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Methamphetamine P NH2 anticorps

L’anticorps anti-Methamphetamine P NH2 Polyclonal Mouton est utilisé pour la détection de Methamphetamine P NH2. Il a été validé pour EIA.
N° du produit ABIN487758
1.092,31 €
Plus frais de livraison 40,00 € et TVA
0.1 mL
Destination: France
Envoi sous 17 jours ouvrables

Aperçu rapide pour Methamphetamine P NH2 anticorps (ABIN487758)

Antigène

Methamphetamine P NH2

Hôte

Mouton

Clonalité

Polyclonal

Application

Enzyme Immunoassay (EIA)
  • Purification

    Ig Fraction

    Immunogène

    Methamphetamine (p)-BSA

    Isotype

    IgG
  • Indications d'application

    ELISA: , 2.5 μg/mL.
    Other applications not tested.
    Optimal dilutions are dependent on conditions and should be determined by the user.

    Restrictions

    For Research Use only
  • Format

    Liquid

    Concentration

    9.72mg/mL (U.V. abs at 280nm)

    Buffer

    20 mM Phosphate, 150 mM Sodium Chloride, pH 7.2 containing 0.09 % Sodium Azide as preservative

    Agent conservateur

    Sodium azide

    Précaution d'utilisation

    This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

    Conseil sur la manipulation

    Avoid repeated freezing and thawing.

    Stock

    -20 °C
  • Antigène

    Methamphetamine P NH2

    Autre désignation

    Methamphetamine (P)

    Classe de substances

    Chemical

    Sujet

    Methamphetamine (METH) is closely related chemically to amphetamine (AMPH). METH is a potent central nervous system stimulant with additional peripheral sympathomimetic effects. METH and AMPH have been used clinically in the treatment of obesity, minimal brain dysfunction, narcolepsy, depression and to counter fatigue. They are also subjected to widespread abuse. METH is an indirect agonists. It causes the release of newly synthesized norepinephrine and dopamine and it blocks the re uptake of these transmitters from the synapse. This can lead to an increase in the concentration of catecholamines in the synapse as well as an overall increase in catecholaminergic activity in the brain. The mechanism of METH induced neurotoxicity for all monoaminergic cell types may lie primarily with the dopaminergic system in the striatum. It may also lie with the interaction between METH induced release of dopamine and its ability to inhibit monoamine oxidase.Synonyms: METH
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