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anti-Human Adipsin Anticorps:
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Human Monoclonal Adipsin Primary Antibody pour ELISA, WB - ABIN2192399
Oppermann, Baumgarten, Brandt, Gottsleben, Kurts, Götze: Quantitation of components of the alternative pathway of complement (APC) by enzyme-linked immunosorbent assays. dans Journal of immunological methods 1990
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Human Monoclonal Adipsin Primary Antibody pour ELISA - ABIN2192401
Oppermann, Kurts, Zierz, Quentin, Weber, Götze: Elevated plasma levels of the immunosuppressive complement fragment Ba in renal failure. dans Kidney international 1992
Show all 2 Pubmed References
Senescent dermal fibroblasts negatively influence fibroblast extracellular matrix-related gene expression (such as MMP1) partly via secretion of complement factor D.
The results suggested that elevated IL-17A and IL-9 expressions and decreased levels of adipsin and CCL11 were positively associated with adult asthma.
MASP-3 is the exclusive pro-factor D activator in resting blood: the lectin and the alternative complement pathways are fundamentally linked.
disturbance of reciprocal relationships between adipsin and leptin in obesity is associated with the development of insulin resistance
TZDs (pioglitazone, rosiglitazone, and ciglitazone) slow tumor cell growth in vitro and in vivo with decreases in cell proliferation and increases in PPARg and adipsin
The strong association of PLIN1, CFD and ADIPOQ genes with adipogenesis prompted authors to study the influence the bone health status as evaluated by quantitative ultrasound (QUS) bone densitometer in a North Indian cohort. Overall, ADIPOQ (rs1501299 and rs3774261) and combined cluster of PLIN1 rs2304796 and rs2304795) and CFD (rs1683563) demonstrated correlation.
our findings set up a novel mechanism for FABP4, adipsin and adiponectin through gut microbiota mediating expression in gut Paneth cells.
Overall adipsin levels among male asbestos industry workers were significantly higher than among control subjects.
Data indicate that a urinary protein, adipsin, was significantly increased in patients with preeclampsia.
Study demonstrates that T2DM patients with beta cell failure are deficient in adipsin.
Complement factor D (FD) is activated by its substrate through interactions outside the active site which lock the unbound native state into an ordered inactive conformation via the self-inhibitory loop in FD.
We demonstrate novel secretion of adipsin and ASP by placental Hofbauer cells.
Increased activation of the alternative complement pathway in vitreous was controlled by disease stage and genetic variation in the complement pathway, supporting a role for complement activation in macular degeneration disease pathogenesis.
Anti factor D Fab fragment inhibits FD proteolytic function by interfering with macromolecular substrate access rather than by inhibiting FD catalysis.
CFD regulates activation of the alternative complement pathway, which is implicated in age related macular degeneration pathogenesis.
CFD may not play a major role in the genetic susceptibility to age-related macular degeneration.
crystal structure of C3bB at 4 A and complex with factor D at 3.5 A; data show how factor B binding to C3b forms open "activation" state of C3bB; Factor D binds open conformation of factor B through a site distant from the catalytic center
As adiponectin and adipsin levels in CSF did not correlate with their levels in plasma, it seems that there could be a secondary intrathecal synthesis of these adipocytokines in multiple sclerosis
increased serum levels in patients with seasonal allergic rhinitis
An essential role for Factor D in the ability of the liver to recover from acute toxic injury, is reported.
The present study identifies adipsin, one of the most abundant and specifically expressed adipose proteins, as a circulating factor linking fat cells and obesity to beta cell function.
Two sides of MGP null arterial disease: chondrogenic lesions dependent on transglutaminase 2 and elastin fragmentation associated with induction of adipsin.
Factor D-deficient mice lacking the alternative complement pathway show significantly reduced survival and increased organ dysfunction, following cecal ligation and puncture when compared with control mice.
local expression of adipsin has a reproductive effect at the feto-maternal interface and possibly plays a role in spontaneous abortion
MASP-1 converted pro-Df to the active form in vitro, although the activation mechanism of pro-Df by MASP-1 is still unclear. Thus, it is clear that MASP-1 is an essential protease of both the lectin and alternative complement pathways.
Adipsin expression was not detected in any transfected clones of 3T3L1 cells exhibiting a complete blockage to differentiation.
Adipsin is a biomarker of gastrointestinal toxicity mediated by a functional gamma-secretase inhibitor
Mesangial immune complex glomerulonephritis is due to Cfd deficiency.
In the process of spontaneous abortion, the innate immune system through adipsin and complement C3 appears to be influential.
Isolation and expression pattern of a novel gene, Ami in Xenopus laevis.
While Hunchback directly represses the eve stripe 3+7 enhancer, study found that in the eve stripe 2+7 enhancer, Hunchback repression is prevented by nearby sequences-this phenomenon is called counter-repression.
Pericardial cells that express the transcription factor Even Skipped adopt distinct fates along the anterior-posterior axis. Among them, the most anterior Antp-Ubx-AbdA-negative cells form a novel cardiac outflow component we call the outflow hanging structure, whereas the Antp-expressing cells differentiate into wing heart precursors.
natural variation in a conserved trait-even-skipped (eve) expression at the cellular blastoderm stage of embryonic development in Drosophila melanogaster, was investigated.
Ubiquitous TER94 expression is "replaced" by expression in an eve pattern when Homie is deleted, and this effect is reversed when the PRE is also removed
Deletion of any individual enhancer reduced Yan occupancy at the other elements, impacting eve expression, cell fate specification, and cardiac function
Data strongly suggests that the eve-grn transcriptional code controls axon guidance, in part, by regulating the level of unc-5 expression.
even-skipped genes were misexpressed in pho mutant embryos
analysis of combinatorial activation and concentration-dependent repression of the Drosophila even skipped stripe 3+7 enhancer; Zld is required for activation; Kni and Hb are dedicated repressors that function by direct DNA binding
Nmo regulates a subset of Eve activities by stimulating Eve-mediated suppression of the odd-skipped (odd) repressor
The repressor activity of Even-skipped is highly conserved, and is sufficient to activate engrailed and to regulate both the spacing and stability of parasegment boundaries.
three distinct mechanisms are required for anterior repression of a single eve enhancer, each in a specific position
the eve enhancer integrates multiple positive and negative transcription factor inputs to restrict eve expression to a single precursor cell and its motoneuron progeny
opposing gradients of two Drosophila transcriptional repressors, Hunchback (Hb) and Knirps (Kni), position several segments by differentially repressing two distinct regulatory regions (enhancers) of the pair-rule gene even-skipped (eve)
The pair-rule segmentation gene even skipped (eve) is required to activate engrailed stripes and to organize odd-numbered parasegments (PSs).
Functional genetic complementation analysis of a eukaryotic cis-regulatory module-the even-skipped stripe 2 enhancer-from four Drosophila species.
A high-throughput method allows for the quantification of eve expression in early embryos.
The homeobox transcription factor even-skipped regulates netrin-receptor expression to control dorsal motor-axon projections in Drosophila.
in developing Drosophila cardiac progenitor cells, the Eve precursors next to the Hh stripe are distinguished from more distant Lbe precursors by locally augmenting Ras signaling via elevating rho transcripts
An eve-->grn-->zfh1 genetic cascade is unique to one of the intersegmental motoneurons, the aCC.
A model of the transcriptional readout of the proximal 1.7 kb of the control region of the Drosophila melanogaster gene even skipped (eve).
The protein encoded by this gene is a member of the trypsin family of peptidases. The encoded protein is a component of the alternative complement pathway best known for its role in humoral suppression of infectious agents. This protein is also a serine protease that is secreted by adipocytes into the bloodstream. Finally, the encoded protein has a high level of expression in fat, suggesting a role for adipose tissue in immune system biology.
C3 convertase activator
, D component of complement (adipsin)
, complement factor D
, complement factor D preproprotein
, properdin factor D
, 28 kDa adipocyte protein
, D component (adipsin) of complement
, endogenous vascular elastase
, complement factor D (adipsin)
, serine protease ami
, complement factor D (adipsin) like
, complementation group F
, group V
, group VI
, LOW QUALITY PROTEIN: complement factor D