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anti-Human TLR3 Anticorps:
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Dog (Canine) Monoclonal TLR3 Primary Antibody pour FACS, ICC - ABIN4360082
Wen, Peng, Li, Wong: The effect of innate immunity on autoimmune diabetes and the expression of Toll-like receptors on pancreatic islets. dans Journal of immunology (Baltimore, Md. : 1950) 2004
Show all 65 Pubmed References
Dog (Canine) Monoclonal TLR3 Primary Antibody pour FACS - ABIN4360083
Funami, Matsumoto, Oshiumi, Akazawa, Yamamoto, Seya: The cytoplasmic 'linker region' in Toll-like receptor 3 controls receptor localization and signaling. dans International immunology 2004
Show all 25 Pubmed References
Dog (Canine) Monoclonal TLR3 Primary Antibody pour FACS, ICC - ABIN4360084
Evangelista, Castro, Alves, Dias, Souza, Reis, Silva, Castañon, Farias, Juliano, Ferreira: Early IFN-γ production together with decreased expression of TLR3 and TLR9 characterizes EAE development conditional on the presence of myelin. dans Autoimmunity 2016
Show all 24 Pubmed References
Dog (Canine) Monoclonal TLR3 Primary Antibody pour ELISA - ABIN4248101
Ranjith-Kumar, Miller, Xiong, Russell, Lamb, Santos, Duffy, Cleveland, Park, Bhardwaj, Wu, Russell, Sarisky, Mbow, Kao: Biochemical and functional analyses of the human Toll-like receptor 3 ectodomain. dans The Journal of biological chemistry 2007
Show all 21 Pubmed References
Human Polyclonal TLR3 Primary Antibody pour FACS, IHC (fro) - ABIN252527
Patole, Gröne, Segerer, Ciubar, Belemezova, Henger, Kretzler, Schlöndorff, Anders: Viral double-stranded RNA aggravates lupus nephritis through Toll-like receptor 3 on glomerular mesangial cells and antigen-presenting cells. dans Journal of the American Society of Nephrology : JASN 2005
Show all 14 Pubmed References
Human Polyclonal TLR3 Primary Antibody pour ICC, IF - ABIN4360085
Hsieh, Chang, Chen, Li, Chuang, Yu, Cheung, Chen, Maa, Leu: The inducible nitric-oxide synthase (iNOS)/Src axis mediates Toll-like receptor 3 tyrosine 759 phosphorylation and enhances its signal transduction, leading to interferon-β synthesis in macrophages. dans The Journal of biological chemistry 2014
Show all 6 Pubmed References
Human Monoclonal TLR3 Primary Antibody pour FACS, IF - ABIN2191973
Matsumoto, Kikkawa, Kohase, Miyake, Seya: Establishment of a monoclonal antibody against human Toll-like receptor 3 that blocks double-stranded RNA-mediated signaling. dans Biochemical and biophysical research communications 2002
Show all 6 Pubmed References
Human Monoclonal TLR3 Primary Antibody pour FACS, IF - ABIN2191974
Oshiumi, Matsumoto, Funami, Akazawa, Seya: TICAM-1, an adaptor molecule that participates in Toll-like receptor 3-mediated interferon-beta induction. dans Nature immunology 2003
Show all 6 Pubmed References
Human Monoclonal TLR3 Primary Antibody pour FACS, IF - ABIN2191972
Matsumoto, Funami, Tanabe, Oshiumi, Shingai, Seto, Yamamoto, Seya: Subcellular localization of Toll-like receptor 3 in human dendritic cells. dans Journal of immunology (Baltimore, Md. : 1950) 2003
Show all 6 Pubmed References
Bird (Avian) Polyclonal TLR3 Primary Antibody pour FACS, IHC (p) - ABIN4360080
Kuzemtseva, de la Torre, Martín, Soldevila, Ait-Ali, Mateu, Darwich: Regulation of toll-like receptors 3, 7 and 9 in porcine alveolar macrophages by different genotype 1 strains of porcine reproductive and respiratory syndrome virus. dans Veterinary immunology and immunopathology 2014
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Using hepatocyte-based culture models for Hepatitis C virus (HCV) infection, we found a portion of HCV dsRNA intermediates to be released from infected cells in extracellular vesicles, which reduces activation of toll-like receptor 3.
the association between TLR3, TLR4 (Montrer TLR4 Anticorps) variants and nine IL-6 (Montrer IL6 Anticorps) polymorphisms, and response to anti-viral treatment during hepatitis C infection.
results suggest that patients with Toll-like receptor 3 gene polymorphisms could be susceptible to periapical pathosis.
Studied associations of 12 single nucleotide polymorphisms (SNPs) within toll (Montrer TLR4 Anticorps) like receptor genes (TLR2, TLR3, TLR4 (Montrer TLR4 Anticorps), and TLR9 (Montrer TLR9 Anticorps)) and genetic predisposition to neonatal severe hepatitis. Found certain SNPS were associated with prognosis of neonatal severe hepatitis.
The current study suggests that TLR3 signaling induces CCL5 (Montrer CCL5 Anticorps) expression via NF-kappaB (Montrer NFKB1 Anticorps) and IRF3 (Montrer IRF3 Anticorps) in bile duct cells, and this pathway may be involved in the pathogenesis of BA.
The down regulation of TRIF (Montrer TRIM69 Anticorps), TLR3, and mitochondrial antiviral signaling protein (MAVS (Montrer MAVS Anticorps)) expressions in chronic hepatitis C correlates with the disease severity and the outcome of hepatitis C virus infection
this study signifies that TLR3 adaptor molecules are necessary for the proper production of cytokines, chemokines and pro-labour mediators after TLR ligation
we found that ORF3 (Montrer ASZ1 Anticorps) protein downregulates TLR3-mediated NF-kappaB (Montrer NFKB1 Anticorps) signaling via TRADD (Montrer TRADD Anticorps) and RIP1 (Montrer UQCRFS1 Anticorps). Our findings provide a new perspective on the cellular response in HEV infection and expand our understanding of the molecular mechanisms of Hepatitis E virus (HEV) pathogenesis in innate immunity.
Increased expressions of TLR-3, TLR-4 (Montrer TLR4 Anticorps) and CD80 (Montrer CD80 Anticorps) mRNA and the level of urinary CD80 (Montrer CD80 Anticorps)/creatinine could be useful markers to differentiate patients of steroid-sensitive nephrotic syndrome in relapse from those with steroid-resistant nephrotic syndrome.
TRIM56 (Montrer TRIM56 Anticorps) may act as a tumor suppressor in multiple myeloma through activation of TLR3/TRIF (Montrer TRIM69 Anticorps) signaling pathway.
these data suggest that TLR3 promotes the clearance of Cm during early and mid-stages of genital tract infection, and that loss of TLR3 is detrimental in the development hydrosalpinx.
TLR3 deficiency in Tlr3 KO mice suppressed the development of chronic contact hypersensitivity reactions, suggesting that TLR3 signaling may participate in the pathogenesis of atopic dermatitis.
The TLR3/TICAM-1 (Montrer TICAM1 Anticorps) pathway inhibits polyposis through suppression of c-Myc (Montrer MYC Anticorps) expression and supports long survival in Apc (Montrer APC Anticorps) (Min/+) mice.
PolyI:C targeted Toll-like receptor 3 with minimal effect on the mitochondrial antiviral-signaling protein (Montrer MAVS Anticorps) pathway.
determined whether the depletion of TLR3 modulated hepatic injury in mice and further aimed to provide mechanistic insights into the TLR3-mediated modulation of diet-induced hepatic inflammation and fat accumulation.
TLR3 signaling contributes to Wallerian degeneration after peripheral nerve injury by affecting Schwann cell activation.
This study establishes a correlation between TLR-3 and TLR-9 (Montrer TLR9 Anticorps) expression with the development of EAE. In addition, evidence of a role for the myelin peptide in targeting the innate inflammatory response to the CNS is presented.
Data show that HCFC2 (Montrer HCFC2 Anticorps) is a critical component of the IRF1 (Montrer IRF1 Anticorps) and IRF2 (Montrer IRF2 Anticorps) transcriptional machinery that regulates Tlr3 gene expression.
the JAK (Montrer JAK3 Anticorps)-STAT (Montrer STAT1 Anticorps) pathway provides a cytokine rheostat mechanism, which enables macrophages to fine-tune their responses to multiple, temporally separated infection events involving the TLR3 and TLR7 (Montrer TLR7 Anticorps) pathways.
These results suggest that testicular innate immune responses to pathogens caused by nano-TiO2 may be involved in the regulatory mechanisms of TAM (Montrer CCNA1 Anticorps)/TLR3 signaling in testicular Sertoli cells.
RIG-I (Montrer DDX58 Anticorps) and TLR3 interact with the pseudoknot of Porcine Reproductive and Respiratory Syndrome Virus 3' untranslated regions. Both RIG-I (Montrer DDX58 Anticorps) and TLR3 are required for the pseudoknot to induce interferon (Montrer IFNA Anticorps) response.
These data demonstrated that TLR2, TLR3 and TLR9 (Montrer TLR9 Anticorps) contribute to NF-kappaB (Montrer NFKB1 Anticorps) activation in response to porcine epidemic diarrhea virus infection, but not RIG-I (Montrer DDX58 Anticorps).
TLR3 is regulated differentially by different genotype 1 PRRSV strains and this seems to be related apparently to the replication levels of each strain, as well as, to the TNF-alpha (Montrer TNF Anticorps) inducing capability.
5 known non-synonymous single nucleotide polymorphisms (SNPs) were characterized in the coding sequences of the porcine TLR3 gene.
Activation of porcine TLR3 signaling is important in stimulating effective responses to PRRSV infection.
The results from this study demonstrate that expression of at least TLR3, TLR7 (Montrer TLR7 Anticorps) and TLR8 (Montrer TLR8 Anticorps) is stimulated upon bovine alpha-herpesvirus infection of the brain.
TLR2, 3, 4, and 8 mRNA expression is strongly upregulated and correlates with the progression of atherosclerosis in the aorta. Fluvastatin significantly inhibited this progress and reduced inflammation via TLR downregulation.
18 SNPs of TLR3 were observed and only 4 polymorphic positions were detected in the domestic breeds and 14 non-synonymous substitutions were observed, most of them in the LRR molecules.
Differential gene expression following TLR stimulation in rag1 (Montrer RAG1 Anticorps)-/- mutant zebrafish tissues and morphological descriptions of lymphocyte-like cell populations
Binding energy (BE) calculation using MM/PBSA method from the TLR3- and TLR22-ligand complexes revealed an adequate binding affinity between TLR22-monomer and dsRNA as like as TLR3-dimer-dsRNA complex.
Full-length tlr3 was functionally characterized.
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns (PAMPs) that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This receptor is most abundantly expressed in placenta and pancreas, and is restricted to the dendritic subpopulation of the leukocytes. It recognizes dsRNA associated with viral infection, and induces the activation of NF-kappaB and the production of type I interferons. It may thus play a role in host defense against viruses. Use of alternative polyadenylation sites to generate different length transcripts has been noted for this gene.
toll-like receptor 3
, toll-like receptor 3-like
, toll-like receptor 3 variant 1