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The protein encoded by TNFRSF4 is a member of the TNF-receptor superfamily. De plus, nous expédions Tumor Necrosis Factor Receptor Superfamily, Member 4 Protéines (46) et Tumor Necrosis Factor Receptor Superfamily, Member 4 Kits (23) et beaucoup plus de produits pour cette protéine.
Showing 10 out of 270 products:
Cat (Feline) Monoclonal TNFRSF4 Primary Antibody pour FACS, WB - ABIN180755
Arch, Thompson: 4-1BB and Ox40 are members of a tumor necrosis factor (TNF)-nerve growth factor receptor subfamily that bind TNF receptor-associated factors and activate nuclear factor kappaB. dans Molecular and cellular biology 1998
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Rat (Rattus) Monoclonal TNFRSF4 Primary Antibody pour FACS, IHC (fro) - ABIN120515
Mallett, Fossum, Barclay: Characterization of the MRC OX40 antigen of activated CD4 positive T lymphocytes--a molecule related to nerve growth factor receptor. dans The EMBO journal 1990
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Rat (Rattus) Monoclonal TNFRSF4 Primary Antibody pour FACS, IHC (fro) - ABIN120516
Paterson, Jefferies, Green, Brandon, Corthesy, Puklavec, Williams: Antigens of activated rat T lymphocytes including a molecule of 50,000 Mr detected only on CD4 positive T blasts. dans Molecular immunology 1988
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Mouse (Murine) Monoclonal TNFRSF4 Primary Antibody pour FACS - ABIN118276
Takeda, Oshima, Hayakawa, Akiba, Atsuta, Kobata, Kobayashi, Ito, Yagita, Okumura: CD27-mediated activation of murine NK cells. dans Journal of immunology (Baltimore, Md. : 1950) 2000
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Mouse (Murine) Monoclonal TNFRSF4 Primary Antibody pour FACS, IHC (fro) - ABIN118274
Higgins, McDonald, Whittle, Crockett, Shields, MacDonald et al.: Regulation of T cell activation in vitro and in vivo by targeting the OX40-OX40 ligand interaction: amelioration of ongoing inflammatory bowel disease with an OX40-IgG fusion protein, but not with an ... dans Journal of immunology (Baltimore, Md. : 1950) 1999
Rat (Rattus) Monoclonal TNFRSF4 Primary Antibody pour FACS, IHC (fro) - ABIN256199
Crook, Hunt: Enrichment of early fetal-liver hemopoietic stem cells of the rat using monoclonal antibodies against the transferrin receptor, Thy-1, and MRC-OX82. dans Developmental immunology 1996
bone marrow-derived mast cell (BMMC)-exosomes facilitated the differentiation of naive CD4 (Montrer CD4 Anticorps)+ T cells to Th2 cells by ligation of OX40L (Montrer TNFSF4 Anticorps) and OX40 between BMMC-exosomes and CD4 (Montrer CD4 Anticorps)+ T cells and represents a novel mechanism of cell-to-cell communication
Crystal structures and NMR data show that the Roquin (Montrer RC3H1 Anticorps)-1 ROQ domain recognizes hexaloops in the SELEX-derived alternative decay element (ADE) and in an ADE-like variant present in the Ox40 3'-UTR (Montrer UTS2R Anticorps) with identical binding modes.
The OX40-OX40 ligand (Montrer TNFSF4 Anticorps) interaction up-regulates intracellular levels of reactive oxygen species in atherogenesis.
OX40 was highly expressed by intratumoral T cells, particularly those of the FoxP3 (Montrer FOXP3 Anticorps)(+) regulatory T-cell (Treg) lineage.
OX40 and IL-7 (Montrer IL7 Anticorps) play synergistic, but distinct roles in the homeostatic proliferation of CD4 (Montrer CD4 Anticorps)(+) effector memory T cells
OX40-OX40L (Montrer TNFSF4 Anticorps) interaction regulates the expression of NFATc1 (Montrer NFATC1 Anticorps), which may play a critical role in atherosclerotic plaque formation, and may therefore have implications with pathophysiology of atherosclerosis.
During CD134 plus CD137 (Montrer TNFRSF9 Anticorps) dual costimulation, IFN-gamma (Montrer IFNG Anticorps) interacts with IL-2 (Montrer IL2 Anticorps) through distinct mechanisms to program maximal expression of effector molecules in antigen-responding T-cells.
OX40 stimulation of virus-specific CD4 (Montrer CD4 Anticorps) T cells promoted expression of the transcriptional repressor Blimp-1 (Montrer PRDM1 Anticorps) and diverted the majority of cells away from follicular Th cell differentiation.
interruption of the OX40-OX40L signaling pathway, with decreases in dietary cholesterol, induces regression of atherosclerosis by induction of IL-5-producing T cells and oxidized low-density lipoprotein-specific IgM and reductions in Th2 and mast cells.
OX40-OX40L (Montrer TNFSF4 Anticorps) interaction promotes proliferation and activation of lymphocytes via NFATc1 (Montrer NFATC1 Anticorps) in ApoE (Montrer APOE Anticorps)-deficient mice.
blocking of both OX-40L (Montrer TNFSF4 Anticorps) and 4-1BBL (Montrer TNFSF9 Anticorps) reversed radiation-enhanced T-cell killing of human tumor targets as well as T-cell survival and activation.
Low OX40 expression is associated with colorectal cancer.
OX40 and its ligand are co stimulators for T lymphocytes.
These studies provide the first direct evidence that ligation of tumour necrosis factor (Montrer TNF Anticorps) superfamily members and their cognate receptors is important for the control of viral lytic replication.
Malaria patients and Plasmodium-infected rodents exhibit enhanced expression of the co-stimulatory receptor OX40 on CD4 (Montrer CD4 Anticorps) T cells, which is abrogated following coordinate PD-1 (Montrer PDCD1 Anticorps) co-inhibitory pathways, which are also upregulated during malaria.
Identified two key amino acid residues within CD134 that are required for its interaction with herpesvirus 6B (HHV-6B) and for HHV-6B entry into cells. One of the residues (K79) allows access of the HHV-6B ligand to CD134.
TL1A (Montrer TNFSF15 Anticorps) increases expression of CD25 (Montrer IL2RA Anticorps), LFA-1 (Montrer ITGAL Anticorps), CD134 and CD154 (Montrer CD40LG Anticorps), and induces IL-22 (Montrer IL22 Anticorps) and GM-CSF (Montrer CSF2 Anticorps) production from effector CD4 (Montrer CD4 Anticorps) T-cells
Data indicate that soluble OX40 and sOX40L plasma levels are increased in early rheumatoid arthritis patients.
High expression of OX40 is associated with type 1 diabetes.
A cysteine-rich domain of CD134 that is critical for binding to the HHV-6B glycoprotein gH/gL/gQ1/gQ2 complex and HHV-6B infection.
The protein encoded by this gene is a member of the TNF-receptor superfamily. This receptor has been shown to activate NF-kappaB through its interaction with adaptor proteins TRAF2 and TRAF5. Knockout studies in mice suggested that this receptor promotes the expression of apoptosis inhibitors BCL2 and BCL2lL1/BCL2-XL, and thus suppresses apoptosis. The knockout studies also suggested the roles of this receptor in CD4+ T cell response, as well as in T cell-dependent B cell proliferation and differentiation.
tumor necrosis factor receptor superfamily, member 4
, tumor necrosis factor receptor superfamily member 4
, OX40 antigen
, OX40L receptor
, tax-transcriptionally activated glycoprotein 1
, ACT35 antigen
, ATC35 antigen
, CD134 antigen
, OX40 cell surface antigen
, OX40 homologue
, TAX transcriptionally-activated glycoprotein 1 receptor
, lymphoid activation antigene ACT35
, tax-transcriptionally activated glycoprotein 1 receptor
, MRC OX40
, Tax-transcriptionally activated glycoprotein 1
, tumor necrosis factor (ligand) superfamily member 4
, tumor necrosis factor superfamily, member 4