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anti-Human FAS Anticorps:
anti-Mouse (Murine) FAS Anticorps:
anti-Rat (Rattus) FAS Anticorps:
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Mouse (Murine) Polyclonal FAS Primary Antibody pour IHC (fro), IHC (p) - ABIN3044338
Jiang, Li, Zhou, Wang, Zhang, Wang: Colistin-induced apoptosis in PC12 cells: involvement of the mitochondrial apoptotic and death receptor pathways. dans International journal of molecular medicine 2014
Show all 14 Pubmed References
Rat (Rattus) Polyclonal FAS Primary Antibody pour WB - ABIN3042386
Liu, Shan, Dong, Liu, Ma, Liu: Combined early fluid resuscitation and hydrogen inhalation attenuates lung and intestine injury. dans World journal of gastroenterology 2013
Show all 13 Pubmed References
Human Polyclonal FAS Primary Antibody pour IHC (p), WB - ABIN3042387
Qin, Ma, Yang, Hu, Zhou, Fu, Tian, Liu, Xu, Shen: A Triterpenoid Inhibited Hormone-Induced Adipocyte Differentiation and Alleviated Dexamethasone-Induced Insulin Resistance in 3T3-L1 adipocytes. dans Natural products and bioprospecting 2015
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Human Monoclonal FAS Primary Antibody pour IHC (fro), IF - ABIN1106610
Vega, Jazirehi, Huerta-Yepez, Bonavida: Rituximab-induced inhibition of YY1 and Bcl-xL expression in Ramos non-Hodgkin's lymphoma cell line via inhibition of NF-kappa B activity: role of YY1 and Bcl-xL in Fas resistance and chemoresistance, respectively. dans Journal of immunology (Baltimore, Md. : 1950) 2005
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Human Monoclonal FAS Primary Antibody pour FACS - ABIN5541252
Hata, Matsuzaki, Takeya, Yoshida, Sonoki, Nagasaki, Kuribayashi, Kawano, Takatsuki: Expression of Fas/Apo-1 (CD95) and apoptosis in tumor cells from patients with plasma cell disorders. dans Blood 1995
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Human Monoclonal FAS Primary Antibody pour FACS - ABIN5541251
Boirivant, Pica, DeMaria, Testi, Pallone, Strober: Stimulated human lamina propria T cells manifest enhanced Fas-mediated apoptosis. dans The Journal of clinical investigation 1997
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Mouse (Murine) Monoclonal FAS Primary Antibody pour CyTox, FACS - ABIN1177304
Enari, Hug, Nagata: Involvement of an ICE-like protease in Fas-mediated apoptosis. dans Nature 1995
Show all 9 Pubmed References
Mouse (Murine) Monoclonal FAS Primary Antibody pour FACS - ABIN2689463
Hiromatsu, Aoki, Makino, Matsumoto, Mizuochi, Gotoh, Nomoto, Ogasawara, Nagata, Yoshikai: Increased Fas antigen expression in murine retrovirus-induced immunodeficiency syndrome, MAIDS. dans European journal of immunology 1994
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Human Polyclonal FAS Primary Antibody pour IHC, IHC (p) - ABIN448412
Horuz, Göktaş, Çetinel, Akça, Aydın, Ekici, Albayrak, Sarıca: Role of TNF-associated cytokines in renal tubular cell apoptosis induced by hyperoxaluria. dans Urolithiasis 2013
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Mouse (Murine) Monoclonal FAS Primary Antibody pour CyTox, FACS - ABIN2689461
Kägi, Vignaux, Ledermann, Bürki, Depraetere, Nagata, Hengartner, Golstein: Fas and perforin pathways as major mechanisms of T cell-mediated cytotoxicity. dans Science (New York, N.Y.) 1994
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These results demonstrated that overexpression of ING4 (Montrer ING4 Anticorps) can induce the apoptosis of melanoma cells and CD3 (Montrer CD3 Anticorps)+ T cells through signaling pathways such as the Fas/FasL (Montrer FASL Anticorps) pathway, and that ING4 (Montrer ING4 Anticorps) gene therapy for melanoma treatment is a novel approach.
Tag7 (Montrer PGLYRP1 Anticorps) activates lymphocytes capable of Fasl (Montrer FASL Anticorps)-Fas-dependent contact killing of virus-infected cells.
These results indicated that cMyc (Montrer MYC Anticorps) and Fas regulated the sensitivity of A549 cells to irradiation by regulating caspase8-mediated Bid (Montrer BID Anticorps) activation and the subsequent association with the mitochondrial pathway of apoptosis.
this study characterized in juvenile systemic lupus erythematosus a distinct profile from adult SLE that comprises increased sFas, sTRAIL, and reduced sFasL (Montrer FASL Anticorps), notably in patients with active disease and with nephritis.
results from the current study showed that the association of FAS-670A/G SNP with idiopathic azoospermia was not found in a population of men with idiopathic infertility.
The Btk (Montrer BTK Anticorps)-dependent PIP5K1gamma lipid kinase activation by Fas counteracts FasL (Montrer FASL Anticorps)-induced cell death.
Study identify genes highly expressed in Kras knockout lung cancer cells, including the Fas receptor gene. Antibodies that activate Fas receptor selectively induced apoptosis in Kras-independent lung cancer cells suggesting that FAS is involved in KRAS-related drug resistance.
The authors observed differential expression of CD95(Fas) in CD27 (Montrer CD27 Anticorps)(+) B-cells from cirrhotic patients that was inversely correlated with peripheral CD27 (Montrer CD27 Anticorps)(+) B-cell frequency. They conclude that peripheral CD27 (Montrer CD27 Anticorps)(+) memory B-cells in cirrhosis exhibit increased sensitivity to Fas-induced apoptosis in an activation-dependent manner to which endotoxin contributes, associated with reduced frequency of circulating memory B-cells.
the analysis of mRNA level showed that disease progression is associated with significantly increased expression of FasR and/or FasL (Montrer FASL Anticorps). In conclusion, our observation seems to confirm that spherical model of cancer lines is more reliable for some sophisticated analysis because of their greater resemblance to the CSCs from human CRC (Montrer CALR Anticorps) samples in comparison to commonly used adherent cells
study indicates FAS-FASL (Montrer FASL Anticorps) promoter SNPs may promote the production of cross-reactive anti-ganglioside antibodies in GBS (Montrer GNB5 Anticorps)
Messenger RNA and protein levels of prostaglandin (PG) E synthase (PGES (Montrer PTGES Anticorps)), PGF2alpha receptor (PGFR), tumor necrosis factor-alpha (TNF (Montrer TNF Anticorps)) and Fas were found to be higher in the corpus luteum of pregnancy than in corpus luteum of the cycle.
Results did not support that K8/K18 (Montrer KRT18 Anticorps) filaments influence the expression of Fas on the surface of luteal cells.
activation of the Fas pathway and presence of FSH (Montrer BRD2 Anticorps) during in vitro maturation increased the incidence of apoptosis in cumulus cells
demonstrated for the first time that normal ejaculated spermatozoa express Fas antigen (Montrer FASL Anticorps); data showed that a large percentage of normal ejaculated spermatozoa of fertile bulls are immunocytochemically positive for Fas
identification of 14-3-3zeta (Montrer YWHAZ Anticorps) as a novel phosphocofilin binding protein involved in the maintenance of the cellular phosphocofilin pool
Both Sharpin (Montrer SHARPIN Anticorps)/Fas and Sharpin (Montrer SHARPIN Anticorps)/Fasl (Montrer FASL Anticorps) compound mutant mice developed an auto-inflammatory phenotype similar to that seen in Sharpin (Montrer SHARPIN Anticorps) null mice, indicating that initiation of apoptosis by FAS signalling is likely not involved in the pathogenesis of this disease.
leucine deprivation induces the expression of miR (Montrer MLXIP Anticorps)-212-5p in a GCN2 (Montrer EIF2AK4 Anticorps)/ATF4 (Montrer ATF4 Anticorps)-dependent manner. miR (Montrer MLXIP Anticorps)-212-5p suppresses lipid accumulation in liver by targeting FAS and SCD1 (Montrer SCD Anticorps) under both normal diet and high-fat diet conditions.
Our data show that loss of Fas activity strongly affects the early development of atopic dermatitis (AD) by leading to Th2-dominant inflammation characterized by dermal infiltration of CD4 (Montrer CD4 Anticorps)+ T cells, neutrophils and increased skin expression of Th2 cytokines.However, Fas/FasL (Montrer FASL Anticorps)-apoptotic pathway is also involved in restricting tissue remodelling and dermal fibrosis during AD.
Hrd1 (Montrer SYVN1 Anticorps)-null B cells exhibited high Fas expression during activation and rapidly underwent Fas-mediated apoptosis, which could be largely inhibited by FasL (Montrer FASL Anticorps) neutralization. Fas mutation in Hrd1 (Montrer SYVN1 Anticorps) KO mice abrogated the increase in B-cell AICD. We identified Hrd1 (Montrer SYVN1 Anticorps) as the first E3 ubiquitin ligase (Montrer MUL1 Anticorps) of the death receptor Fas and Hrd1 (Montrer SYVN1 Anticorps)-mediated Fas destruction as a molecular mechanism in regulating B-cell immunity.
FAS contributes to mitochondrial dysfunction, steatosis development, and insulin (Montrer INS Anticorps) resistance under high fat diet.
anti-apoptotic molecules BclxL (Montrer BCL2L1 Anticorps) and Bcl-2 (Montrer BCL2 Anticorps) and the pro-apoptotic factors BAD and BID (Montrer BID Anticorps) cooperate to promote migration of triple-negative breast cancer cells stimulated with cl-CD95L (Montrer FASL Anticorps).
These findings reveal a role for MOAP-1 (Montrer MOAP1 Anticorps) in Fas signaling in the liver by promoting MTCH2 (Montrer MTCH2 Anticorps)-mediated tBid recruitment to mitochondria.
The in vivo delivery of CRISPR/Cas9 could maintain liver homeostasis and protect hepatocytes from Fas-mediated cell apoptosis in the fulminant hepatic failure model.
This study demonstrated that Ischemic neurons release sFasL (Montrer FASL Anticorps), which contributes to M1-microglial polarization.
Fas was expressed on fetal pig pancreatic cells, both beta and non-beta cells, and the level of expression could be upregulated by exposure to interleukin 1beta
The expression of FAS and FAS ligand (Montrer FASL Anticorps) in splenic macrophages co-infected with porcine circovirus 2 and porcine reproductive and respiratory syndrome virus is reported
The present results may provide some insights to understand the role of Fas/FasL (Montrer FASL Anticorps) in the spinal cord but not motor cortex with neuronal apoptosis and neuroplasticity in monkeys subjected to hemisection spinal cord injury.
Ligustrazine has notable protective effects on pulmonary ischemia/reperfusion injury inhibiting Fas/FasL (Montrer FASL Anticorps) mRNA express in lung tissue and decreasing apoptosis.
The protein encoded by this gene is a member of the TNF-receptor superfamily. This receptor contains a death domain. It has been shown to play a central role in the physiological regulation of programmed cell death, and has been implicated in the pathogenesis of various malignancies and diseases of the immune system. The interaction of this receptor with its ligand allows the formation of a death-inducing signaling complex that includes Fas-associated death domain protein (FADD), caspase 8, and caspase 10. The autoproteolytic processing of the caspases in the complex triggers a downstream caspase cascade, and leads to apoptosis. This receptor has been also shown to activate NF-kappaB, MAPK3/ERK1, and MAPK8/JNK, and is found to be involved in transducing the proliferating signals in normal diploid fibroblast and T cells. Several alternatively spliced transcript variants have been described, some of which are candidates for nonsense-mediated mRNA decay (NMD). The isoforms lacking the transmembrane domain may negatively regulate the apoptosis mediated by the full length isoform.
APO-1 cell surface antigen
, CD95 antigen
, Delta Fas/APO-1/CD95
, FAS 827dupA
, FASLG receptor
, Fas (TNF receptor superfamily, member 6)
, Fas AMA
, TNF receptor superfamily member 6
, apoptosis antigen 1
, apoptosis-mediating surface antigen FAS
, tumor necrosis factor receptor superfamily member 6
, tumor necrosis factor receptor superfamily, member 6
, Fas antigen
, 14-3-3 protein zeta/delta
, factor activating exoenzyme S
, protein kinase C inhibitor protein 1
, Fas (TNF receptor superfamily member)
, apo-1 antigen
, Fas antigen (ATP1)
, Fas receptor
, Tumor necrosis factor receptor superfamily, member 6
, apoptosis (APO-1) antigen 1 ( FAS ), member 6
, Fas receptor CD95