CLN5 anticorps (AA 140-220)

N° du produit ABIN7219404

Cet anticorps anti-CLN5 Polyclonal Lapin (ABIN7219404) détecte spécifiquement CLN5 dans WB et ELISA. L’anticorps est réactif avec des échantillons de Humain, Rat et Souris.

Aperçu rapide pour CLN5 anticorps (AA 140-220) (ABIN7219404)

Antigène: CLN5 (Ceroid-Lipofuscinosis, Neuronal 5 (CLN5))

Reactivité: Humain, Rat, Souris

Hôte: Lapin

Clonalité: Polyclonal

Conjugué: Cet anticorp CLN5 est non-conjugé

Application: Western Blotting (WB), ELISA

Détail du produit anti-CLN5 anticorps

Épitope: AA 140-220

Fonction: Rabbit Anti-CLN5 Polyclonal Antibody

Specificité: CLN5 Polyclonal Antibody detects endogenous levels of CLN5 protein.

Purification: The antibody was affinity-purified from rabbit antiserum by affinity-chromatography using epitope-specific immunogen.

Immunogène: Synthesized peptide derived from the Internal region of human CLN5 at AA range: 140-220

Isotype: IgG

Information d'application

Indications d'application: Optimal working dilutions should be determined experimentally by the investigator. Suggested starting dilutions are as follows: WB 1:500-1:2000,ELISA 1:10000,Not yet tested in other applications.

Restrictions: For Research Use only

Stockage

Format: Liquid

Concentration: 1 mg/mL

Buffer: Liquid in PBS containing 50 % glycerol, 0.5 % BSA and 0.02 % sodium azide.

Agent conservateur: Sodium azide

Précaution d'utilisation: This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

Stock: -20 °C

Stockage commentaire: Stable for one year at -20°C from date of shipment. For maximum recovery of product, centrifuge the original vial after thawing and prior to removing the cap. Aliquot to avoid repeated freezing and thawing.

Date de péremption: 12 months

Détails sur CLN5

Antigène: CLN5 (Ceroid-Lipofuscinosis, Neuronal 5 (CLN5))

Autre désignation: CLN5

Sujet: CLN5, Ceroid-lipofuscinosis neuronal protein 5, Protein CLN5CLN5 is one of eight which have been associated with neuronal ceroid lipofuscinoses (NCL). Also referred to as Batten disease, NCL comprises a class of autosomal recessive, neurodegenerative disorders affecting children. The genes responsible likely encode proteins involved in the degradation of post-translationally modified proteins in lysosomes. The primary defect in NCL disorders is thought to be associated with lysosomal storage function.

Poids moléculaire: 48kD

ID gène: 1203

UniProt: O75503