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anti-Human AGT Anticorps:
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Human Polyclonal AGT Primary Antibody pour ICC, IF - ABIN447446
Dimitrijevic, Rissler, Luts, Edvinsson: Reduced expression of angiotensin II and angiotensin receptor type 1 and type 2 in resistance arteries from nasal lesions in granulomatosis with polyangiitis (Wegener's granulomatosis). dans Scandinavian journal of rheumatology 2011
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Human Monoclonal AGT Primary Antibody pour WB - ABIN1882204
Desong Liu, Fang Lu, Songhui Zhai, Liu Wei, Shi Ma, Xiuying Chen, Liqun Dong, Yannan Guo, Jin Wu, Zheng Wang: Renin-angiotensin system gene polymorphisms in children with Henoch-Schönlein purpura in West China. dans Journal of the renin-angiotensin-aldosterone system : JRAAS 2010
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Human Monoclonal AGT Primary Antibody pour WB - ABIN1882205
Kieć-Wilk, Olszanecka, Mikołajczyk, Kawecka-Jaszcz et al.: [Role of the M235T (c.704c>T) polymorphism of angiotensynogen gene as well as A724A (c.2171G>A) polymorphism of SERCA2a gene in ethiopathogenesis of left ventricular hypertrophy in essential... dans Przegla̧d lekarski 2010
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Human Monoclonal AGT Primary Antibody pour ELISA, WB - ABIN968948
Xu, Carretero, Lin, Cavasin, Shesely, Yang, Reudelhuber, Yang: Role of cardiac overexpression of ANG II in the regulation of cardiac function and remodeling postmyocardial infarction. dans American journal of physiology. Heart and circulatory physiology 2007
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Human Monoclonal AGT Primary Antibody pour ELISA, WB - ABIN559812
Jain, Li, Patil, Kumar: HNF-1alpha plays an important role in IL-6-induced expression of the human angiotensinogen gene. dans American journal of physiology. Cell physiology 2007
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Human Polyclonal AGT Primary Antibody pour IHC, IHC (p) - ABIN4352882
Bachmann, Burté, Pramana, Conte, Brown, Orimadegun, Ajetunmobi, Afolabi, Akinkunmi, Omokhodion, Akinbami, Shokunbi, Kampf, Pawitan, Uhlén, Sodeinde, Schwenk, Wahlgren, Fernandez-Reyes, Nilsson: Affinity proteomics reveals elevated muscle proteins in plasma of children with cerebral malaria. dans PLoS pathogens 2014
The reduced urinary AGT (Montrer AGXT Anticorps)/creatinine in Autralian Indigenous pregnant women may reflect subclinical renal dysfunction which limits the ability of the kidney to maintain sodium balance and could indicate an increased risk of pregnancy complications and/or future renal disease.
ACE2 (Montrer ACE2 Anticorps) and other enzymes can form ANG-(1 (Montrer ANGPT1 Anticorps)-7) directly or indirectly from either the decapeptide ANG I or from ANG II. [review]
The ACE (Montrer ACE Anticorps) and AGT (Montrer AGXT Anticorps) gene polymorphisms are not associated with the progress of diabetes developing into retinopathy in Chinese patients with type 2 diabetes.
AGT (Montrer AGXT Anticorps) M235T and T174M variants contribute to an increased risk of developing preeclampsia (PE), and for M235T to PE severity.
Data, including data using network analysis, suggest that angiotensinogen (AGT), mitogen-activated protein kinase-14 (MAPK14 (Montrer MAPK14 Anticorps)), and prothrombin (Montrer F2 Anticorps) (F2) in placental villous tissues are core factors in early embryonic development; these studies involved proteomics and bioinformatics analysis of altered protein expression in placental villous tissue from early recurrent miscarriage patients in comparison to control tissues.
The AGT (Montrer AGXT Anticorps) (M235T) gene polymorphism do not seem to have a significant effect on the development of clinical properties or cardiovascular comordities of acromegalic patients.
Angiotensin II has a role in increasing glomerular permeability by beta-arrestin mediated nephrin (Montrer NPHS1 Anticorps) endocytosis
After donor nephrectomy, increasing uAGT (Montrer DPAGT1 Anticorps) levels can be the result of activation of the intrarenal renin (Montrer REN Anticorps)-angiotensin system affecting the compensatory changes in the remaining kidney.
M235T polymorphism of the AGT (Montrer AGXT Anticorps) gene seems unrelated to the development or the clinical course of endometriosis.
AGT (Montrer AGXT Anticorps) missense polymorphisms are not associated with diabetic nephropathy in our subset of Slovenian type 2 diabetes mellitus patients
ANG II is up-regulated in serum and heart tissues of mice with EAM and that ANG II significantly drives monocyte/macrophage infiltration through the C-C chemokine receptor 2/5 (CCR2/5) axis.
results established that A20 (Montrer TNFAIP3 Anticorps) is involved in the renoprotective effect by calcitriol via negatively modulating the NF-kappaB (Montrer NFKB1 Anticorps) pathway and necroptotic pathway in AngII-induced renal injury.
NLRP3 (Montrer NLRP3 Anticorps) gene deletion attenuates Ang II-induced NLRP3 (Montrer NLRP3 Anticorps) inflammasome activation, phenotypic transformation from a contractile phenotype to a synthetic phenotype and proliferation in primary mice Vascular Smooth Muscle Cells.
adipocyte-derived Agt (Montrer AGXT Anticorps) has essentially no contribution to the plasma concentration and no impact on blood pressure compared to liver-derived Agt (Montrer AGXT Anticorps).
Lung ischemia-reperfusion injury causes a dysregulation of circulating Ang 2 (Montrer ANGPT2 Anticorps) levels and plasma PREP (Montrer PREP Anticorps) activity, although no direct link between both phenomena could be shown.
Inhibition of TLR4 (Montrer TLR4 Anticorps) ameliorates AngII-impaired cavernosal relaxation, decreases TNF-alpha (Montrer TNF Anticorps) levels, and restores Nitric Oxide bioavailability, demonstrating that TLR4 (Montrer TLR4 Anticorps) partly mediates AngII-induced cavernosal dysfunction.
Our study is the first to show the important role of IL-6 (Montrer IL6 Anticorps) in regulating cardiac pathogenesis via inflammation and apoptosis during AngII-induced hypertension. We also provide a novel link between IL-6 (Montrer IL6 Anticorps)/STAT3 (Montrer STAT3 Anticorps) and EndoG (Montrer ENDOG Anticorps)/MEF2A (Montrer MEF2A Anticorps) pathway that affects cardiac hypertrophy during AngII stimulation.
this study demonstrated that Ang II could increase TRPC6 (Montrer TRPC6 Anticorps) induced Ca(2 (Montrer CA2 Anticorps)+) influx and enhance autophagy through increasing reactive oxygen species levels in podocytes, and autophagy could protect Ang II-treated podocytes.
These results implied that AngII could effectively induce EpiCs to differentiate into vascular smooth muscle-like cells through the AT1 receptor (Montrer AGTRAP Anticorps).
Results suggest the involvement of angiotensin II (Ang II), through its angiotensin type-1 receptor (AT1R (Montrer AGTRAP Anticorps)) in the inflammation induced by Aah (Montrer ASPH Anticorps) venom, in the heart and the aorta.
In conclusion, endothelial vWF (Montrer VWF Anticorps) knockdown prevented angiotensin II-induced ET-1 (Montrer EDN1 Anticorps) upregulation through attenuation of NOX-mediated O2- production.
Data suggest that intra-adrenal metabolism of Ang II to Ang III is required for zona glomerulosa cell-mediated relaxation of adrenal arterioles but not for aldosterone secretion.
NADPH oxidase (Montrer NOX1 Anticorps) plays an important role in proMMP-2 expression and activation and MMP-2 (Montrer MMP2 Anticorps) mediated SMC (Montrer DYM Anticorps) proliferation occurs through the involvement of Spm (Montrer NPC1 Anticorps)-Cer (Montrer CBLN1 Anticorps)-S1P (Montrer MBTPS1 Anticorps) signaling axis under ANG II stimulation of PASMCs
The metabolism of angiotensin II (Ang II) to angiotensin III (Ang III) and its role in the vasorelaxation response in adrenal arteries are reported.
The study identified the serine phosphorylation (p-Ser (Montrer SIGLEC1 Anticorps)) sites induced by PKC-Beta (Montrer PRKCB Anticorps) activation or AGT, which inhibits insulin (Montrer INS Anticorps)-induced p-Tyr (Montrer TYR Anticorps) sites on IRS2 (Montrer IRS2 Anticorps) and its signals in endothelial cells.
Data suggest up-regulation of AGT in granulosa cells and of Ang II in follicular fluid during preovulatory period; Ang II appears to amplify stimulatory effects of luteinizing hormone on secretion of progesterone/prostaglandins by granulosa cells.
Data suggest that angiotensin II promotes uptake/accumulation of iron (non-transferrin (Montrer Tf Anticorps) bound iron) into vascular endothelial cells; such iron accumulation appears to depend on activation of angiotensin type 1 receptor and promotes oxidative stress.
a critical role for H(2)O(2) in angiotensin-II signaling to the endothelial cytoskeleton in a novel pathway that is critically dependent on MARCKS, Rac1, and c-Abl.
The objective of this study was to characterize the profiles of Ang-(1-7), MAS receptor, ACE(2), NEP and PEP during the ovulatory process in cattle.
Fetal adrenal cells in primary culture respond to angiotensin-II by increasing aldosterone production and aldosterone synthase (Montrer CYP11B2 Anticorps) [P450c18/CYP11B2 (Montrer CYP11B2 Anticorps)] activity.
ANG II inhibits bTREK-1 K(+) channels by a Ca(2+)-dependent mechanism that does not require the depletion of membrane-associated PIP(2).
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8
, angiotensin ll
, angiotensinogen (PAT)
, zC8A9.1 (angiotensinogen )
, Serpin A8