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Bcl-2 Kit ELISA

Kit ELISA Bcl-2 Humain, Colorimetric test pour la quantification de Humain Bcl-2.
N° du produit ABIN4993797
586,60 €
Plus frais de livraison 40,00 € et TVA
96 tests
Destination: France
Envoi sous 7 à 10 jours ouvrables

Aperçu rapide pour Bcl-2 Kit ELISA (ABIN4993797)

Antigène

Voir toutes Bcl-2 (BCL2) Kits ELISA
Bcl-2 (BCL2) (B-Cell CLL/lymphoma 2 (BCL2))

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Humain

Méthode de détection

Colorimetric

Type de méthode

Sandwich ELISA

Gamme de detection

50.25-30000 pg/mL

Application

ELISA

Type d'échantillon

Cell Culture Supernatant, Plasma, Serum
  • Seuil minimal de détection

    50.25 pg/mL

    Fonction

    This assay employs the quantitative sandwich enzyme immunoassay technique for the quantitative detection of human Bcl-2.

    Analytical Method

    Quantitative

    Specificité

    BCL2

    Ingrédients

    plate, standard, diluent

    Matériel non inclus

    pipettes, tubes, reader
  • Volume d'échantillon

    100 μL

    Durée du test

    3 - 4 h

    Plaque

    Pre-coated

    Restrictions

    For Research Use only
  • Buffer

    0.02 % sodium azide

    Agent conservateur

    Sodium azide

    Précaution d'utilisation

    This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

    Stock

    4 °C
  • Antigène Voir toutes Bcl-2 (BCL2) Kits ELISA

    Bcl-2 (BCL2) (B-Cell CLL/lymphoma 2 (BCL2))

    Autre désignation

    BCL2

    Sujet

    B-cell lymphoma 2 (Bcl-2), is the founding member of the Bcl-2 family of regulator proteins that regulate cell death (apoptosis), by either inducing (pro-apoptotic) or inhibiting (anti-apoptotic) apoptosis. Bcl-2 is specifically considered an important anti-apoptotic protein and is thus classified as an oncogene.

    NCBI Accession

    NM_000633

    UniProt

    P10415

    Pathways

    Signalisation MAPK, Signalisation PI3K-Akt, Apoptose, Caspase Cascade in Apoptosis, Regulation of Muscle Cell Differentiation, Cell-Cell Junction Organization, Skeletal Muscle Fiber Development, Autophagy, Smooth Muscle Cell Migration, Negative Regulation of intrinsic apoptotic Signaling
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