IVNS1ABP anticorps (AA 50-100)

N° du produit ABIN7451899

Cet anticorps Lapin Polyclonal détecte spécifiquement IVNS1ABP dans WB et IP. Il présente une réactivité envers Humain.

Aperçu rapide pour IVNS1ABP anticorps (AA 50-100) (ABIN7451899)

Antigène: IVNS1ABP (Influenza Virus NS1A Binding Protein (IVNS1ABP))

Reactivité: Humain

Hôte: Lapin

Clonalité: Polyclonal

Conjugué: Cet anticorp IVNS1ABP est non-conjugé

Application: Western Blotting (WB), Immunoprecipitation (IP)

Détail du produit anti-IVNS1ABP anticorps

Épitope: AA 50-100

Fonction: Rabbit anti-NS1-BP Antibody, Affinity Purified

Purification: Affinity Purified

Immunogène: between AA 50 and 100

Isotype: IgG

Information d'application

Indications d'application:

IP: 2 - 5 μg/mg lysate

WB: 1:2,000 - 1:10,000

Restrictions: For Research Use only

Stockage

Concentration: 1000 μg/mL

Buffer: Tris-citrate/phosphate buffer, pH 7 to 8 containing 0.09 % Sodium Azide

Agent conservateur: Sodium azide

Précaution d'utilisation: This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

Stock: 4 °C

Date de péremption: 12 months

Détails sur IVNS1ABP

Antigène: IVNS1ABP (Influenza Virus NS1A Binding Protein (IVNS1ABP))

Autre désignation: NS1-BP

Classe de substances: Influenza Protein

Sujet: Background: NS1-binding protein (NS1-BP) plays a role in cell division and in the dynamic organization of the actin skeleton by functioning as a stabilizer of actin filaments via its association with F-actin through Kelch repeats. NS1-BP may be a component of the cellular splicing machinery with a role in pre-mRNA splicing and may mediate the inhibition of splicing by NS/influenza virus NS1A protein. NS1-BP has also been found to function as modifier of the AHR/Aryl hydrocarbon receptor pathway by increasing the concentration of AHR available to activate transcription [taken from the Universal Protein Resource (UniProt) Q9Y6YO].

ID gène: 10625

NCBI Accession: NP_006460

UniProt: Q9Y6Y0

Pathways: Negative Regulation of intrinsic apoptotic Signaling