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anti-Human CDKN2D Anticorps:
anti-Mouse (Murine) CDKN2D Anticorps:
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We unexpectedly found that p19INK4d plays an important role in human terminal erythropoiesis
p19(INK4D) is one of the key mediators of miR (Montrer MLXIP Anticorps)-125b activity during the onset of megakaryocyte polyploidization.
p19 may play an important role in the ototoxic effects of cisplatin and is probably involved in the pathogenesis of hearing loss.
MiR (Montrer MLXIP Anticorps)-451 inhibited the proliferation of esophageal squamous cell carcinoma cells by targeting CDKN2D and MAP3K1 (Montrer MAP3K1 Anticorps) expression.
Data indicate that upon oxidative DNA damage, cyclin-dependent kinase (Montrer CDK1 Anticorps) inhibitor p19INK4d strongly binds to and relaxes chromatin.
CDKN2D repression by PML (Montrer PML Anticorps)/RARalpha (Montrer RARA Anticorps) disrupts both cell proliferation and differentiation in the pathogenesis of acute promyelocytic leukemia (Montrer PML Anticorps).
p19-INK4d inhibits neuroblastoma (Montrer ARHGEF16 Anticorps) cell growth, induces differentiation and is hypermethylated and downregulated in MYCN (Montrer MYCN Anticorps)-amplified neuroblastomas.
CDKN2D-WDFY2 (Montrer ZFYVE22 Anticorps) fusion could be an important molecular signature for understanding and classifying sub-lineages among heterogeneous high-grade serous ovarian carcinomas.
Mutations in CDKN2D is associated with sporadic parathyroid adenoma.
P19(INK4d) expression is a poor prognostic factor in ovarian cancer patients.
The expression of three tumor suppressor genes encoded in the INK4/ARF (Montrer CDKN2A Anticorps) locus (p15(INK4b (Montrer CDKN2B Anticorps)), p16(INK4a (Montrer CDKN2A Anticorps)), and p19(ARF (Montrer CDKN2A Anticorps))) was decreased in E6AP (Montrer ube3a Anticorps)(-/-) embryo fibroblasts.
TWIST1 (Montrer TWIST1 Anticorps)-E protein heterodimeric complexes may thus constitute the main active forms of TWIST1 (Montrer TWIST1 Anticorps) with regard to senescence inhibition over the time course of breast tumorigenesis.
Rescue from early-onset hearing loss in a mouse model lacking the cyclin-dependent kinase (Montrer CDK1 Anticorps) inhibitor p19Ink4d.
p19 INK4d controls hematopoietic stem cells in a cell-autonomous manner during genotoxic stress and through the microenvironment during aging
Cdk4 (Montrer CDK4 Anticorps) and Cdk6 (Montrer CDK6 Anticorps) cooperate in hematopoietic tumor development and suggest a role for Cdk6 (Montrer CDK6 Anticorps) in sequestering INK4 (Montrer CDKN2A Anticorps) proteins away from Cdk4 (Montrer CDK4 Anticorps).
Deletion of p19(Ink4d) (p19), a cyclin-dependent kinase (Montrer CDK1 Anticorps) CDK (Montrer CDK4 Anticorps) inhibitor gene, in mice results in spontaneous development of tumors in multiple organs and tissues.
We propose that p19INK4d participates in the cellular mechanisms that trigger senescence by contributing to chromatin compaction
CDK5 (Montrer CDK5 Anticorps)-mediated phosphorylation of p19INK4d avoids DNA damage-induced neurodegeneration in mouse hippocampus and prevents loss of cognitive functions.
an asymmetrical distribution pattern for Cdkn2d transcripts in 2-cell embryos.
Ectopic expression of RUVBL2 (Montrer RUVBL2 Anticorps) decreases the levels of ARF (Montrer CDKN2A Anticorps), whereas knockdown of RUVBL2 (Montrer RUVBL2 Anticorps) results in a marked increase in ARF (Montrer CDKN2A Anticorps) levels. In addition, RUVBL2 (Montrer RUVBL2 Anticorps) down-regulates the levels of p53 (Montrer TP53 Anticorps) in an ARF (Montrer CDKN2A Anticorps)-dependent manner.
The protein encoded by this gene is a member of the INK4 family of cyclin-dependent kinase inhibitors. This protein has been shown to form a stable complex with CDK4 or CDK6, and prevent the activation of the CDK kinases, thus function as a cell growth regulator that controls cell cycle G1 progression. The abundance of the transcript of this gene was found to oscillate in a cell-cycle dependent manner with the lowest expression at mid G1 and a maximal expression during S phase. The negative regulation of the cell cycle involved in this protein was shown to participate in repressing neuronal proliferation, as well as spermatogenesis. Two alternatively spliced variants of this gene, which encode an identical protein, have been reported.
cyclin-dependent kinase inhibitor 2D (p19, inhibits CDK4)
, cyclin-dependent kinase 4 inhibitor D
, Cyclin-dependent kinase 4 inhibitor D
, CDK inhibitor p19INK4d
, cell cycle inhibitor, Nur77 associating protein
, cyclin-dependent kinase 4 inhibitor D p19
, inhibitor of cyclin-dependent kinase 4d