SIRPG anticorps (PE)

N° du produit ABIN2663096

Détail du produit anti-SIRPG anticorps (PE)

Antigène: SIRPG (Signal-Regulatory Protein gamma (SIRPG))

Reactivité: Humain

Hôte: Souris

Clonalité: Monoclonal

Conjugué: Cet anticorp SIRPG est conjugé à/à la PE

Application: Western Blotting (WB), ELISA, Flow Cytometry (FACS), Immunohistochemistry (IHC)

Purification: The antibody was purified by affinity chromatography, and conjugated with PE under optimal conditions. The solution is free of unconjugated PE and unconjugated antibody.

Clone: LSB2-20

Isotype: IgG1 kappa

Information d'application

Indications d'application: Optimal working dilution should be determined by the investigator.

Restrictions: For Research Use only

Stockage

Buffer: Phosphate-buffered solution, pH 7.2, containing 0.09 % sodium azide and 0.2 % (w/v) BSA .

Agent conservateur: Sodium azide

Précaution d'utilisation: This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

Conseil sur la manipulation: Protect from prolonged exposure to light. Do not freeze.

Stock: 4 °C

Stockage commentaire: The antibody solution should be stored undiluted between 2°C and 8°C.

Détails sur SIRPG

Antigène: SIRPG (Signal-Regulatory Protein gamma (SIRPG))

Autre désignation: CD172g (SIRPG Produits)

Synonymes: anticorps CD172g, anticorps SIRP-B2, anticorps SIRPB2, anticorps SIRPgamma, anticorps bA77C3.1, anticorps SIRPG, anticorps signal regulatory protein gamma, anticorps signal-regulatory protein gamma, anticorps SIRPG, anticorps LOC716034, anticorps LOC102138723

Sujet: Signal Regulatory Proteins (SIRPs) comprise a family of several transmembrane glycoproteins (SIRPα, SIRPβ, and SIRPγ) that belong to immunoglobulin superfamily. SIRPγ is also known as CD172g or SIRPβ2 which is primarily expressed on T cells and a subpopulation of B cells. Like CD172a, the ligand of CD172g is CD47. But the binding affinity with CD172g is lower than CD172a. The biological functions of CD172g include regulation of T-cell transendothelial migration and induction of T cell apoptosis.