Methamphetamine anticorps
Cet anticorps Mouton Polyclonal détecte spécifiquement Methamphetamine dans EIA.
N° du produit ABIN318601
1.185,85 €
Plus frais de livraison 40,00 € et TVA
1 mL
Destination:
France
Envoi sous 17 jours ouvrables
Aperçu rapide pour Methamphetamine anticorps (ABIN318601)
Antigène
Methamphetamine (M-Amp)
Hôte
Mouton
Clonalité
Polyclonal
Conjugué
Cet anticorp Methamphetamine est non-conjugé
Application
Enzyme Immunoassay (EIA)
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Purification
- Purified IgG prepared by affinity chromatography on Protein G
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Immunogène
- Para-amino-metamphetamine conjugated to keyhole limpet hemocyanin
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Isotype
- IgG
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Indications d'application
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ELISA.
Other applications not tested.
Optimal dilutions are dependent on conditions and should be determined by the user. -
Restrictions
- For Research Use only
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Format
- Liquid
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Concentration
- 5.0 mg/mL
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Buffer
- Phosphate buffered saline containing 0.09 % Sodium Azide (NaN3)
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Agent conservateur
- Sodium azide
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Précaution d'utilisation
- This product contains Sodium Azide: a POISONOUS AND HAZARDOUS SUBSTANCE, which should be handled by trained staff only.
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Conseil sur la manipulation
- Avoid repeated freezing and thawing.
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Stock
- 4 °C/-20 °C
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Stockage commentaire
- Store the antibody undiluted at 2-8 °C for one month or (in aliquots) at -20 °C for longer.
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- Methamphetamine (M-Amp)
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Autre désignation
- Methamphetamine
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Classe de substances
- Chemical
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Sujet
- Methamphetamine (METH) is closely related chemically to amphetamine (AMPH). METH is a potent central nervous system stimulant with additional peripheral sympathomimetic effects. METH and AMPH have been used clinically in the treatment of obesity, minimal brain dysfunction, narcolepsy, depression and to counter fatigue. They are also subjected to widespread abuse. METH is an indirect agonists. It causes the release of newly synthesized norepinephrine and dopamine and it blocks the re uptake of these transmitters from the synapse. This can lead to an increase in the concentration of catecholamines in the synapse as well as an overall increase in catecholaminergic activity in the brain. The mechanism of METH induced neurotoxicity for all monoaminergic cell types may lie primarily with the dopaminergic system in the striatum. It may also lie with the interaction between METH induced release of dopamine and its ability to inhibit monoamine oxidase.
Antigène
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