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IRF5 anticorps

IRF5 Reactivité: Humain BCA Hôte: Souris Monoclonal 11F4A09 unconjugated
N° du produit ABIN2666392
  • Antigène Voir toutes IRF5 Anticorps
    IRF5 (Interferon Regulatory Factor 5 (IRF5))
    Reactivité
    • 69
    • 48
    • 22
    • 6
    • 5
    • 5
    • 4
    • 4
    • 3
    • 3
    • 3
    Humain
    Hôte
    • 72
    • 8
    • 1
    • 1
    Souris
    Clonalité
    • 65
    • 17
    Monoclonal
    Conjugué
    • 50
    • 6
    • 5
    • 3
    • 3
    • 3
    • 3
    • 3
    • 3
    • 3
    Cet anticorp IRF5 est non-conjugé
    Application
    • 46
    • 29
    • 10
    • 10
    • 9
    • 5
    • 4
    • 4
    • 2
    • 1
    • 1
    • 1
    • 1
    Biochemical Assay (BCA)
    Clone
    11F4A09
    Isotype
    IgG2b kappa
    Top Product
    Discover our top product IRF5 Anticorps primaire
  • Indications d'application
    Optimal working dilution should be determined by the investigator.
    Restrictions
    For Research Use only
  • Concentration
    0.5 mg/mL
    Buffer
    phosphate-buffered solution, pH 7.2, containing 0.05 % sodium azide.
    Agent conservateur
    Sodium azide
    Précaution d'utilisation
    This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
    Stock
    4 °C
    Stockage commentaire
    Upon receipt, store undiluted between 2°C and 8°C.
  • Antigène
    IRF5 (Interferon Regulatory Factor 5 (IRF5))
    Autre désignation
    IRF5 (IRF5 Produits)
    Synonymes
    anticorps IRF5, anticorps irf5, anticorps SLEB10, anticorps AW491843, anticorps mirf5, anticorps im:7155364, anticorps zgc:76986, anticorps interferon regulatory factor 5, anticorps interferon regulatory factor 5 L homeolog, anticorps IRF5, anticorps irf5, anticorps Irf5, anticorps irf5.L
    Sujet
    IRF-5 is a transcription factor belonging to the interferon regulatory factor family. It controls induction of type I IFN genes as well as inflammatory cytokines and chemokines through TLRs and the MyD88-TRAF6 pathway. Several genetic variations of IRF-5 have been reported as a risk factor of Systemic lupus erythematosus (SLE), which is caused by excess IFNα induction.
    Pathways
    Signalisation TLR, Autophagy
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