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anti-Human KIT Ligand Anticorps:
anti-Mouse (Murine) KIT Ligand Anticorps:
anti-Rat (Rattus) KIT Ligand Anticorps:
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Human Polyclonal KIT Ligand Primary Antibody pour IF (p), IHC (fro) - ABIN669936
Goldstein, Goss, Hatzistergos, Rangel, Seidler, Saur, Hare: Adult c-Kit(+) progenitor cells are necessary for maintenance and regeneration of olfactory neurons. dans The Journal of comparative neurology 2014
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Human Polyclonal KIT Ligand Primary Antibody pour ELISA, ICC - ABIN4352192
Reber, Da Silva, Frossard: Stem cell factor and its receptor c-Kit as targets for inflammatory diseases. dans European journal of pharmacology 2006
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Human Polyclonal KIT Ligand Primary Antibody pour Func, IHC (p) - ABIN2476604
Sakhaee, Alpern, Poindexter, Pak: Citraturic response to oral citric acid load. dans The Journal of urology 1992
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Mouse (Murine) Polyclonal KIT Ligand Primary Antibody pour Func, ELISA - ABIN2476605
Chou, Flygare, Lodish: Fetal hepatic progenitors support long-term expansion of hematopoietic stem cells. dans Experimental hematology 2013
Authors show here that cyclin F (Montrer CCNF Anticorps), a substrate receptor F-box protein (Montrer FBXW10 Anticorps) for the SCF (Skp1 (Montrer SKP1 Anticorps)/Cul1 (Montrer CUL1 Anticorps)/F-box) family of E3 ubiquitin ligases, is a bona fide AKT (Montrer AKT1 Anticorps) substrate.
A statistically significant relationship was found between stem cell factor level and oocyte maturation, embryo quality, and clinical pregnancy.
PAR2 (Montrer F2RL1 Anticorps) plays a direct role in melanogenesis by increasing SCF secretion from keratinocytes.
We did not find any KITLG mutation in two affected individuals with familial progressive hyperpigmentation and hypopigmentation.
results suggest that SCF can accelerate cell homing and the maturation of the pulp-dentin complex in human immature teeth.
SCF is a critical regulatory factor for conceptus development and implantation during pregnancy in pigs.
the critical physiological role of the KIT-ET3 (Montrer EDN3 Anticorps)-NO pathway in fulfilling high demand (exceeding basal level) of endothelium-dependent NO generation for coping with atherosclerosis, pregnancy, and aging, is reported.
results revealed that the Th17-mediated inflammatory environment promotes mast cell accumulation through keratinocyte-derived SCF
Bone marrow adipocytes synthesize SCF, promoting hematopoietic stem cell proliferation/regeneration.
Hypoxia microenvironment in hepatocellular carcinoma up-regulates HIF2 (Montrer EPAS1 Anticorps)/SCF expression, which in turn promotes angiogenesis and HCC (Montrer FAM126A Anticorps) metastasis.
The results of this study suggest that NAR (Montrer CPSF4 Anticorps) relieves Lop (Montrer MIP Anticorps)-induced constipation by increasing the levels of interstitial cells of Cajal markers (c-Kit and SCF), as well as AQP3 (Montrer AQP3 Anticorps). Thus, NAR (Montrer CPSF4 Anticorps) may be effective as a candidate in patients suffering from lifestyle-induced constipation.
persistent distention/stretch on colonic smooth muscle cells could suppress SCF production probably through Ca(2 (Montrer CA2 Anticorps)+) -ERK (Montrer EPHB2 Anticorps)-AP-1 (Montrer JUN Anticorps)-miR (Montrer MLXIP Anticorps)-34c deregulation.
Activation of c-kit signalling by SCF promotes migration of cardiac stem cells with increased phosphorylation of CXCR4 (Montrer CXCR4 Anticorps)-serine 339, p38 mitogen-activated protein kinase (p38 MAPK (Montrer MAPK14 Anticorps)) and extracellular regulated protein kinases 1/2 (ERK1/2).
These findings support the view that SCF, in addition to its pro-proliferative effects, is important for the differentiation of MCs (Montrer SMCP Anticorps).
In vitro, SCF induced the phosphorylation of p38 MAPK (Montrer MAPK14 Anticorps) and cofilin (Montrer CFL1 Anticorps), leading to the migration of cardiac stem cells.
MAPK3 (Montrer MAPK3 Anticorps)/1 participates in primordial follicle activation through mTORC1-KITL signaling.
MicroRNA-205 maintains T cell development following stress by regulating Foxn1 (Montrer FOXN2 Anticorps) and its two regulated targets, stem cell factor and ccl25 (Montrer CCL25 Anticorps), following stress.
These findings indicate the SCF/Kit signaling insufficiency may contribute to the underdevelopment of ICCs and intestinal motility dysfunction upon hypoxia exposure.
SCF/c-kit signaling may potentiate chronic hypoxia-induced vascular remodeling by modulating ERK activation. Inhibition of c-kit activity may be a potential strategy to alleviate PH
Oocyte-derived signals interact locally to mediate granulosa and theca cell function. SCF has a role in modulating this local interaction.
little evidence was found for the KITLG gene being linked to variation in colour
FGF7 (Montrer FGF7 Anticorps) may be an important regulator for oocyte growth and its action is mediated via the KIT/KITLG signaling pathway.
chi-miR (Montrer MYLIP Anticorps)-204-5p and chi-miR (Montrer MYLIP Anticorps)-211 could change the expression levels of the KITLG gene and reduce granulosa cell proliferation.
Results suggest that the KITLG gene is a strong candidate gene affecting litter size in goats.
This gene encodes the ligand of the tyrosine-kinase receptor encoded by the KIT locus. This ligand is a pleiotropic factor that acts in utero in germ cell and neural cell development, and hematopoiesis, all believed to reflect a role in cell migration. In adults, it functions pleiotropically, while mostly noted for its continued requirement in hematopoiesis. Two transcript variants encoding different isoforms have been found for this gene.
, c-Kit ligand
, familial progressive hyperpigmentation 2
, kit ligand
, mast cell growth factor
, steel factor
, stem cell factor
, C-kit ligand
, Steel factor
, cloud gray
, hematopoietic growth factor KL
, steel factor/kit ligand
, stem cell factor KL-1
, KIT ligand precursor form 1
, KIT ligand precursor form 4
, c-kit ligand
, stem cell factor, CSF
, mast cell growth factor (white heifer disease)
, Mast cell growth factor
, stem cell factor homolog