Influenza A Virus H1N1 anticorps

N° du produit ABIN643615

Cet anticorps Chèvre not_set détecte spécifiquement Influenza A Virus H1N1 dans IF, IHC (p) et H. Il présente une réactivité avec des échantillons de Influenza A Virus H1N1.

Aperçu rapide pour Influenza A Virus H1N1 anticorps (ABIN643615)

Antigène: Influenza A Virus H1N1

Reactivité: Influenza A Virus H1N1

Hôte: Chèvre

Conjugué: Inconjugué

Application: Immunofluorescence (IF), Immunohistochemistry (Paraffin-embedded Sections) (IHC (p)), Haemagglutination (H)

Détail du produit

Specificité: DS-PB-01152 is specific for Influenza A virus H1N1 glycoprotein and does not react with Influenza B, RSV, Para 1-3 or Adenovirus. It does not react with HEp-2 cells but may react with some chicken cellular proteins.

Attributs du produit: Goat Anti-Influenza A H1N1

Purification: Purified

Immunogène: Influenza A, strain USSR (H1N1)

Isotype: IgG

Information d'application

Restrictions: For Research Use only

Stockage

Format: Liquid

Concentration: IgG concentration 1.0 mg/ml

Buffer: Phosphate buffered saline pH 7.2

Agent conservateur: Sodium azide

Précaution d'utilisation: This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

Stock: 4 °C

Détail du antigène

Antigène: Influenza A Virus H1N1

Autre désignation: Influenza A H1N1

Classe de substances: Influenza Virus

Sujet: Influenza type A viruses are divided into subtypes based on the antigenic differences of two viral surface proteins, hemagglutinin (H) and neuraminidase (N). On infection of the respiratory tract, the hemagglutinin molecule binds to sialic acid-containing receptors on the epithelial cells resulting in endocytosis. Once the virus has been engulfed, the hemagglutinin allows the viral membrane to fuse with the endosomal membrane. Neuraminidase functions to aid viral release from host cells by cleaving terminal sialic acid residues from carbohydrate moieties on the cell surface. Subtype antigenic variations result from a process known as antigenic drift whereby these surface proteins constantly mutate in order to evade the host immune response. Subtype A (H1N1) was the cause of Spanish flu that killed approximately 500,000 people between 1918-1919.