vessel formation in both the vascular and lymphatic systems.
Expression of Drosophila Myc (dMyc) suppresses, whereas loss of dMyc enhances, ectopically activated JNK (Montrer MAPK8 Anticorps) signaling-induced cell death. dMyc impedes physiologically activated JNK (Montrer MAPK8 Anticorps) pathway-mediated cell death. Loss of dMyc triggers JNK (Montrer MAPK8 Anticorps) pathway activation and JNK (Montrer MAPK8 Anticorps)-dependent cell death.
tissue-specific downregulation of the Drosophila homolog of human c-myc proto-oncogene (dMyc) suppresses tau-mediated morphological and functional deficits by reducing abnormal tau hyperphosphorylation and restoring the heterochromatin loss.
dMyc has an essential role in preventing JNK (Montrer MAPK8 Anticorps)-mediated retinal glial activation
the key target of the Psi/MED network in controlling developmentally regulated tissue growth is the transcription factor MYC.
Myc dosage plays crucial role in determining sex-specific size in Drosophila larvae and adult tissue. Double dose of Myc in females serves at least twice in development to promote sexual size dimorphism.
BicC (Montrer BICC1 Anticorps) down regulates Myc in the Malpighian tubule.
activation of the TOR-Myc axis in midgut stem and progenitor cells influences a variety of traits in Drosophila
Drosophila adult muscle precursors display homing behavior to muscle niche and the niche-driven Insulin (Montrer INS Anticorps)-Notch (Montrer NOTCH1 Anticorps)-dMyc cascade plays a key role in setting the activated state of adult muscle precursors.
a functional link between Myc, a renowned oncogene (Montrer RAB1A Anticorps), and the essential nucleotide biosynthetic enzyme CTPsyn.
MYC and S6K (Montrer RPS6KB1 Anticorps) cooperate through coordinate activation of the essential Pol I transcription initiation factor TIF-1A (Montrer RRN3 Anticorps).
The protein encoded by this gene is a multifunctional, nuclear phosphoprotein that plays a role in cell cycle progression, apoptosis and cellular transformation. It functions as a transcription factor that regulates transcription of specific target genes. Mutations, overexpression, rearrangement and translocation of this gene have been associated with a variety of hematopoietic tumors, leukemias and lymphomas, including Burkitt lymphoma. There is evidence to show that alternative translation initiations from an upstream, in-frame non-AUG (CUG) and a downstream AUG start site result in the production of two isoforms with distinct N-termini. The synthesis of non-AUG initiated protein is suppressed in Burkitt's lymphomas, suggesting its importance in the normal function of this gene.
avian myelocytomatosis viral oncogene homolog
, class E basic helix-loop-helix protein 39
, myc proto-oncogene protein
, myc-related translation/localization regulatory factor
, proto-oncogene c-Myc
, transcription factor p64
, v-myc myelocytomatosis viral oncogene homolog
, c-myc proto-oncogene
, avian myelocytomatosis viral (v-myc) oncogene homolog
, Avian myelocytomatosis viral (v-myc) oncogene homolog
, myelocytomatosis viral oncogene homolog
, v-myc avian myelocytomatosis viral oncogene homolog
, cellular myelocytomatosis oncogene
, Proto-oncogene c-Myc
, Transcription factor p64
, transcriptional regulator Myc-A
, MYC II
, transcriptional regulator Myc-B
, Myc proto-oncogene protein
, CG10798 gene product from transcript CG10798-RB
, Diminutive protein
, lethal (1) G0354
, lethal (1) G0359