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TNFSF18 anticorps
TNFSF18
Reactivité: Souris
WB, FACS, IHC, IP
Hôte: Rat
Monoclonal
5F1
unconjugated
N° du produit ABIN2664998
Détail du produit anti-TNFSF18 anticorps
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Antigène
Voir toutes TNFSF18 Anticorps
TNFSF18
(Tumor Necrosis Factor (Ligand) Superfamily, Member 18 (TNFSF18))
Reactivité
Toutes les réactivités sur TNFSF18 Anticorps
Souris
Hôte
Toutes les hôtes sur TNFSF18 Anticorps
Rat
Clonalité
Toutes les clonalités sur TNFSF18 Anticorps
Monoclonal
Conjugué
Tous les conjugués à travers TNFSF18 Anticorps
Cet anticorp TNFSF18 est non-conjugé
Application
Tous les applications à travers TNFSF18 Anticorps.
Western Blotting (WB), Flow Cytometry (FACS), Immunohistochemistry (IHC), Immunoprecipitation (IP)
Purification
The antibody was purified by affinity chromatography.
Clone
5F1
Isotype
IgG2a kappa
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Information d'application
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Indications d'application
Optimal working dilution should be determined by the investigator.
Restrictions
For Research Use only
Stockage
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Concentration
0.5 mg/mL
Buffer
Phosphate-buffered solution, pH 7.2, containing 0.09 % sodium azide.
Agent conservateur
Sodium azide
Précaution d'utilisation
This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
Stock
4 °C
Stockage commentaire
The antibody solution should be stored undiluted between 2°C and 8°C.
Détails sur TNFSF18
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Antigène
TNFSF18
(Tumor Necrosis Factor (Ligand) Superfamily, Member 18 (TNFSF18))
Autre désignation
GITR Ligand (TNFSF18 Produits )
Synonymes
anticorps Gitrl, anticorps AITRL, anticorps GITRL, anticorps TL6, anticorps hGITRL, anticorps TNLG2A, anticorps tumor necrosis factor (ligand) superfamily, member 18, anticorps TNF superfamily member 18, anticorps Tnfsf18, anticorps TNFSF18
Sujet
GITR ligand (GITRL) is a member of the TNF superfamily, also known as TNFSF18 or TL6. It is expressed on B cells, macrophages, dendritic cells, and endothelial cells. The expression of GITRL on the dendritic cell surface can be transiently upregulated, and then rapidly downregulated, by LPS stimulation. GITRL binds to its receptor, GITR, to costimulate T cell activation, proliferation, and cytokine production, inhibit the suppressive functions of CD4+CD25+ Tregs, and induce macrophage activation. GITR-GITRL interaction has been implicated as an essential regulator of the immune response to self antigens and phathogens.
Vus récemment
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