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Aurora Kinase B anticorps (AA 1-344)

L’anticorps Souris Monoclonal anti-Aurora Kinase B a été validé pour WB, ELISA et FACS. Il convient pour détecter Aurora Kinase B dans des échantillons de Humain.
N° du produit ABIN5775901

Aperçu rapide pour Aurora Kinase B anticorps (AA 1-344) (ABIN5775901)

Antigène

Voir toutes Aurora Kinase B (AURKB) Anticorps
Aurora Kinase B (AURKB)

Reactivité

  • 147
  • 62
  • 32
  • 12
  • 4
  • 4
  • 2
  • 2
  • 1
  • 1
  • 1
  • 1
  • 1
Humain

Hôte

  • 122
  • 46
  • 3
  • 1
Souris

Clonalité

  • 100
  • 73
Monoclonal

Conjugué

  • 87
  • 13
  • 7
  • 6
  • 4
  • 4
  • 4
  • 4
  • 4
  • 4
  • 4
  • 4
  • 4
  • 2
  • 2
  • 2
  • 2
  • 2
  • 2
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
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  • 1
Cet anticorp Aurora Kinase B est non-conjugé

Application

  • 87
  • 77
  • 57
  • 35
  • 28
  • 14
  • 14
  • 13
  • 13
  • 13
  • 12
  • 4
  • 3
  • 3
  • 3
  • 2
  • 2
  • 2
  • 1
  • 1
Western Blotting (WB), ELISA, Flow Cytometry (FACS)

Clone

AT2B1
  • Épitope

    • 49
    • 18
    • 12
    • 10
    • 9
    • 8
    • 8
    • 7
    • 6
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    • 3
    • 3
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    • 2
    • 2
    • 2
    • 2
    • 1
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    • 1
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    • 1
    • 1
    • 1
    • 1
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    • 1
    AA 1-344

    Immunogène

    Recombinant human Aurora kinase B (1-344aa) purified from E. coli

    Isotype

    IgG2b kappa
  • Restrictions

    For Research Use only
  • Format

    Liquid

    Concentration

    1 mg/mL

    Stock

    4 °C,-20 °C,-80 °C

    Stockage commentaire

    Can be stored at +2°C to +8°C for 1 week. For long term storage, aliquot and store at -20°C to -80°C. Avoid repeated freezing and thawing cycles.
  • Antigène

    Aurora Kinase B (AURKB)

    Autre désignation

    Aurora B

    Sujet

    The aurora family (A, B and C) are serine threonine kinases and key regulators of chromosome segregation during mitosis. Aurora kinase B is a chromosomal passenger protein that regulates chromosome segregation and cytokinesis. Aurora kinase B is associated with the level of genetic instability within tumours and patient survival. It is strongly expressed in exponentially proliferating bronchial epithelial cells in culture and that this expression is markedly reduced in confluent cells. It is also shown that almost all tumours show higher levels of Aurora kinase B expression than their matched normal lung tissues, which could therefore simply be a consequence of a higher proliferative index, or be typical of the progenitor cell and atypical of the bulk of normal lung cells.

    NCBI Accession

    NP_004208

    Pathways

    TCR Signaling, Cycle Cellulaire, Maintenance of Protein Location, Hepatitis C, Toll-Like Receptors Cascades
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