NDUFA13 anticorps (AA 159-172)

N° du produit ABIN636278

L’anticorps Lapin Polyclonal anti-NDUFA13 a été validé pour WB. Il convient pour détecter NDUFA13 dans des échantillons de Humain.

Aperçu rapide pour NDUFA13 anticorps (AA 159-172) (ABIN636278)

Antigène: NDUFA13 (NADH Dehydrogenase (Ubiquinone) 1 alpha Subcomplex, 13 (NDUFA13))

Reactivité: Humain

Hôte: Lapin

Clonalité: Polyclonal

Conjugué: Cet anticorp NDUFA13 est non-conjugé

Application: Western Blotting (WB)

Détail du produit anti-NDUFA13 anticorps

Épitope: AA 159-172

Réactivité croisée (Details): This antibodys immunogens shares 85 % sequence identity with its Mouse and rat homologs.

Purification: Affinity chromatography purified

Immunogène: GRIM19 antibody was raised in rabbit using AA 159-172 QAETDRRTLQMLRE of the human GRIM-19 protein as the immunogen.

Isotype: IgG

Information d'application

Indications d'application: WB: 1:500-1:1,000
Optimal conditions should be determined by the investigator.

Restrictions: For Research Use only

Stockage

Concentration: Lot specific

Buffer: Affinity purified IgG supplied in PBS with 0.02 % NaN3.

Agent conservateur: Sodium azide

Précaution d'utilisation: This product contains Sodium Azide: a POISONOUS AND HAZARDOUS SUBSTANCE, which should be handled by trained staff only.

Conseil sur la manipulation: Avoid repeated freeze/thaw cycles.
Dilute only prior to immediate use.

Stock: 4 °C/-20 °C

Stockage commentaire: Store at 4 °C for short term storage. Aliquot and store at -20 °C for long term storage.

Détails sur NDUFA13

Antigène: NDUFA13 (NADH Dehydrogenase (Ubiquinone) 1 alpha Subcomplex, 13 (NDUFA13))

Autre désignation: GRIM19

Sujet: Antisense expression of Genes associated with Retinoid-IFN-induced Mortality (GRIM), GRIM-19 confers a strong resistance against IFN/RA-induced death by reducing the intracellular levels of GRIM-19 protein. Overexpression of GRIM-19 enhances cell death in response to IFN/RA. GRIM-19 is primarily a nuclear protein whose expression is induced by the IFN/RA combination.

Pathways: Protein targeting to Nucleus, Negative Regulation of intrinsic apoptotic Signaling