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anti-Human VEGFC Anticorps:
anti-Mouse (Murine) VEGFC Anticorps:
anti-Rat (Rattus) VEGFC Anticorps:
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Human Polyclonal VEGFC Primary Antibody pour IHC (fro), IHC (p) - ABIN258871
Zampell, Yan, Avraham, Daluvoy, Weitman, Mehrara: HIF-1α coordinates lymphangiogenesis during wound healing and in response to inflammation. dans FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2012
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Human Polyclonal VEGFC Primary Antibody pour IF (p), IHC (p) - ABIN731723
Zhuo, Jia, Song, Lu, Ding, Wang, Song, Fu, Luo: The CXCL12-CXCR4 chemokine pathway: a novel axis regulates lymphangiogenesis. dans Clinical cancer research : an official journal of the American Association for Cancer Research 2012
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Human Polyclonal VEGFC Primary Antibody pour IP, ELISA - ABIN541859
Krishnan, Kirkin, Steffen, Hegen, Weih, Tomarev, Wilting, Sleeman: Differential in vivo and in vitro expression of vascular endothelial growth factor (VEGF)-C and VEGF-D in tumors and its relationship to lymphatic metastasis in immunocompetent rats. dans Cancer research 2003
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Human Monoclonal VEGFC Primary Antibody pour ICC, IHC (fro) - ABIN438679
Moussai, Mitsui, Pettersen, Pierson, Shah, Suárez-Fariñas, Cardinale, Bluth, Krueger, Carucci: The human cutaneous squamous cell carcinoma microenvironment is characterized by increased lymphatic density and enhanced expression of macrophage-derived VEGF-C. dans The Journal of investigative dermatology 2010
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Human Polyclonal VEGFC Primary Antibody pour FACS, IHC (p) - ABIN650694
Yan, Zhu, Yu, Ji, Zhang, Ji, Yan, Chen, Liu, Yin, Lin: Expression of vascular endothelial growth factor C and chemokine receptor CCR7 in gastric carcinoma and their values in predicting lymph node metastasis. dans World journal of gastroenterology : WJG 2004
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Rat (Rattus) Polyclonal VEGFC Primary Antibody pour IP, ELISA - ABIN1589911
Maertens, Erpicum, Detry, Blacher, Lenoir, Carnet, Péqueux, Cataldo, Lecomte, Paupert, Noel: Bone marrow-derived mesenchymal stem cells drive lymphangiogenesis. dans PLoS ONE 2014
These findings thus underscore a role for posterior cardinal (Montrer CARD8 Anticorps) vein and VegfC in patterning the head kidney prior to organ assembly and function.
Vegfc acts through ERK (Montrer MAPK1 Anticorps) to induce sprouting and differentiation of trunk lymphatic progenitors.
data not only reveal a non-canonical function of Mt2 (Montrer MT2 Anticorps) in angiogenesis, but also propose Mt2 (Montrer MT2 Anticorps) as a novel regulator of vegfc expression.
Vegfc signaling increases mafba (Montrer MAFB Anticorps) expression to control downstream transcription
Vegfc is dispensable for facial lymphatic sprouting but not for the complete development of the facial lymphatic network.
In the embryo, phenotypes driven by increased Vegfc are suppressed in the absence of Ccbe1 (Montrer CCBE1 Anticorps), and Vegfc-driven sprouting is enhanced by local Ccbe1 (Montrer CCBE1 Anticorps) overexpression. Moreover, Vegfc- and Vegfr3 (Montrer FLT4 Anticorps)-dependent Erk (Montrer MAPK1 Anticorps) signaling is impaired in the absence of Ccbe1 (Montrer CCBE1 Anticorps).
Vegfc has an essential role in lymphangiogenesis [review]
The parallel growth of motoneuron axons with the dorsal aorta depends on Vegfc/Vegfr3 (Montrer FLT4 Anticorps) signaling in zebrafish.
Vegfc acts in two distinct modes during development: as a paracrine factor secreted from arteries to guide closely associated lymphatic vasculature and as an autocrine factor to drive migratory persistence during angiogenesis.
Rspo1-Wnt-VegfC-Vegfr3 signaling plays a crucial role as an endothelial-autonomous permissive cue for developmental angiogenesis.
VEGF-C and VEGF (Montrer VEGFA Anticorps)-C156S genes have roles in the pro-lymphangiogenic growth factor therapy of lymphedema
Transcription of the vascular endothelial growth factor C gene (VEGF-C) and translation of the corresponding protein were significantly up-regulated in swine umbilical vein endothelial cells with classical swine fever virus acute infection.
No difference in bioactivity was detected between porcine relaxin-1 (Montrer RLN1 Anticorps) and recombinant human relaxin-2 (Montrer RLN1 Anticorps) in either mice or rats.
During progressive ischemia, functional and metabolic benefits of intramyocardial VEGF-C gene transfer were apparent. VEGF-C-induced collateral formation occurred at the site of gene transfer
SPARC (Montrer SPARC Anticorps) expression was inversely associated with the degree of malignancy and it had a negative correlation with VEGF-C and VEGF-D (Montrer Figf Anticorps) expression. Results suggest SPARC (Montrer SPARC Anticorps) might function as a tumor suppressor inhibiting angiogenesis and lymphangiogenesis in ovarian cancer by reducing the expression of VEGF-C and VEGF-D (Montrer Figf Anticorps).
VEGF-A/VEGF (Montrer VEGFA Anticorps)-C analysis showed higher positivity in metastatic nodes and higher positivity in the surrounding negative nodes from positive cases in comparison with nonmetastatic patients.
this study shows that decidual NK cells facilitate the interaction between trophoblastic and endothelial cells via VEGF-C and HGF (Montrer HGF Anticorps)
Lymphangiogenesis during tubulointerstitial fib (Montrer CTGF Anticorps)rosis to be associated with increased expression of CTGF and VEGF-C in human obstructed nephropathy as well as in diabetic kidney disease. vitro, CTGF induced VEGF-C production in HK-2 cells, while CTGF siRNA suppressed transforming growth factor beta1-induced VEGF-C upregulation.
Smad4 (Montrer SMAD4 Anticorps) expression negatively correlated with VEGF-A (Montrer VEGFA Anticorps) and VEGF-C in colon cancer
study is the first to describe the mechanism of leptin (Montrer LEP Anticorps)-promoted lymphangiogenesis by upregulating VEGF-C expression in chondrosarcomas.
Retroperitoneal tumour progression in EOC patients is associated with high VEGF-C expression.
Mechanistic investigations indicated that BDNF (Montrer BDNF Anticorps) facilitated VEGF-C-dependent lymphangiogenesis through the MEK (Montrer MAP2K1 Anticorps)/ERK (Montrer EPHB2 Anticorps)/mTOR (Montrer FRAP1 Anticorps) signaling pathway.
no difference in the levels of VEGF-A (Montrer VEGFA Anticorps), VEGF-C, and VEGF-D (Montrer Figf Anticorps) in pre-eclampsia compared to normotensive pregnant women stratified by HIV status
Results has shown that VEGF-C was highly expressed in non-small cell lung cancer (NSCLC) tissues and metastatic lymph nodes. VEGF-C expression levels was significantly correlated with lymph node metastasis in NSCLC. Along with CXCR4 (Montrer CXCR4 Anticorps), VEGF-C might synergically promote lymphatic metastasis in lung cancer and might be a clinical predictor of lymph node metastasis in NSCLC patients.
As shown in mouse model of kidney fibrosis CTGF (Montrer CTGF Anticorps) is significantly involved in fibrosis-associated renal lymphangiogenesis through regulation of, and direct interaction with, VEGF-C.
Fluid shear stress regulates vascular remodeling via VEGFR-3 (Montrer FLT4 Anticorps) activation, independently of its ligand, VEGF-C, in the uterus during pregnancy.
A novel heparin conjugate (LHbisD4) is shown to prevent lymphangiogenesis by blocking the vascular endothelial growth factor C (VEGF-C) induced signaling pathway.
lymphangiogenesis is regulated by two distinct proteolytic mechanisms of ligand activation: one in which VEGFC activation by ADAMTS3 (Montrer Adamts2 Anticorps) and CCBE1 (Montrer CCBE1 Anticorps) spatially and temporally patterns developing lymphatics, and one in which VEGFD (Montrer Figf Anticorps) activation by a distinct proteolytic mechanism may be stimulated during inflammatory lymphatic growth
These results reveal an unexpected role for VEGF-C, a major lymphangiogenic growth factor, in the transition to fetal liver erythropoiesis.
Results suggest that interleukin-6 (IL-6 (Montrer IL6 Anticorps)) increases VEGF-C induction and lymphangiogenesis may involve, at least in part, Src (Montrer SRC Anticorps)-FAK (Montrer PTK2 Anticorps)-STAT3 (Montrer STAT3 Anticorps) cascade in lymphatic endothelial cells (LECs).
Data show that heparanase-1 (HPA-1 (Montrer HPSE Anticorps)) induced shedding of heparan sulfate chain from syndecan-1 (SDC-1 (Montrer SDC1 Anticorps)) facilitated the release of vascular endothelial growth factor C (VEGF-C) from SDC-1 (Montrer SDC1 Anticorps)/VEGF-C complex into the medium of hepatocarcinoma cell.
Data show that in the MCF-7 breast cancer cell line, only MT1X (Montrer MT1X Anticorps) metallothioneins (MTs (Montrer NEU2 Anticorps)) positively correlated with vascular endothelial growth factor C (VEGFC).
The findings in this study strongly suggest the following: i) that VEGF-C promotes the proliferative activity and migratory ability of mesenchymal stem cell ; and ii) VEGF-C and Tgfb (Montrer TGFB1 Anticorps) reciprocally regulate mesenchymal stem cell commitment to differentiation into lymphatic endothelial or osteoblastic phenotypes, respectively.
The authors show that VEGF-C is necessary for perinatal lymphangiogenesis, but required for adult lymphatic vessel maintenance only in the intestine.
The protein encoded by this gene is a member of the platelet-derived growth factor/vascular endothelial growth factor (PDGF/VEGF) family, is active in angiogenesis and endothelial cell growth, and can also affect the permeability of blood vessels. This secreted protein undergoes a complex proteolytic maturation, generating multiple processed forms which bind and activate VEGFR-3 receptors. Only the fully processed form can bind and activate VEGFR-2 receptors. This protein is structurally and functionally similar to vascular endothelial growth factor D.
vascular endothelial growth factor C
, vascular endothelial growth factor c
, FLT4 ligand DHM
, vascular endothelial growth factor-related protein
, flt4 ligand
, vascular endothelial growth factor C isoform 129
, vascular endothelial growth factor C isoform 184