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MAGE-Like 2 anticorps (AA 578-847)

Cet anticorps Lapin Polyclonal détecte spécifiquement MAGE-Like 2 dans ELISA, WB, IHC (p) et FACS. Il présente une réactivité avec des échantillons de Humain.
N° du produit ABIN7875833
644,88 €
Plus frais de livraison 40,00 € et TVA
100 μg
Destination: France
Envoi sous 6 à 9 jours ouvrables

Aperçu rapide pour MAGE-Like 2 anticorps (AA 578-847) (ABIN7875833)

Antigène

Voir toutes MAGE-Like 2 (MAGEL2) Anticorps
MAGE-Like 2 (MAGEL2)

Reactivité

  • 34
  • 2
  • 2
  • 2
  • 2
  • 1
Humain

Hôte

  • 34
  • 1
Lapin

Clonalité

  • 34
  • 1
Polyclonal

Conjugué

  • 10
  • 2
  • 2
  • 2
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
Cet anticorp MAGE-Like 2 est non-conjugé

Application

  • 13
  • 13
  • 11
  • 8
  • 8
  • 5
  • 3
  • 1
  • 1
ELISA, Western Blotting (WB), Immunohistochemistry (Paraffin-embedded Sections) (IHC (p)), Flow Cytometry (FACS)
  • Épitope

    • 15
    • 8
    • 3
    • 1
    • 1
    AA 578-847

    Fonction

    MAGEL2 Antibody / MAGE-like protein 2

    Purification

    Antigen affinity purified

    Immunogène

    An E.coli-derived human recombinant protein (amino acids Q578-A847) was used as the immunogen for the MAGEL2 antibody.

    Isotype

    IgG
  • Indications d'application

    Optimal dilution of the MAGEL2 antibody should be determined by the researcher.

    Restrictions

    For Research Use only
  • Format

    Lyophilized

    Buffer

    0.5 mg/mL if reconstituted with 0.2 mL sterile DI water

    Stock

    4 °C,-20 °C

    Stockage commentaire

    After reconstitution, the MAGEL2 antibody can be stored for up to one month at 4oC. For long-term, aliquot and store at -20oC. Avoid repeated freezing and thawing.
  • Antigène

    MAGE-Like 2 (MAGEL2)

    Autre désignation

    MAGEL2

    Sujet

    Prader-Willi syndrome (PWS) is caused by the loss of expression of imprinted genes in chromosome 15q11-q13 region. Affected individuals exhibit neonatal hypotonia, developmental delay, and childhood-onset obesity. Necdin (NDN), a gene involved in the terminal differentiation of neurons, localizes to this region of the genome and has been implicated as one of the genes responsible for the etiology of PWS. This gene is structurally similar to NDN, is also localized to the PWS chromosomal region, and is paternally imprinted, suggesting a possible role for it in PWS.

    UniProt

    Q9UJ55
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