Il n’y a pas de produits dans votre liste de comparaison.
Votre panier est vide.
Afficher toutes les espèces
Afficher tous les synonymes
Sélectionnez vos espèces et l'application
anti-Human Angiotensin II/III Anticorps:
anti-Rat (Rattus) Angiotensin II/III Anticorps:
anti-Dog (Canine) Angiotensin II/III Anticorps:
Vous arrivez à notre recherche pré-filtrée.
Human Monoclonal Angiotensin II/III Primary Antibody pour IHC (p), IHC - ABIN253542
Ni, Ma, Wang, Liu, Zhang, Lv, Ni, Chen, Ruan, Liu: Activation of renin-angiotensin system is involved in dyslipidemia-mediated renal injuries in apolipoprotein E knockout mice and HK-2 cells. dans Lipids in health and disease 2014
Show all 9 Pubmed References
In the Asian populations, ACE I/D and AGTR1 A1166C may contribute to Diabetic Nephropathy (DN) susceptibility in patients with T2DM by different genetic models. However, the role of AGT M235T needs to be further evaluated. [review & meta-analysis]
In coronary artery disease patients, M235T among several AGT gene polymorphisms is associated with elevated serum-CRP levels with AGT C allele as the significant factor for patients with serum-CRP level of more than 1 mg/L.
High urinary angiotensinogen levels are associated with albuminuria and left ventricular hypertrophy
Renin angiogensin system activation, particularly renal RAS activation indicated by elevated urinary Ang excretion, exerts a fundamental role in the pathogenesis of hypertension in the general population.
AGT C/T and AT2 G/A polymorphisms were not significantly associated with aneurysmal subarachnoid hemorrhage (aSAH) after controlling for potential confounders. However, a strong trend was identified for a dominant effect of the G allele of the AT2 G/A SNP. Downregulation of the local RAS may contribute to the formation of cerebral aneurysms and subsequent presentation with aSAH.
Patients homozygous for the C allele of the AGT C/T polymorphism experienced a higher rate of rupture from aneurysms smaller than 7 mm.
Among East Asians the angiotensinogen variant M235T is associated with cardiovascular disease.
The AGT Thr-Thr genotype frequency was significantly higher among WLs (25.9%) compared with S/J (4.2%) and controls (12.8%).
angiotensinogen-6 A/G and angiotensinogen-20 A/C polymorphisms were not associated with the antihypertensive response to telmisartan treatment in Chinese patients with hypertension.
the T174M polymorphism in the AGT gene was associated with diabetic nephropathy in Asians.
Contractile responses to angiotensin II are preserved in narrow lumen human cirrhotic splanchnic arteries and are comparatively augmented in early disease. Angiotensin-(1-7) had no vasodilatory effect on adrenergic tone, however, attenuated angiotensin II-induced contractility, possibly through an Ang-(1-7)-AT1R interaction, and thus may contribute to pathological vasodilatation in human cirrhosis.
Ang II regulates sympathetic and parasympathetic nerve-mediated excitation and contraction.
AGT gene polymorphism rs5050T>G is associated with the risk of coronary aneurysm in children with Kawasaki disease.
Angiotensinogen (AGT) rs5050 (GG) found to be associated with poor prognosis in astrocytoma patients
Isoliquiritigenin alleviated the Ang II-induced hypertensive renal injury through suppressing inflammation cytokines, excessive deposition of extracellular matrix and oxidative stress-induced apoptosis via Nrf2 and NF-kappaB pathways.
findings indicate that AGT adapts unique serpin features for hormone delivery and binds renin through concerted movements in the N-terminal tail and in its main body to modulate angiotensin release.
The rs7079 C to A substitution reduced the binding of miR-31-5p/miR-584-5p to the 3' UTR of AGT, possibly altering the risk of lead poisoning.
These results suggest that Angiotensin II induces CTGF expression and extracellular matrix accumulation through a special TGF-beta-independent interaction between the NF-kappaB and Smad2/3 signals elicited by the AT1/PKCalpha/p38 MAPK pathway.
The possible contribution of the I/D in the ACE gene, M235T and T174M in the angiotensinogen (AGT) gene polymorphisms with ischemic stroke in young Mexican population.
AGT M235T gene polymorphism may represent a genetic modifier to vascular morbidities in Egyptian patients with sickle cell disease.
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8