KCNJ6 anticorps (FITC)

N° du produit ABIN705348

Cet anticorps anti-KCNJ6 est un anticorps Lapin Polyclonal détectant KCNJ6 dans WB. Adapté pour Humain, Souris et Rat.

Aperçu rapide pour KCNJ6 anticorps (FITC) (ABIN705348)

Antigène: KCNJ6 (Potassium Inwardly-Rectifying Channel, Subfamily J, Member 6 (KCNJ6))

Reactivité: Humain, Souris, Rat

Hôte: Lapin

Clonalité: Polyclonal

Conjugué: Cet anticorp KCNJ6 est conjugé à/à la FITC

Application: Western Blotting (WB)

Détail du produit anti-KCNJ6 anticorps (FITC)

Réactivité croisée: Humain, Souris, Rat

Purification: Purified by Protein A.

Immunogène: KLH conjugated synthetic peptide derived from human GIRK2

Isotype: IgG

Information d'application

Indications d'application: IF(IHC-P)(1:50-200)

Restrictions: For Research Use only

Stockage

Format: Liquid

Concentration: 1 μg/μL

Buffer: Aqueous buffered solution containing 0.01M TBS ( pH 7.4) with 1 % BSA, 0.03 % Proclin300 and 50 % Glycerol.

Agent conservateur: Sodium azide

Précaution d'utilisation: This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

Stock: -20 °C

Stockage commentaire: Store at -20°C. Aliquot into multiple vials to avoid repeated freeze-thaw cycles.

Date de péremption: 12 months

Détails sur KCNJ6

Antigène: KCNJ6 (Potassium Inwardly-Rectifying Channel, Subfamily J, Member 6 (KCNJ6))

Autre désignation: Girk2

Sujet:

Synonyms: inwardly rectying subfamily J member 6, Kir3.2, BIR1, G protein activated inward rectier potassium channel 2, G protein-activated inward rectier potassium channel 2, GIRK-2, Inward rectier K+ channel Kir3.2, IRK6_HUMAN, KATP-2, Kcnj6, Kcnj7, Potassium channel, Potassium channel inwardly rectying subfamily J member 6.

Background: This potassium channel may be involved in the regulation of insulin secretion by glucose and/or neurotransmitters acting through G-protein-coupled receptors. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium, as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium.

ID gène: 3763