CCL19 Protéine
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- Antigène Voir toutes CCL19 Protéines
- CCL19 (Chemokine (C-C Motif) Ligand 19 (CCL19))
- Type de proteíne
- Recombinant
- Activité biologique
- Active
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Origine
- Souris
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Source
- Escherichia coli (E. coli)
- Séquence
- GANDAEDCCL SVTQRPIPGN IVKAFRYLLN EDGCRVPAVV FTTLRGYQLC APPDQPWVDR IIRRLKKSSA KNKGNSTRRS PVS
- Attributs du produit
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Purified recombinant Mouse MIP3 beta protein
Expression System: E.coli
Bioactivity: Determined by its ability to chemoattract human mature dendritic cells using a concentration range of 10.0-100.0 ng/mL. - Pureté
- > 98 % pure
- niveau d'endotoxine
- < 0.1 ng per µg (1 EU/µg).
- Top Product
- Discover our top product CCL19 Protéine
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- Indications d'application
- Each Investigator should determine their own optimal working dilution for specific applications.
- Restrictions
- For Research Use only
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- Format
- Lyophilized
- Reconstitution
- Reconstitute in distilled water to 0.1-1.0 mg/mL.
- Buffer
- Supplied lyophilized with no additives.
- Agent conservateur
- Without preservative
- Conseil sur la manipulation
- Avoid repeated freeze/thaw cycles.
- Stock
- RT/-20 °C
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- Antigène
- CCL19 (Chemokine (C-C Motif) Ligand 19 (CCL19))
- Autre désignation
- MIP3 beta (CCL19 Produits)
- Synonymes
- CKb11 Protein, ELC Protein, MIP-3b Protein, MIP3B Protein, SCYA19 Protein, CCL19 Protein, Scya19 Protein, exodus-3 Protein, C-C motif chemokine ligand 19 Protein, chemokine (C-C motif) ligand 19 Protein, CCL19 Protein, ccl19 Protein, Ccl19 Protein
- Sujet
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MIP-3 beta is a CC chemokine that is expressed in the thymus, lymph nodes and in activated bone marrow stromal cells and signals through the CCR7 receptor. MIP-3 beta is a chemoattractant for T and B lymphocytes and myeloid progenitor cells. Human MIP-3 beta is active on Mouse cells.
Alternative Names: MIP beta-3, MIP beta-3 protein, ELC protein, Macrophage Inflammatory Protein-3 beta protein, MIP-3 beta protein, Exodus-3 protein, CCL19 protein, MIP beta 3 protein, MIP3 beta, MIP beta 3 - Poids moléculaire
- 9.2 kDa
- Pathways
- Positive Regulation of Immune Effector Process
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