MFAP1 anticorps (AA 83-437)

N° du produit ABIN7876941

L’anticorps anti-MFAP1 Polyclonal Lapin est utilisé pour la détection de MFAP1 dans des échantillons de Humain et Souris. Il a été validé pour WB, ELISA, FACS et IF.

Aperçu rapide pour MFAP1 anticorps (AA 83-437) (ABIN7876941)

Antigène: MFAP1 (Microfibrillar Associated Protein 1 (MFAP1))

Reactivité: Humain, Souris

Hôte: Lapin

Clonalité: Polyclonal

Conjugué: Cet anticorp MFAP1 est non-conjugé

Application: Western Blotting (WB), ELISA, Flow Cytometry (FACS), Immunofluorescence (IF)

Détail du produit anti-MFAP1 anticorps

Épitope: AA 83-437

Fonction: MFAP1 Antibody / Microfibrillar-associated protein 1

Purification: Antigen affinity purified

Immunogène: An E.coli-derived human recombinant protein (D83-K437) was used as the immunogen for the MFAP1 antibody.

Isotype: IgG

Information d'application

Indications d'application: Optimal dilution of the MFAP1 antibody should be determined by the researcher.

Restrictions: For Research Use only

Stockage

Format: Lyophilized

Buffer: 0.5 mg/mL if reconstituted with 0.2 mL sterile DI water

Stock: 4 °C,-20 °C

Stockage commentaire: After reconstitution, the MFAP1 antibody can be stored for up to one month at 4oC. For long-term, aliquot and store at -20oC. Avoid repeated freezing and thawing.

Détails sur MFAP1

Antigène: MFAP1 (Microfibrillar Associated Protein 1 (MFAP1))

Autre désignation: MFAP1

Sujet: Microfibrillar-associated protein 1 is a protein that in humans is encoded by the MFAP1 gene. Microfibrils are an important component of the extracellular matrix of many tissues and can either associate with or without elastin. Several microfibril associated proteins (MFAPs) have been cloned, including MFAP1, MFAP3 and MFAP4. The MFAP1 and MFAP3 genes are localized near the fibrillin genes FBN1 and FBN2, respectively. Mutations in FBN1 are linked to Marfan syndrome. Mutations in FBN2 have been linked to congenital contractural arachnodactyly. This suggests roles for MFAP1 and MFAP3 in heritable diseases affecting microfibrils. Deletion of MFAP4 was found in 30 of 31 patients with Smith-Magenis syndrome (SMS), a clinically recognizable multiple congenital anomaly/mental retardation syndrome

UniProt: P55081