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anti-Human VEGFB Anticorps:
anti-Mouse (Murine) VEGFB Anticorps:
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Data show that metformin treatment reduces serum vascular endothelial growth factor B (VEGF-B) levels and ameliorates insulin (Montrer INS Anticorps) resistance.
VEGFA (Montrer VEGFA Anticorps) and VEGFB associated sub-network, kinase insert domain receptor (KDR (Montrer KDR Anticorps)), fibronectin 1 (FN1 (Montrer FN1 Anticorps)), transforming growth factor beta induced (TGFBI (Montrer TGFBI Anticorps)) and proliferating cell nuclear antigen (PCNA (Montrer PCNA Anticorps)) found to interact with at least two of the three hub genes.
Frameshift mutations of VEGFB gene is associated with stomach and colorectal cancers.
plasma VEGF levels before treatment were lower in patients with schizophrenia and that their VEGF levels increased after treatment.
fluid shear stress induces the synthesis of Insulin growth factor-2 and vascular endothelial growth factor (VEGF) B and D, which in turn transactivate MMP-12.
MMP9 (Montrer MMP9 Anticorps) may activate VEGF-B via PI3K (Montrer PIK3CA Anticorps)/Akt (Montrer AKT1 Anticorps) signaling pathway.
Roles of vascular endothelial growth factor in amyotrophic lateral sclerosis.
Low VEGFB and VEGFD (Montrer Figf Anticorps) gene expression is associated with early-stage non-small cell lung cancer.
Our study suggested that VEGF-B was an angiogenesis factor in vitro and that ERK1/2 (Montrer MAPK1/3 Anticorps) and p38 (Montrer CRK Anticorps)-related signaling pathways were involved in these VEGF-B activities.
VEGF-B has possible roles in cardiac protection, energy metabolism support, and neuroprotectin [review]
These data therefore support a tightly controlled, paracrine signaling mechanism of VEGF-B to VEGFR1 (Montrer FLT1 Anticorps).
VEGFB-Induced Vascular Remodeling in Adipose Tissue Requires VEGF (Montrer VEGFA Anticorps)/VEGFR2 (Montrer KDR Anticorps). VEGFB-Induced Vascular Remodeling Improves Insulin (Montrer INS Anticorps) Supply, Signaling, and Function in Obese Mice.
VEGF-B is dispensable for normal cardiac function under unstressed conditions and for high fat diet-induced metabolic changes.
VEGF-B is a vascular remodeling factor promoting cancer metastasis.
Data indicate that vascular endothelial growth factor B knockout (Vegf-b-/-) mice showed impaired nerve repair with concomitant impaired trophic function.
Data indicate that VEGF-B is a high-affinity VEGFR-1 (Montrer FLT1 Anticorps) ligand that, unlike PlGF (Montrer PGF Anticorps), cannot efficiently induce signaling downstream of VEGFR-1 (Montrer FLT1 Anticorps).
results demonstrate that the vascular endothelium can function as an efficient barrier to excess muscle lipid uptake even under conditions of severe obesity and type 2 diabetes, and that this barrier can be maintained by inhibition of VEGF-B signalling
the RTEF-1 (Montrer TEAD4 Anticorps)-driven increase of VEGF-B plays an important role in communication between the endothelium and myocardium
Study indicates that VEGF-B, instead of acting as an angiogenic factor (Montrer VEGFA Anticorps), exerts direct neuroprotective effects through FLT1 (Montrer FLT1 Anticorps).
Data show that no differences in vascular density, perfusion or immune cell infiltration upon altered Vegfb gene dosage were noted.
PCR-SSCP and DNA sequencing methods were applied to reveal 3 SNPs and a duplication in the bovine VEGF-B gene among 675 samples belonging to three native Chinese cattle breeds.
This gene encodes a member of the PDGF (platelet-derived growth factor)/VEGF (vascular endothelial growth factor) family. The VEGF family members regulate the formation of blood vessels and are involved in endothelial cell physiology. This member is a ligand for VEGFR-1 (vascular endothelial growth factor receptor 1) and NRP-1 (neuropilin-1). Studies in mice showed that this gene was co-expressed with nuclear-encoded mitochondrial genes and the encoded protein specifically controlled endothelial uptake of fatty acids. Alternatively spliced transcript variants encoding distinct isoforms have been identified.