BAI1 anticorps

N° du produit ABIN636927

Détail du produit anti-BAI1 anticorps

Antigène: BAI1 (Brain-Specific Angiogenesis Inhibitor 1 (BAI1))

Reactivité: Humain

Hôte: Lapin

Clonalité: Polyclonal

Conjugué: Cet anticorp BAI1 est non-conjugé

Application: Immunohistochemistry (Paraffin-embedded Sections) (IHC (p))

Purification: Affinity chromatography purified

Immunogène: BAI1 antibody was raised in rabbit using a synthetic peptide conjugated to KLHd as the immunogen.

Information d'application

Indications d'application: IHC-P: 20 µg/mL
Optimal conditions should be determined by the investigator.

Restrictions: For Research Use only

Stockage

Concentration: Lot specific

Buffer: Purified by Immunoaffinity Chromatography and supplied in PBS with 0.1 % NaN3.

Agent conservateur: Sodium azide

Précaution d'utilisation: This product contains Sodium Azide: a POISONOUS AND HAZARDOUS SUBSTANCE, which should be handled by trained staff only.

Conseil sur la manipulation: Avoid repeated freeze/thaw cycles.
Dilute only prior to immediate use.

Stock: 4 °C/-20 °C

Stockage commentaire: Store at 4 °C for short term storage. Aliquot and store at -70 °C for long term storage.

Détails sur BAI1

Antigène: BAI1 (Brain-Specific Angiogenesis Inhibitor 1 (BAI1))

Autre désignation: BAI1 (BAI1 Produits)

Synonymes: anticorps BAI1, anticorps GDAIF, anticorps B830018M07Rik, anticorps R75078, anticorps mKIAA4089, anticorps brain-specific angiogenesis inhibitor 1, anticorps adhesion G protein-coupled receptor B1, anticorps BAI1, anticorps ADGRB1, anticorps LOC100228084, anticorps Adgrb1

Sujet: Brain-specific angiogenesis inhibitor 1 is a protein that in humans is encoded by the BAI1 gene. Angiogenesis is controlled by a local balance between stimulators and inhibitors of new vessel growth and is suppressed under normal physiologic conditions. Angiogenesis has been shown to be essential for growth and metastasis of solid tumors. In order to obtain blood supply for their growth, tumor cells are potently angiogenic and attract new vessels as results of increased secretion of inducers and decreased production of endogenous negative regulators.

Pathways: Signalisation p53