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Human GSTM1 Kit ELISA pour Sandwich ELISA - ABIN417506
Singh, Prasad, Singh, Singh, Gupta, Paliwal, Pandey, Gupta: Human Glutathione S-Transferase Enzyme Gene Polymorphisms and Their Association With Neurocysticercosis. dans Molecular neurobiology 2016
Human GSTM1 Kit ELISA pour Sandwich ELISA - ABIN840218
Wu, Lu, Tang, Zhang, Yuan, Li, Di Wu, Sun, Lu, Xia, Chen, Sha, Wang: GSTM1 and GSTT1 null polymorphisms and male infertility risk: an updated meta-analysis encompassing 6934 subjects. dans Scientific reports 2013
Polymorphisms of GSTM1 is associated with lung cancer.
The loss of GSTM1 was significantly associated with incident kidney and heart failure, independent of traditional risk factors. These results suggest GSTM1 function is a potential treatment target for the prevention of kidney and heart failure
results suggest that combined deletion polymorphisms of GSTT1 (Montrer GSTT1 Kits ELISA) and GSTM1 can have implications in the prediction of the clinical course of the disease
On comparing the prevalence of GSTM1 null polymorphism among the group with subjects with tobacco habits and no oral lesions, oral leukoplakia, and oral squamous cell carcinoma, it was observed that there was a statistically significant association between GSTM1 null polymorphism and the different groups (P < 0.01).
The present case-control study found that GSTM1 and GSTT1 (Montrer GSTT1 Kits ELISA) polymorphism are not associated with JOAG (Montrer MYOC Kits ELISA) risk in North Indian population. The present meta-analysis suggested that there might be a significant association of GSTM1 null genotype with glaucoma (JOAG (Montrer MYOC Kits ELISA) & POAG) risk.
Pesticide-exposed individuals with inherited susceptible metabolic genotypes (particularly, null genotype for GSTM1 and the PON1 (Montrer PON1 Kits ELISA) 192R allele) appear to have an increased risk of genotoxic DNA damage.
182 Hungarian bladder cancer cases and 78 cancer-free controls were investigated. It was not possible to establish a particular impact of NAT2 (Montrer SLC38A1 Kits ELISA)*6A and *7B genotypes (15 cases, 8%, 5 controls, 7%). GSTT1 (Montrer GSTT1 Kits ELISA) exerted no marked influence on bladder cancer (negative 21% cases vs. 22% controls). The portion of GSTM1 negative bladder cancer patients was increased (63% cases vs. 54% controls).
double-null genotype of the GSTM1 gene is not associated with ALL development or its progression.
We assessed the effect of allelic deletions in the GSTM1 and GSTT1 (Montrer GSTT1 Kits ELISA) genotypes on lung cancer overall survival through a systematic review of the scientific literature after applying predefined inclusion and exclusion criteria
GSTM1 genetic polymorphisms are not associated with the development of graft-versus-host disease after allogeneic hematopoietic stem cell transplantation.
Six proteins were regulated at both basal and inducible levels exhibiting the largest dynamic range of Nrf2 (Montrer NFE2L2 Kits ELISA) regulation: cytochrome CYP2A5, GSTM3 (Montrer glutathione S-transferase mu 3 (brain) Kits ELISA), GSTM1, ENTPD5 (Montrer ENTPD5 Kits ELISA),UDPGDH (Montrer UGDH Kits ELISA), and EPHX1 (Montrer EPHX1 Kits ELISA).
mitochondrial Gstb1 is induced under oxidative stress
the gene (GSTM1) encoding the detoxification enzyme glutathione S-transferase M1 is transcriptionally upregulated by Myb (Montrer MYB Kits ELISA)
The Gstm1 gene was represented by two EST (Montrer MAP3K8 Kits ELISA) clones with similar levels of downregulation.
We overexpressed Hoxa9 (Montrer HOXA9 Kits ELISA) and Meis1 (Montrer MEIS1 Kits ELISA) in primary hematopoietic cells. Arrays identified c-Myb (Montrer MYB Kits ELISA) and a c-Myb (Montrer MYB Kits ELISA) target (Gstm1) among the genes with the strongest response to Hoxa9 (Montrer HOXA9 Kits ELISA)/Meis1 (Montrer MEIS1 Kits ELISA).
Genetic variants that cause a decremental change in expression of Gstm1 may permit an environment of exaggerated oxidative stress, leading to susceptibility to vascular remodeling and atherosclerosis
The role of polymorphisms of glutathione S-transferases GSTM1, M3, P1, T1 and A1 in susceptibility to alcoholic liver disease. In addition, oxidant stress is proposed to be an important pathogenic factor in liver damage related to alcohol.
Cytosolic and membrane-bound forms of glutathione S-transferase are encoded by two distinct supergene families. At present, eight distinct classes of the soluble cytoplasmic mammalian glutathione S-transferases have been identified: alpha, kappa, mu, omega, pi, sigma, theta and zeta. This gene encodes a glutathione S-transferase that belongs to the mu class. The mu class of enzymes functions in the detoxification of electrophilic compounds, including carcinogens, therapeutic drugs, environmental toxins and products of oxidative stress, by conjugation with glutathione. The genes encoding the mu class of enzymes are organized in a gene cluster on chromosome 1p13.3 and are known to be highly polymorphic. These genetic variations can change an individual's susceptibility to carcinogens and toxins as well as affect the toxicity and efficacy of certain drugs. Null mutations of this class mu gene have been linked with an increase in a number of cancers, likely due to an increased susceptibility to environmental toxins and carcinogens. Multiple protein isoforms are encoded by transcript variants of this gene.
GST HB subunit 4
, GST class-mu 1
, HB subunit 4
, S-(hydroxyalkyl)glutathione lyase
, glutathione S-alkyltransferase
, glutathione S-aralkyltransferase
, glutathione S-aryltransferase
, glutathione S-transferase M1
, glutathione S-transferase Mu 1
, GST 3-3
, GST Yb1
, glutathione S-transferase Yb-1 subunit
, glutathione-S-transferase mu type 1 (Yb1)
, glutathione-S-transferase, mu type 1 (Yb1)
, glutathione S-transferase M4
, glutathione S-transferase mu 4
, GST 1-1
, glutathione S-transferase GT8.7
, glutathione-S-transferase, mu 1
, glutathione S-transferase mu 1
, glutathione S-transferase mu 2
, glutathione S-transferase, mu 2