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Septin 5 anticorps

L’anticorps anti-Septin 5 Polyclonal Lapin est utilisé pour la détection de Septin 5 dans des échantillons de Humain et Rat. Il a été validé pour WB et FACS.
N° du produit ABIN7882234
644,88 €
Plus frais de livraison 40,00 € et TVA
100 μg
Destination: France
Envoi sous 6 à 9 jours ouvrables

Aperçu rapide pour Septin 5 anticorps (ABIN7882234)

Antigène

Voir toutes Septin 5 (SEPT5) Anticorps
Septin 5 (SEPT5)

Reactivité

  • 51
  • 29
  • 11
  • 2
Humain, Rat

Hôte

  • 44
  • 7
Lapin

Clonalité

  • 44
  • 6
  • 1
Polyclonal

Conjugué

  • 28
  • 4
  • 4
  • 2
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
Cet anticorp Septin 5 est non-conjugé

Application

  • 48
  • 24
  • 24
  • 13
  • 13
  • 9
  • 5
  • 5
  • 5
  • 3
  • 2
  • 1
  • 1
Western Blotting (WB), Flow Cytometry (FACS)
  • Fonction

    SEPT5 Antibody / Septin 5

    Séquence

    HYENYRAHCI QQMTSKLTQD

    Purification

    Antigen affinity purified

    Immunogène

    Amino acids HYENYRAHCIQQMTSKLTQD were used as the immunogen for the SEPT5 antibody.

    Isotype

    IgG
  • Indications d'application

    Optimal dilution of the SEPT5 antibody should be determined by the researcher.

    Restrictions

    For Research Use only
  • Format

    Lyophilized

    Buffer

    0.5 mg/mL if reconstituted with 0.2 mL sterile DI water

    Stock

    4 °C,-20 °C

    Stockage commentaire

    After reconstitution, the SEPT5 antibody can be stored for up to one month at 4oC. For long-term, aliquot and store at -20oC. Avoid repeated freezing and thawing.
  • Antigène

    Septin 5 (SEPT5)

    Autre désignation

    SEPT5

    Sujet

    This gene is a member of the septin gene family of nucleotide binding proteins, originally described in yeast as cell division cycle regulatory proteins. Septins are highly conserved in yeast, Drosophila, and mouse and appear to regulate cytoskeletal organization. Disruption of septin function disturbs cytokinesis and results in large multinucleate or polyploid cells. This gene is mapped to 22q11, the region frequently deleted in DiGeorge and velocardiofacial syndromes. A translocation involving the MLL gene and this gene has also been reported in patients with acute myeloid leukemia. Alternative splicing results in multiple transcript variants. The presence of a non-consensus polyA signal (AACAAT) in this gene also results in read-through transcription into the downstream neighboring gene (GP1BB, platelet glycoprotein Ib), whereby larger, non-coding transcripts are produced.

    UniProt

    Q99719

    Pathways

    Synaptic Vesicle Exocytosis
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