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Human Monoclonal IL5 Primary Antibody pour CyTOF, ELISA (Capture) - ABIN4900785
Xavier-Elsas, Santos-Maximiano, Queto, Mendonça-Sales, Joseph, Gaspar-Elsas, Vargaftig: Ectopic lung transplantation induces the accumulation of eosinophil progenitors in the recipients' lungs through an allergen- and interleukin-5-dependent mechanism. dans Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology 2007
Show all 13 Pubmed References
Mouse (Murine) Monoclonal IL5 Primary Antibody pour ICS, Neut - ABIN1176999
Abrams: Immunoenzymetric assay of mouse and human cytokines using NIP-labeled anti-cytokine antibodies. dans Current protocols in immunology / edited by John E. Coligan ... [et al.] 2008
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Human Monoclonal IL5 Primary Antibody pour Inhibition, ELISA - ABIN2689772
Abrams, Roncarolo, Yssel, Andersson, Gleich, Silver: Strategies of anti-cytokine monoclonal antibody development: immunoassay of IL-10 and IL-5 in clinical samples. dans Immunological reviews 1992
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Human Monoclonal IL5 Primary Antibody pour Inhibition, ELISA - ABIN2689771
Butterfield, Leiferman, Abrams, Silver, Bower, Gonchoroff, Gleich: Elevated serum levels of interleukin-5 in patients with the syndrome of episodic angioedema and eosinophilia. dans Blood 1992
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Human Monoclonal IL5 Primary Antibody pour CyTOF, FACS - ABIN4900611
Sen, Chunsong, Baojun, Linjie, Qun, San, Qiuping, Junyan, Zhang, Jinquan: Aberration of CCR7 CD8 memory T cells from patients with systemic lupus erythematosus: an inducer of T helper type 2 bias of CD4 T cells. dans Immunology 2004
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Human Monoclonal IL5 Primary Antibody pour ELISA - ABIN2689775
Denburg, Silver, Abrams: Interleukin-5 is a human basophilopoietin: induction of histamine content and basophilic differentiation of HL-60 cells and of peripheral blood basophil-eosinophil progenitors. dans Blood 1991
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Mouse (Murine) Monoclonal IL5 Primary Antibody pour ELISA - ABIN2689774
Sander, Andersson, Andersson: Assessment of cytokines by immunofluorescence and the paraformaldehyde-saponin procedure. dans Immunological reviews 1991
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Human Monoclonal IL5 Primary Antibody pour FACS - ABIN4898071
Alisa, Boswell, Pathan, Ayaru, Williams, Behboudi: Human CD4(+) T cells recognize an epitope within alpha-fetoprotein sequence and develop into TGF-beta-producing CD4(+) T cells. dans Journal of immunology (Baltimore, Md. : 1950) 2008
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Human Monoclonal IL5 Primary Antibody pour FACS - ABIN4898070
Blom, Poulsen, Jensen, Hansen, Poulsen: IL-33 induces IL-9 production in human CD4+ T cells and basophils. dans PLoS ONE 2011
The concentration of Th2-related cytokines (IL-5 and IL-13) in asthmatic children with Rhinovirus(+) was significantly higher than those with Rhinovirus(-).
IL-5 is expressed intrathecally in tick-borne encephalitis, but its pathogenetic role remains unclear.
eosinophilia in FE is secondary to dysregulation of IL-5 production in PBMC (and their component subsets).
Data indicate that interleukin-5 (IL-5) was that only serum cytokines show statistical significance between severe chronic obstructive pulmonary disease (COPD) and controls.
Serum IL-5 and IL-13 are reliable biomarkers for the blood eosinophilia asthma phenotype.
Longitudinal co-variations between inflammatory cytokines, lung function and patient reported outcomes in patients with asthma
Obesity alters the lung neutrophil infiltration to enhance breast cancer metastasis through IL5 and GM-CSF.
simvastatin was demonstrated to inhibit IL-5-induced CCR3 expression and chemotaxis of eosinophils mediated via the mevalonate pathway.
Interleukin-5/-targeted treatments offer promises to patients with eosinophilic respiratory disorders; review
Data indicate that interleukin 5 (IL-5)-associated single nucleotide polymorphisms (SNPs) were not associated with carotid intima-media thickness.
Post-liver transplant patients with food allergy showed a unique cytokine profile in response to various stimuli, with extremely elevated IL-5, low IL-10 secretion.
high levels of IL-33 and a high IL-33/soluble ST2 ratio correlates with elevated levels of IFN-gamma, TNF-alpha and IL-17alpha as well as IL-5, demonstrating that IL-33 has pleiotropic effects.
Production of IL-5 in response to both extract and the Bet v1-derived peptide mix strongly suggested that T cells were a major source of IL-5.
TRPV1 expression level, IL-4 level, and rs4790522 site mutation are the main risk factors inducing bronchial asthma in children
It has been shown in this study that the level of IL-5, one of the markers of eosinophilic inflammation, is higher in EBC of children with atopic asthma than in those with nonatopic asthma.
IL-5 may be part of protective mechanisms operating in early atherosclerosis, at least in women
Results showed that PAX2 expression is significantly overexpressed in esophageal squamous cell carcinoma tissues and IL-5 is identified as PAX2's effector for metastasis.
At a fixed concentration of 10-10 M, FF had significantly higher suppressive effects on interferon (IFN)-gamma, interleukin (IL)-2 and IL-17 release, but not IL-5 or tumor necrosis factor (TNF)-alpha, vs. MF.
There was an improvement of the in vitro-cytokine responses in iL-5, IL-10, and interferon-gamma after liver transplantations in end-stage liver disease pediatric patients.
Subsequent IL-5-stimulated signaling.
we have studied the gene expression changes associated with IL-5 up-regulation in eosinophils
binding of IL-5 to IL-5Ralpha receptors enhances angiogenic responses by stimulating the expression of HSP70-1 via the eNOS signaling pathway.
Both pre- and post-transcriptional processes may be involved in the AR modulation of ILC2 IL-5 and IL-13 production.
IL-33 acts directly on bone marrow ILC2s, making them an early source of IL-5 and part of a process that is central in IL-33-driven eosinophilia.
these studies establish a basal defect in eosinophilopoiesis in IL-33- and ST2-deficient mice and a mechanism whereby IL-33 supports eosinophils by driving both systemic IL-5 production and the expansion of IL-5Ralpha-expressing precursor cells
Increased production of IL-5 from Peyer's patch cells and the restored Th1-type immune response might cause the production of abnormal IgA and might induce the glomerular deposition of IgA in IGA nephropathy.
E. granulosus infection remarkably reduces the severity of OVA-induced airway inflammation likely through enhancing IL-10 and down-regulation of IL-5 and IL-17A.
selective proliferation of IgM rheumatoid factor-secreting B-1a cells is induced by co-stimulation by the specific pathogen antigen and IL-5 in the development of MC in Capillaria hepatica-infected mice
IL5, a cytokine involved in allergic and infectious diseases, facilitates metastatic colonization through recruitment of sentinel eosinophils and regulation of other inflammatory/immune cells in the microenvironment of the distal lung.
In mice, low-dose IL-2-anti-IL-2 antibody complexes drove group 2 innate lymphoid cells (ILC2) to produce IL-5 and proliferate.
Data (including data from knockout mice) suggest that up-regulation of IL5 production in lungs during influenza virus infection is due to infiltration of natural killer cells and alveolar macrophages into infected lung tissue.
A decrease in the levels of IL-5, IL-9, and IL-6R in the BALF.
eosinophils express CAR4 following IL-5 or allergen exposure, and that CAR4 is involved in regulating the lung transcriptome associated with allergic airway inflammation
Loa loa exhibits a differential survival and development in different strains of cytokine or cytokine receptor gene knockout mice with IL-4R and IL-5 playing critical roles in the host resistance to L. loa infection.
In mice 8 hours afterthe single administration of Immunovac-VP-4 the levels of IL-1beta, IL-6, IL- 12, IL-5 increased significantly. However their concentration differed depending on the method of administration.
Protection of montelukast on OVA-induced eosinophilic gastroenteritis via modulating IL-5, eotaxin-1 and MBP expression.
Together, these studies support the conclusion that surfactant protein D increases susceptibility to Cryptococcus neoformans infection by promoting Cryptococcus neoformans-driven pulmonary IL-5 and eosinophil infiltration.
IL5 induced eosinophils and cysteinyl leukotrienes are involved in the pathology of mite antigen-induced chronic asthma model.
OprF-I actively stimulated production of IL-2 that is a factor of growth and differentiation of lymphocytes, natural killers and cytotoxic lymphocytes; as well as IL-5, IL-O10, TNF-alpha and IFN-gamma.
This study showed that pronounced lung eosinophilia in horses can be transient, abate without specific treatment, and in this instance, lack correlation to upregulation of expression of either IL-4 or IL-5.
The protein encoded by this gene is a cytokine that acts as a growth and differentiation factor for both B cells and eosinophils. This cytokine is a main regulator of eosinopoiesis, eosinophil maturation and activation. The elevated production of this cytokine is reported to be related to asthma or hypereosinophilic syndromes. The receptor of this cytokine is a heterodimer, whose beta subunit is shared with the receptors for interleukine 3 (IL3) and colony stimulating factor 2 (CSF2/GM-CSF). This gene, together with those for interleukin 4 (IL4), interleukin 13 (IL13), and CSF2, form a cytokine gene cluster on chromosome 5. This cytokine, IL4, and IL13 are found to be regulated coordinately by long-range regulatory elements spread over 120 kilobases on chromosome 5q31.
B-cell differentiation factor I
, T-cell replacing factor
, eosinophil differentiation factor
, B-cell growth factor II
, cytotoxic T-lymphocyte inducer
, Interleukin 5 (colony-stimulating factor eosinophil)
, Interleukin 5 (colony-stimulating factor, eosinophil)
, colony-stimulating factor, eosinophil
, interleukin 5
, interleukin 5 (colony-stimulating factor, eosinophil)