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KRT14 encodes a member of the keratin family, the most diverse group of intermediate filaments. De plus, nous expédions Keratin 14 Protéines (16) et Keratin 14 Kits (7) et beaucoup plus de produits pour cette protéine.
Showing 10 out of 316 products:
Human Monoclonal KRT14 Primary Antibody pour ICC, FACS - ABIN335216
Bauwens, De Groot, Ramaekers, Veldman, Huizing: Expression of intermediate filament proteins in the adult human vestibular labyrinth. dans The Annals of otology, rhinology, and laryngology 1992
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Human Monoclonal KRT14 Primary Antibody pour ICC, FACS - ABIN335329
Smedts, Ramaekers, Troyanovsky, Pruszczynski, Link, Lane, Leigh, Schijf, Vooijs: Keratin expression in cervical cancer. dans The American journal of pathology 1992
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Human Monoclonal KRT14 Primary Antibody pour ICC, FACS - ABIN335217
Bonfrer, Groeneveld, Korse, van Dalen, Oomen, Ivanyi: Monoclonal antibody M3 used in tissue polypeptide-specific antigen assay for the quantification of tissue polypeptide antigen recognizes keratin 18. dans Tumour biology 1994
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Human Monoclonal KRT14 Primary Antibody pour FACS, IHC (p) - ABIN1687795
Kang, Kannan, Zhang, Martinez, Rosin, Eaves: Characterization of Epithelial Progenitors in Normal Human Palatine Tonsils and Their HPV16 E6/E7-Induced Perturbation. dans Stem cell reports 2015
Cow (Bovine) Polyclonal KRT14 Primary Antibody pour IHC (fro), IHC (p) - ABIN113455
Herrmann, van der Hoeven, Grone, Stewart, Langbein, Kaiser, Liebisch, Gosch, Buchkremer, Drobnik, Schmitz, Stremmel: Mice with targeted disruption of the fatty acid transport protein 4 (Fatp 4, Slc27a4) gene show features of lethal restrictive dermopathy. dans The Journal of cell biology 2003
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findings indicate that ouro1 and ouro2 participate in the process of tail regression as immune antigens and highlight the possibility that the acquired immune system contributes not only to self-defense but also to remodeling processes in morphogenesis.
K14 was coexpressed with alphav-integrin (Montrer ITGAV Anticorps) in fetal and adult corneas and cultured corneolimbal epithelium, and colony-forming efficiency (an indicator of stem cell activity) was similar in cells from both sources.
Loss of keratin 14 is associated with epidermolysis bullosa.
vimentin (Montrer VIM Anticorps) regulates the differentiation switch via modulation of K5/K14 expression. Moreover, because there was a significant correlation between high vimentin (Montrer VIM Anticorps)-K14 expression and recurrence/poor survival in oral cancer patients, vimentin (Montrer VIM Anticorps)-K14 together may prove to be the novel markers for the prognostication of human oral cancer.
We concluded that smoking habits were capable of inducing changes in global DNA methylation (Montrer HELLS Anticorps), miR (Montrer MLXIP Anticorps)-9-3 methylation status and K19 (Montrer KRT19 Anticorps) expression.
data demonstrates that keratinocyte migration requires the interaction between vimentin (Montrer VIM Anticorps) and keratins at the -YRKLLEGEE- sequence at the helical 2B domain of viment
Immunocytochemistry using this antibody cocktail comprises five antibodies recognising p63 (Montrer RPE65 Anticorps), and cytokeratins ( 7, 18, 5 and 14) showed good sensitivity and specificity for diagnosing breast cancers. Thus, this method is useful for mammary cytology using FNA
identification of 29 different mutations in KRT5 (Montrer KRT5 Anticorps) and KRT14, 11 of which were novel, in a Polish cohort of epidermolysis bullosa simplex patients
findings reveal K14 as a key regulator of metastasis and establish the concept that K14(+) epithelial tumor cell clusters disseminate collectively to colonize distant organs
all keratins tested, except for keratin 14, were evenly expressed in all trophoblast cells. Keratin 14 was expressed in a subset of CK7 (Montrer KRT7 Anticorps) positive cells
report a family with a novel heterozygous missense mutation p.Leu418Gln in the KRT14 gene causing epidermolysis bullosa simplex with variable phenotype
Analysis of K14 variants with single or multiple substitutions of cysteine residues points to a spatial and temporal hierarchy in how Cys-4/Cys-40 and Cys-367 regulate keratin assembly in vitro and filament dynamics in live keratinocytes
Disulfide bonding is a novel and important mechanism regulating the assembly, intracellular organization, and dynamics of K14-containing intermediate filaments in skin keratinocytes.
K14-VEGF transgenic mouse a valuable model to study novel immunotherapies for psoriasis
autoantibodies in Scurfy (Montrer FOXP3 Anticorps) mice and patients with IPEX (Montrer FOXP3 Anticorps) target keratin 14
nipple connective tissue and its development: insights from the keratin 14-PTHrP (Montrer PTHLH Anticorps) mouse
The keratin-14 IL-4 (Montrer IL4 Anticorps) transgenic (Tg) mouse model of atopic dermatitis (AD) is characterized by skin infiltration of T cells, early up-regulation of T(h)2 cytokines and late surge of T(h)1 cytokines.
These studies provide a potential mechanism by which deltaNp63 directly governs the expression of K14 in a keratinocyte-specific manner.
Study describes the expression profile of claudins and keratin markers in the accelerated epidermal permeability barrier formation of K14-CaSR (Montrer CASR Anticorps) transgenic mice during development as compared to the wild type from E12.5 to newborn stages.
IL-6 (Montrer IL6 Anticorps) and IFN-gamma (Montrer IFNG Anticorps) stimulated VEGF-A (Montrer VEGFA Anticorps) mRNA production in the skin and in primary keratinocytes of keratin 14 IL-4 (Montrer IL4 Anticorps) transgenic mice
DeltaNp63 alone can restore the expression of the basal keratins and reinitiate the failed epidermal differentiation program in the skin of p63 (Montrer CKAP4 Anticorps) null animals.
This gene encodes a member of the keratin family, the most diverse group of intermediate filaments. This gene product, a type I keratin, is usually found as a heterotetramer with two keratin 5 molecules, a type II keratin. Together they form the cytoskeleton of epithelial cells. Mutations in the genes for these keratins are associated with epidermolysis bullosa simplex. At least one pseudogene has been identified at 17p12-p11.
, cytokeratin 14
, keratin 14 (epidermolysis bullosa simplex, Dowling-Meara, Koebner)
, keratin, type I cytoskeletal 14
, keratin 10
, keratin 10 (epidermolytic hyperkeratosis; keratosis palmaris et plantaris)
, epidermolysis bullosa simplex, Dowling-Meara, Koebner
, keratin complex 1, acidic, gene 14
, type I keratin Ka14
, cytokeratin VII
, epidermal keratin VII
, type I alpha-keratin 14
, ouroboros 2
, keratin, type I cytoskeletal 14-like